Physiotherapeutic theory for neurological rehabili
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Summary phys.theory: MS introduction
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Physiotherapeutic theory for neurological rehabili
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Katholieke Universiteit Leuven (KU Leuven)
Summary of the MS intro lesson given by prof G.Verheyden from the course of physiotherapeutic theory. It is a summary of the powerpoint slides and additional items noted during the lesson. The slides were in English and my own notes are always in Dutch as much as possible. It is therefore a mix of ...
Physiotherapeutic theory for neurological rehabili
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Phys theory: MS: intro
Disease course & global evaluation
Chronic progressive disease with inflammation and degeneration processes in the brain and spinal cord
Leasie in hersenen
Leaie in RM= meer impact dan in motor area’s
- Veel tracts multiple tracts
- MS=white matter aangevallen (myeline)
- RM= weinig / geen mogelijkheid neuroplast.
- MS= kan nog neuroplast. Zijn
Hypertonia/spast in MS ≠ hersenen of RM leasie
- RM meer gelokaliseerd in een reflexogene manier
- Type/pheothype
- In RM= meer prob onder leasie
- Meer risico spasme abductie &flex&extensie
Diagose moet snel gesteld worden
Kripen hersenen= mindr plek voor uitvoeren complexere taken
- Geen neuroreserve meer
Diagnose kan makkelijker & technischer gemaakt worden
- Vroeger = min 2 symptome vertonen
Vaak pas zeer laat diagnose krijgen
- Vroeger minder gefocusd op sec. progressie
Meer op relapsing-remitting
- CIS clinically important symptoms (figuur)
Differentiation between inflammation and degeneration
- Manifistations in P are different
- Lot of inflammation -> medication
- If not it is a questionmark…
- With matter lesions on the T2 scan, it can also occur in the SC
1. DIFFERENT TYPES OF MS
‘Relapsing-remitting’ versus
‘progressive MS’
‘Active’ versus ‘non-active’
The classification that we know now,
but this will change, there are
researches happening
CIS can be active or not active
When the symptom doesn’t come back
it is not active
Active can lead to a RRMS when it does
come back several times
RRMS
- Active: a lot of lesions in the brain
that progress
- Not-active: not that much lesions in the brain, and no progression in the brain
Active & met progressive
- Active= Elke 2 j. scan & verergering zien
- Progressive= symptome
Nala Melis Pagina 1
, Phys theory: MS: intro
1.1. PROGRESSIVE MS
1.2. RRMS – PIRA
Progression In absence of Relapse Activity
PIRA: there can be a progression happening but very
subtle
Typically in RRMS
We should measure the activity and the progression to
know how the patient is doing
Behavioural changes that may be happening
T25-FW=time 25 foot walk
- 7.62m zo snel mogelijk
- Wandelsnelheid
Misschein eerder kijken naar activiteit ipv progressie
Nala Melis Pagina 2
, Phys theory: MS: intro
2. CLINICAL COURSE OF MS
Future: new names and terms
Maybe the 2 questionmarks will emerge
There is always a progression?
Neurodegenerative disease: it is present in the early phase but not realy visible, think about PIRA!
Health behaviour: smoking is proven to be very negative for your progression
Not every patient benefits from drugs (medication)
3. Disease Modifying drugs (DMD) for RRMS / PMS
4. SYMPTOMS
Veel invissible symptomen
- Cogitieve functie,…
Fatigue= numer 1
Ahankelijk per P welke symptomen
Pijn
- Neuorplastisch
- Neuropatisch
Emotional changes
Symptomen
- Fatigue
- Weakenss
- Spasms
- Cognition
- Depression
- Reduced walking
- Sensory-loss
- Balance problems
- Increased temperature will lead to a worse performance
- Dual tasking problems
Symptom interconnectivities
Nala Melis Pagina 3
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