CCRN REVIEW (A+ GRADED ALREADY) 2024.

SIADH correct answers too much ADH low Na, hypo-osmolar, low UOP ADH correct answers made in hypothalamus stored in pituitary released to kidney and makes kidney hold onto h20 serum osmolarity correct answers Na X2 275-295 low=fluid overloaded high=concentrated SIADH CAUSES correct answers oat cell carcinoma (bronchogenic CA)-makes its own ADH Viral PNA Head Problem inc. serum osmo, anesthesia, analgesics, stress COMPLICATIONS OF SIADH correct answers Sz's TREATMENT of SIADH correct answers get rid of causes fluid restrictions hypertonic solutions (3%, D5NS, D51/2NS) give 25-50cc/hr d/t CHF DI correct answers No ADH (no h20 at kidney) inc Na+ levels, inc osmolarity, inc. UOP (spec grav 1.001-1.005) DI Causes correct answers head problems dilantin DI Treatment correct answers Give ADH (PItressin or vasopressin) Give fluids to increase Intravascular volume monitor UOP *monitor fo ischemia Hypoglycemia correct answers CVS s/s tachycardia, palpitations, diaphoresis, irritable, restlessness CNS s/s confusion, lethargy, slurred speech, sz, coma hypoglycemia pathophys correct answers low glucose-adrenal medulla knows and releases adrenaline-liver releases glycogen which is converted into glucose to increase BGL if block in adrenaline or liver cant convert glycogen into glucose (AKA BETA BLOCKERS) then CVS s/s wont occur