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CCRN REVIEW QUESTIONS WITH ANSWERS 2024. $14.49   Add to cart

Exam (elaborations)

CCRN REVIEW QUESTIONS WITH ANSWERS 2024.

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CCRN REVIEW QUESTIONS WITH ANSWERS 2024.

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  • August 14, 2024
  • 28
  • 2024/2025
  • Exam (elaborations)
  • Questions & answers
  • CCRN
  • CCRN
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CCRN REVIEW QUESTIONS WITH
ANSWERS 2024

SIADH - ANSWER: ➡ too much ADH t t tt t t t




low Na, hypo-osmolar, low UOP
t t t t




ADH - ANSWER: ➡ made in hypothalamus
t t tt t t t




stored in pituitary t t




released to kidney and makes kidney hold onto h20
t t t t t t t t




serum osmolarity - ANSWER: ➡ Na X2
t t t tt t t t




275-295
low=fluid overloaded t




high=concentrated



SIADH CAUSES - ANSWER: ➡ oat cell carcinoma (bronchogenic CA)-makes its own ADH
t t t tt t t t t t t t t




Viral PNA t




Head Problem t




inc. serum osmo, anesthesia, analgesics, stress
t t t t t




COMPLICATIONS OF SIADH - ANSWER: ➡ Sz's t t t t tt t




TREATMENT of SIADH - ANSWER: ➡ get rid of causes t t t t tt t t t t




fluid restrictions
t




hypertonic solutions (3%, D5NS, D51/2NS) give 25-50cc/hr d/t CHF
t t t t t t t t

,DI - ANSWER: ➡ No ADH (no h20 at kidney)
t t tt t t t t t t




inc Na+ levels, inc osmolarity, inc. UOP (spec grav 1.001-1.005)
t t t t t t t t t




DI Causes - ANSWER: ➡ head problems
t t t tt t t




dilantin


DI Treatment - ANSWER: ➡ Give ADH (PItressin or vasopressin)
t t t tt t t t t t




Give fluids to increase Intravascular volume
t t t t t




monitor UOP t




*monitor fo ischemia t t




Hypoglycemia - ANSWER: ➡ CVS s/s t t tt t t




tachycardia, palpitations, diaphoresis, irritable, restlessness t t t t




CNS s/s t




confusion, lethargy, slurred speech, sz, coma t t t t t




hypoglycemia pathophys - ANSWER: ➡ low glucose->adrenal medulla knows and releases t t t tt t t t t t t




adrenaline->liver releases glycogen which is converted into glucose to increase BGL
t t t t t t t t t t t




if block in adrenaline or liver cant convert glycogen into glucose (AKA BETA BLOCKERS) then
t t t t t t t t t t t t t t




CVS s/s wont occur
t t t t




DKA s/s - ANSWER: ➡ only in insulin dependent diabetics
t t t tt t t t t t




BGL 400-900 t




dehydrated (4-6L lost) t t




No circulating insulin
t t




+acidosis b/c body breaks down fat into ketones t t t t t t t

, Kussmaul (inc rate depth to blow off c02) t t t t t t t




DKA and HHNK treatment - ANSWER: ➡ insulin gtt
t t t t t tt t t




IVF (more for HHNK)
t t t




NS 1st- to hydrate vascular compartment
t t t t t




1/2NS to hydrate cell t t t




D51/2NS to prevent hypoglycemia t t t




**for every decrease in pH by 0.1, K+increases by 0.6
t t t t t t t t t




HHNK vulnerable population - ANSWER: ➡ OLD AGE (pancrease gets tired)
t t t t tt t t t t t




diet controlled diabetics
t t




people on TPN (gets 80% glucose IV->pancreatic fatigue
t t t t t t t




pancreatitis


HHNK s/s - ANSWER: ➡ BGL 1000-2000
t t t tt t t




Severe dehydration (6-8L lost) t t t




+insulin (prevents breakdown of fat) t t t t




No acidosis (baby breaths)
t t t




somogyi phenomenon - ANSWER: ➡ Rapid decrease in serum glucose, usually at night, that
t t t tt t t t t t t t t t




generates the release of glucose-elevating hormones that manifests as an elevated glucose level
t t t t t t t t t t t t t




in the morning.
t t t




dehydration is not a component of this t t t t t t




NPH peak - ANSWER: ➡ 6-10 hrs
t t t tt t t

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