Exam (elaborations)
NR 507 Advanced Pathophysiology MIDTERM EXAM 2024 CHAMBERLAIN UNIVERSITY EXAM 1
- Institution
- Chamberlain School Of Nursing
NR 507 Advanced Pathophysiology MIDTERM EXAM 2024 CHAMBERLAIN UNIVERSITY EXAM 1
[Show more]
Content preview
NR 507 Advanced Pathophysiology MIDTERM EXAM 2024 CHAMBERLAIN UNIVERSITY EXAM 1 Hypersensitivity: Typ e 1 -Mediated IgE -inflammation due to mast cell degranultion -anaphylactic , asthma, hay fever treatment: epinephrine Hypersensitivity: Type 2 -Cytotoxic reaction: tissue specific -binds to the antigen ON THE CELL SURFACE -macrophages are primary effecto rs cells involved cause tissue damage or alter function Hypersensitivity 2 -Graves Disease (hyperthyroidism) -alters function not destroy -blood transfusions reaction - transfused erythrocytes are destroyed by agglutination or lysis -drug allergies -hemolytic anemia Hypersensitivity: type 3 -Not organ specific -antibody binds to soluble antigen OUTSIDE THE CELL SURFACE that was released into the blood or bodi ly fluids, and the complex is then deposited in the tissues Hypersensitiv ity: Type 3 -rheumatoid arthritis -Systemic Lupus Erythematosus (SLE) -Raynaud's Hives (urticaria) Hypersensitivity Type 1 First responders to innate the immune system Neutrophils Allergic contact dermatitis Hypersensitivity Type IV Type 2 cytotoxic hypersensitivity mediated by: -IgG IgM -macrophages are primary effort cells - Serum Sickness type 3 hypersensitivity Hypersensitivity IV, -is more of a delayed immune response. -mediated T-cells attack tissue directly (no antibodies) Autoimmune -diseases in which the body makes antibodies directed against its own tissues - Primary immunodeficiency -Genetic; inherited -result of single gene defects -B and T cell deficiencies secondary immunodeficiency -acquired Common cause of secondary immunodeficiency Malnutrition Most patients diagnosed with SLE will have a positive ? antinuclear antibody (ANA ) SLE (lupus) -Facial rash -vasculitis - tissue inflammation Renal disease associated with autoimmunity? Glomerul onephritis Sjogren's syndrome Inflammation in salivary and lacrimal glands SLE Autoantibodies and auto -active t -cells against DNA and nucleoprotein Sjögren's syndrome immune changes: Autoantibodies and auto -reactive t -cells against apoptotic cellsThe benefits of buying summaries with Stuvia:
Guaranteed quality through customer reviews
Stuvia customers have reviewed more than 700,000 summaries. This how you know that you are buying the best documents.
Quick and easy check-out
You can quickly pay through credit card or Stuvia-credit for the summaries. There is no membership needed.
Focus on what matters
Your fellow students write the study notes themselves, which is why the documents are always reliable and up-to-date. This ensures you quickly get to the core!
Frequently asked questions
What do I get when I buy this document?
You get a PDF, available immediately after your purchase. The purchased document is accessible anytime, anywhere and indefinitely through your profile.
Satisfaction guarantee: how does it work?
Our satisfaction guarantee ensures that you always find a study document that suits you well. You fill out a form, and our customer service team takes care of the rest.
Who am I buying these notes from?
Stuvia is a marketplace, so you are not buying this document from us, but from seller NURSMERIT. Stuvia facilitates payment to the seller.
Will I be stuck with a subscription?
No, you only buy these notes for $13.49. You're not tied to anything after your purchase.
Can Stuvia be trusted?
4.6 stars on Google & Trustpilot (+1000 reviews)
50155 documents were sold in the last 30 days
Founded in 2010, the go-to place to buy study notes for 14 years now