Summary of lecture notes containing detailed info:
Anti hypertensives
Heart failure
Antiplateletes and anticoagulants
Hyperlipidaemia
Ischaemic heart disease
Anti arrhythmithic
Essential/primary hypertension:bp>140/90mmHg with no
identifiable cause -
develop gradually over many yrs. occurs after
early adulthood (prevalence 4 with ages
·Patients 140:high cardiac output with normal peripheral
resistance
older patients:normal/I cardiac outputwith a peripheral
resistance
,·
secondary hypertension:high by caused by underlying condition
that tends to appear suddenly
x linked to:obstructive sleep apnoed. Kidney problems, advenal
gland tumours, thyroid problems, congenital defect of blood
sympathetic nerves
ctions
A of Angiotensin II that 4 bp:
1) Stir. advenal cortexto release aldo.
2) Release of ADH from pithatary which 4
(inactives water reabsorption in kidney:4 D.D.
4
(actives 3) Stimulates thirst
I
4) causes vasoconstriction:4 b.p.
5) cardiac,vascular hypertrophy-- muscle
mass:4 cardiac output
, ↑ athophysiologies of hypertension
Atherosclerosis:development of fatty plaques,cholesterol in
·
wall of arteries as part of inflammatory response - over yrs.
↳ laque gets bigger eventually causing vessel occlusion and
thrombus formation
·
other pathophysiologies:
1) Trouble with memory/understanding:due to uncontrolled
high up affecting ability to think, remember, learn
2) Dementia:narrowed/blocked arteries can limit blood flow to
brain):vascular demential or stroke that interrups blood to
brain
·
A abdominal
:
aorta
B c Kidney in artery
bulge
=
+
D:aortic aneurysm: clinical significance as may
rupture with massive blood,likely death
E F +
COMMON
=
iliac artery
Mechanisms controlling DD
BD CO resistance
-
=
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