TNCC Exam Already Passed
- ✔✔✔Prehospital shock index pg. 85
____________ ___________ Injury: shearing or tearing. Diagnosed with MRI. Widespread microscopic
hemorrhage. - ✔✔✔Diffuse Axonal Injury (Cannot recover from this; shearing/tearing portion DOES
NOT heal)
____________ ___________ Injury: shearing or tearing. Diagnosed with MRI. Widespread microscopic
hemorrhage. - ✔✔✔Diffuse Axonal Injury (Cannot recover from this; shearing/tearing portion DOES
NOT heal)
______________ Shock: Spinal cord injury at any level. Transient loss of reflex below the level of injury.
Variable duration. S & S: flaccidity, loss of reflexes, bowel/bladder dysfunction. - ✔✔✔Spinal
______________ Shock: Spinal cord injury at any level. Transient loss of reflex below the level of injury.
Variable duration. S & S: flaccidity, loss of reflexes, bowel/bladder dysfunction. - ✔✔✔Spinal
__________________ Shock: Spinal cord injury at T6 or above. Temporary loss of vasomotor tone and
sympathetic innervation. Temporary duration usually <72 hours. S & S: hypotension, bradycardia, loss of
ability to sweat below level of injury. - ✔✔✔Neurogenic
__________________ Shock: Spinal cord injury at T6 or above. Temporary loss of vasomotor tone and
sympathetic innervation. Temporary duration usually <72 hours. S & S: hypotension, bradycardia, loss of
ability to sweat below level of injury. - ✔✔✔Neurogenic
___________________: Impairs thrombin production and platelet function
_____________ ______________: Impairs thrombin production
_______________________: Results in depletion of clotting factors through hemodilution and the
impaired ability to produce clotting factors. - ✔✔✔Trauma Triad of Death
,1. Hypothermia
2. Metabolic Acidosis
3. Coagulopathy
___________________: Impairs thrombin production and platelet function
_____________ ______________: Impairs thrombin production
_______________________: Results in depletion of clotting factors through hemodilution and the
impaired ability to produce clotting factors. - ✔✔✔Trauma Triad of Death
1. Hypothermia
2. Metabolic Acidosis
3. Coagulopathy
???;.][''''''''''''''''''' - ✔✔✔this card was created by a cat. enjoy.
.. activation: .... are found in the carotid sinus and along the aortic arch, are sensitive to the degree of
stretch in the arterial wall. When the receptors sense a decrease in stretch, they stimulate the
sympathetic nervous system to release Epi, norepi, causing stimulation of cardiac activity and
constriction of blood vessels, which causes a rise in heart rate and diastolic blood pressure - ✔✔✔
Baroreceptors:
... describes the concept of under treatment of pain. - ✔✔✔Oligoanalgesia
... describes the concept of under treatment of pain. - ✔✔✔Oligoanalgesia
... describes the concept of under treatment of pain. - ✔✔✔Oligoanalgesia
... is a principle that confuses on prevention rather than intervention. - ✔✔✔Damage control
resuscitation
,... is a principle that confuses on prevention rather than intervention. - ✔✔✔Damage control
resuscitation
... is a principle that confuses on prevention rather than intervention. - ✔✔✔Damage control
resuscitation
... is a test that requires fluid to be sent to the lab and is considered the gold standard for identifying CSF
- ✔✔✔Beta2-Transferrin
... is a test that requires fluid to be sent to the lab and is considered the gold standard for identifying CSF
- ✔✔✔Beta2-Transferrin
... is a test that requires fluid to be sent to the lab and is considered the gold standard for identifying CSF
- ✔✔✔Beta2-Transferrin
... is a triad of assessment findings; widening pulse pressure, bradycardia, and diminished respiratory
effort - ✔✔✔Cushing ( it is an attempt to increase MAP against elevated ICP, ultimately trying to cause
a rise in CPP)
... is a triad of assessment findings; widening pulse pressure, bradycardia, and diminished respiratory
effort - ✔✔✔Cushing ( it is an attempt to increase MAP against elevated ICP, ultimately trying to cause
a rise in CPP)
... is a triad of assessment findings; widening pulse pressure, bradycardia, and diminished respiratory
effort - ✔✔✔Cushing ( it is an attempt to increase MAP against elevated ICP, ultimately trying to cause
a rise in CPP)
... is damaged brain tissue usually caused by blunt trauma - ✔✔✔Cerebral contusion
, ... is damaged brain tissue usually caused by blunt trauma - ✔✔✔Cerebral contusion
... is damaged brain tissue usually caused by blunt trauma - ✔✔✔Cerebral contusion
... is defined as the pressure gradient across the brain tissue, or the difference between the pressures of
the cerebral artery and venous vessels. - ✔✔✔CPP; CPP = map - icp
... is defined as the pressure gradient across the brain tissue, or the difference between the pressures of
the cerebral artery and venous vessels. - ✔✔✔CPP; CPP = map - icp
... is defined as the pressure gradient across the brain tissue, or the difference between the pressures of
the cerebral artery and venous vessels. - ✔✔✔CPP; CPP = map - icp
... is the initial post traumatic inflammatory response. ... ... activates this response and ... are sent to the
injury sites, activating signaling pathways that mobilize inflammatory cells. - ✔✔✔Immune response;
tissue hypoxia;neutrophils
... is the initial post traumatic inflammatory response. ... ... activates this response and ... are sent to the
injury sites, activating signaling pathways that mobilize inflammatory cells. - ✔✔✔Immune response;
tissue hypoxia;neutrophils
... is the initial post traumatic inflammatory response. ... ... activates this response and ... are sent to the
injury sites, activating signaling pathways that mobilize inflammatory cells. - ✔✔✔Immune response;
tissue hypoxia;neutrophils
... is the shifting of brain tissue with displacement into another compartment as the result of bleeding or
edema. - ✔✔✔Herniation
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