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2024 WALDEN NURS 6630 PSYCHOPHARMACOLOGY STUDY GUIDE EXAM WITH CORRECT ANSWERS
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2024 WALDEN NURS 6630 PSYCHOPHARMACOLOGY STUDY GUIDE EXAM WITH CORRECT ANSWERS
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2024 WALDEN NURS 6630PSYCHOPHARMACOLOGY STUDY
GUIDE EXAM WITH CORRECT
ANSWERS
The synaptic cleft - CORRECT-ANSWERSis the gap between the presynaptic
neuron and the postsynaptic neuron; it contains proteins and scaffolding and
molecular forms of "synaptic glue" to reinforce the connection between the
neurons. Receptors are present on both sides of this cleft and are key
elements of chemical neurotransmission.
classic synaptic neurotransmission - CORRECT-ANSWERSstimulation of a
presynaptic neuron (e.g., by neurotransmitters, light, drugs, hormones,
nerve impulses) causes electrical impulses to be sent to its axon terminal.
These electrical impulses are then converted into chemical messengers and
released to stimulate the receptors of a postsynaptic neuron.
The cytochrome P450 (CYP) enzyme system - CORRECT-ANSWERSmediates
how the body metabolizes many drugs, including antipsychotics.
The CYP enzyme - CORRECT-ANSWERSin the gut wall or liver converts the
drug into a biotransformed product in the bloodstream. After passing through
the gut wall and liver, the drug will exist partly as unchanged drug and partly
as biotransformed drug.
agonist - CORRECT-ANSWERSproduces a conformational change in the G-
protein-linked receptor that turns on the synthesis of second messenger to
the greatest extent possible
full agonist - CORRECT-ANSWERSis generally represented by the naturally
occurring neurotransmitter itself, although some drugs can also act in as full
a manner as the natural neurotransmitter
Constitutive activity - CORRECT-ANSWERSin the absence of agonist, the
receptor's conformation is such that it leads to a low level of activity
Antagonists - CORRECT-ANSWERSblocks agonists (both full and partial) from
binding to G-protein-linked receptors, thus preventing agonists from causing
maximum signal transduction and instead changing the receptor's
conformation back to the same state as exists when no agonist is present
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