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Samenvatting Perifere cardiovasculaire centra - beïnvloeding $3.17
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Samenvatting Perifere cardiovasculaire centra - beïnvloeding

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hier wordt nog eens samengevat welke factoren de perifere cardiovasculaire centra beïnvloeden.

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  • January 16, 2022
  • 2
  • 2021/2022
  • Summary
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Baroreceptorreflex = drukreceptoren
Ligging: wand van de arterie thv carotissinus en Pulmonale stretch
aortaboog mechanoreceptoren
Prikkeling: uitrekking arteriewand Mechanoreceptoren long 
Gevolg van prikkeling: geprikkeld bij inademing 
Atriale stretch of volumereceptoren
- Signalen naar CVC beïnvloeden CVC 
Gevolg van prikkeling:
- Vasodilatatie reflectoire tachycardie
- Vasodilatatie
- Lagere hartfrequentie
- Hogere hartfrequentie
 Acute regeling bloeddruk via neg terugkoppeling.  Toename hartritme
Bloeding  mindere prikkeling volumereceptoren bij iedere inademing
Drempelwaarde  50 mmHg
 reflectoire vasoconstrictie + vrijstelling
 Toenemende druk  toenemende activiteit
hypovolemische hormonen
Max waarde  150 à 200 mmHg
Slaan bij bloedverlies vroeger alarm dan de
baroreceptoren  ze moeten rapper detecteren
dan dat de druk in arteriën kan zakken



Perifere CVC beïnvloeding
Reflexen bij spierarbeid
- Mechanoreceptoren: proprioceptoren Andere reflexen
Effect: tachycardie en vasoconstrictie - Somatische pijn
- Metaboreceptoren: geactiveerd door  tachycardie +
Chemoreceptorreflex
vrijgekomen metabolieten tijdens spieractiviteit hypertensie
Lokalisatie: glomus caroticum thv aorta
Effect: hogere bloeddruk - Viscerale pijn
Prikkeling bij:
 grotere toename bij isometrische inspanning  bradycardie +
- Hypoxie
syncope
- Hypercapnie
- Seksuele
- Acidose
stimulatie
- Hypotensie
 tachycardie +
Gevolgen prikkeling:
hypertensie
- Orthosympatische constrictie arteriolen
- Koud water in
- Tachycardie
gezicht
Ondersteunen hersendoorbloeding bij lage arteriële BD
 bradycardie

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