A 1-4 page document written by a final year medical student with distinction grades in the uploaded modules. These notes are concise and of very high quality - using a combination of textbooks, lectures, and current guidelines (NICE and RCOG). These documents are the only resource you should need f...
Pathophysiology
1) Major mechanisms:
- Inflammaton of uterus (e.g. UTI, intra-amniotc infecton) breakdown of foetal membranes (by
bacterial enzymes, infammatory cells etc.)
- Exiessive uterine stretih (e.g. twins, polyhydramnios) early actvaton of Ferguson refex
- Deiidual haemorrhage (e.g. placental abrupton) uterine contractons to prevent bleed
- Maternal stress (e.g. depression) or Foetal stress (e.g. IUGR) Actvaton of HPA axis
2) This leads to
- A breakdown in the mechanisms responsible for maintaining uterine quiescence throughout
pregnancy OR
- A short circuitng or overwhelming of the normal parturiton cascade
3) Labour is prematurely triggered
Complicatons
Prematurity is the BIGGEST cause of Neonatal death (i.e. death in frst 28 days of life)
Neurodevelopmental defiit (Cerebral palsy, learning difcultes, visual impairment)
Diagnosis/Screening
Examinaton
- Abdo exam - Uterine iontraitons –more intense and frequent than Braxton hicks
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