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Resumen de clase de farmacologia
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Resumen de clase de farmacologia
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“EN CONTRA DEL TROMBO”ANTIPLAQUETARIOS – ANTIAGREGANTES
Modificación de enzimas: COX1-TX–bloqueo Receptores – Inhibición P2Y12 Complejo – Inhibición: IIb/IIIa
Clopidogrel Tirofibán
ASA Ticagrelor Abxicimab
Prasugrel Eptifibatide
Cangrelol
ANTICOAGULANTES
Indirectos Directos
Orales Parenterales Orales
Vit k - Bloqueo Heparinas/Heparinoides Otros Rivaroxabán
Heparina no Heparinas de bajo Pentasacárido Derivados de Hirudina Dabigatran
Warfarina fraccionada peso molecular Apixabán
Heparina Enoxaparina Fondaparinux Lepirudina Edoxabán
Sódica Dalteparina Bivalirudina
FIBRINOLÍTICOS
No fibrino – específicos Fibrino – específicos
**Estreptocinasa Alteplasa
Urocinasa Tenecteplasa
COAGULANTES
- PROTAMINA: antagonista de la heparina
- Ácido tranexámico: se une al lugar de fijación de la lisina en el plasminógeno, evitando que éste se pueda unir a la fibrina y así, aunque el
activador tisular del plasminógeno convierta el plasminógeno en plasmina, ésta no podrá lisar la fibrina.
- Vitamina K fitomenadiona es el antídoto de la Warfarina
- Idarucizumab fragmento humanizado de un anticuerpo monoclonal que se une al dabigatran (anticoagulante directo de vía oral)
- Andexanet alfa análogo recombinante del factor Xa, con alta afinidad por los fármacos inhibidores de ese factor (Rivaroxabán, Apixabán,
Edoxabán)
- Factores de coagulación II, VII, VIII, IX, X usados en profilaxis, reposición perioperatoria y tto de sangrados, en pacientes con hemofilias por
deficiencia de los factores dependientes de vitamina K
- Etamsilato (ciclonamina) tiene la capacidad de estabilizar la pared de los capilares, aumentando su resistencia, corrigiendo la adhesión
plaquetaria anormal y disminuyendo el tiempo de sangría.
- Aprotinina polipéptido endógeno que inhibe las kalicreínas, la plasmina y la tripsina disminuyendo los efectos trombolíticos.
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