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NR 507 Week 6 Quiz 6 (Version 2), NR 507: Advanced Pathophysiology, Chamberlain College of Nursing. (Secure HIGHSCORE) $12.49   Add to cart
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NR 507 Week 6 Quiz 6 (Version 2), NR 507: Advanced Pathophysiology, Chamberlain College of Nursing. (Secure HIGHSCORE)
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NR 507 Week 6 Quiz 6 (Version 2), NR 507: Advanced Pathophysiology, Chamberlain College of Nursing. (Secure HIGHSCORE)

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  • May 12, 2023
  • 10
  • 2022/2023
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  • nr 507 week 6 quiz 6 version 2
  • nr 507 advanced pathophysiology
  • chamberlain college of nursing secure highscore

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NR 507 Week 6 Quiz
NR 507: Advanced Pathophysiology
(Secure HIGHSCORE)

, Advanced Pathophysiology Week 6 Quiz

Question 1
pts
_____ is a chronic inflammatory joint disease characterized by stiffening and fusion of the spine and
sacroiliac joints.

Fibromyalgia

Ankylosing spondylitis

Paget disease
You Answered

Rheumatoid arthritis
Ankylosing spondylitis (AS) (spondyloarthritis) is a chronic, inflammatory joint disease
characterized by stiffening and fusion (ankylosis) of the spine and sacroiliac joints.

Question 2
pts
Considering the pathophysiology of osteoporosis, what are the effects of extracellular signal regulated
kinases (ERKs) and receptor activator of RANKL on osteoblasts and osteoclasts?
Correct!

ERKs and RANKL increase the life span of osteoclasts and decrease the life span of osteoblasts.

ERKs and RANKL increase the life span of osteoblasts and decrease the life span of osteoclasts.

ERKs increase the life span of osteoclasts and RANKL decreases the life span of osteoblasts.

ERKs increase the life span of osteoblasts and RANKL decreases the life span of osteoclasts.
In addition to ERKs, RANKL is required for the antiapoptotic and thus longer life span of
osteoclasts. These effects increase the life span of osteoclasts (i.e., longer bone
resorbing) and shorten the life span of the bone-forming cells, or osteoblasts.

Question 3
pts
Rhabdomyolysis is characterized by

lysis of skeletal muscle cells through the initiation of the complement cascade.

paralysis of skeletal muscles resulting from impaired nerve supply.

smooth muscle degeneration resulting from ischemia.

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