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Gebitspathologie college 5
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Gebitspathologie college 5 Nageluisterd en voorzien van college aantekeningen
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Gebitspathologiecollege
5:
Fracturen
en
slijtage
Boek!H8
en
9
PATHOLOGY
OF
DENTAL
HARD
TISSUES
Four
categories
of
disorders:
I.
Pre-‐eruptive:
deviating
a)
number,
b)morphology,
c)
structure
II.
Eruptive:
abnormal
time
/
location
III.
Posteruptive
:d)
aging/wear,
....
IV.
Syndromes
AGEING
ENAMEL
The
enamel
wears
1.
Wear
is
used
in
forensics
and
anthropology
for
age
estimation
(risky)
2.
The
permeability
of
the
enamel
changes
through
uptake
of
ions
AGEING
DENTINE:
becomes
more
mineralised
and
harder
Dead
tract:
empty
tubules
(opaque
areas)
result
from
peripheral
irritation
Sclerosis
(minerals
in
tubules)
makes
dentinal
areas
more
translucent
(see
root
apex)
Fluorescence
under
green
light
increases
ROOT
APEX
IN
AGING
Root
becomes
translucent,
starting
from
the
apex
Correlation
with
age:
0.55
to
0.88
AGEING
PULP:
Physiologic
secondary
dentine
narrows
the
pulp,
disproportionally
at
bottom
and
roof
chamber:
pulp
length
7
mm
shorter
in
elderly.
Tertiary
dentine
(upon
external
stimuli)
with
few
and
irregular
tubules,
is
a
poor
barrier,
e.g.
reactionary
by
odontoblasts
and
reparative
dentine
by
a
new
generation
of
odontoblasts
(see
ch.
5)
PULP
OBLITERATION
In
elderly
but
also
after
trauma
at
a
young
age
1
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