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Bank Bates Nursing Guide to Physical Examination and History Taking 3rd Edition by Beth Hogan-Quigley , Mary Louis Palm Chapter 1-24 | 9781975161095 | All Chapters with Answers and Rationals $17.99   Add to cart

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Bank Bates Nursing Guide to Physical Examination and History Taking 3rd Edition by Beth Hogan-Quigley , Mary Louis Palm Chapter 1-24 | 9781975161095 | All Chapters with Answers and Rationals

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Bank Bates Nursing Guide to Physical Examination and History Taking 3rd Edition by Beth Hogan-Quigley , Mary Louis Palm Chapter 1-24 | 9781975161095 | All Chapters with Answers and Rationals

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  • August 21, 2024
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Test Bank Bates Nursing Guide to Physical Examination and
History Taking 3rd Edition by Beth Hogan-Quigley , Mary Louis
Palm Chapter 1-24 | 9781975161095 | All Chapters with
Answers and Rationals
What is the most common symptom in clinical practice? - ANSWER: headache

What is the life prevalence of headache (percentage)? - ANSWER: 30 %

primary headache - ANSWER: without an identified underlying disease

secondary headache - ANSWER: with an identified underlying disease

Headache warning signs - ANSWER: Progressively frequent or severe over a 3-month period.
Sudden onset like a "thunderclap" or "the worst headache of my life".
New onset after age 50 years.
Aggravated or relieved by change in position.
Precipitated by Valsalva maneuver.
Associated symptoms of fever, night sweats, or weight loss.
Presence of cancer, HIV infection, or pregnancy.
Recent head trauma.
Associated papilledema, neck stiffness, or focal neurologic deficits.

three most important attributes of headache - ANSWER: severity, chronologic pattern, associated
symptoms

examples of primary headaches - ANSWER: migraine
tensions
cluster
chronic daily

examples of secondary headaches - ANSWER: underlying structural
systemic
infectious causes

ex. meningitis
subarachnoid hemorrhage

subarachnoid hemorrhage - ANSWER: Thunderclap headache, usually the worst headache of their
lives.

reaching maximal intensity over several minutes occurring 70 % of patients

sentinel leak headache - ANSWER: vascular leak into the subarachnoid space

If headache is severe and of sudden onset, consider - ANSWER: subarachnoid hemorrhage OR
meningitis

new and persisting, progressively severe headaches raise concerns - ANSWER: tumor
abscess
mass lesion

what time of headaches are episodic and tend to peak over several hours - ANSWER: migraine and
tensions headaches

, unilateral headaches occurs in - ANSWER: migraine and cluster headachess

tensions headaches arise in what area of the head - ANSWER: temporal areas

cluster headaches arise in what are of the head - ANSWER: retro orbital

nausea and vomiting are associated with what headaches - ANSWER: migraine
brain tumors
subarachnoid hemorrhage

spark photospisas - ANSWER: flashes of light

fortifications - ANSWER: zig-zag arcs of light

scotomas - ANSWER: areas of visual loss with surrounding normal vision

acute sinusitis - ANSWER: valsalva maneuvers and leaning forward may increase pain

valsalva and lying down may increase pain from - ANSWER: mass lesions due to changing intracranial
pressure

medication for overuse headacche may cause headaches if - ANSWER: they present for more than 15
days a month for 3 months and reverts to less than 15 days when medication is discontinued

percentage of genetic inheritance with migraine - ANSWER: 30 to 50 percent

hyperopia - ANSWER: farsightedness

presbyopia - ANSWER: impaired vision as a result of aging

myopia - ANSWER: nearsightedness

If sudden visual loss is unilateral and painless consider - ANSWER: vitreous hemorrhage from diabetes
or trauma

macular degeneration


retinal detachment

retinal vein occlusion

central retinal artery occlusion

vitreous hemorrhage - ANSWER: Sudden loss of vision with floaters, usually secondary to diabetic
retinopathy

macular degeneration - ANSWER: progressive damage to the macula of the retina

retinal detachment - ANSWER: two layers of the retina separate from each other

Retinal vein occlusion - ANSWER: Blockage of central or branch retinal vein due to compression from
nearby arterial atherosclerosis. Retinal hemorrhage and venous engorgement, edema in affected
area.

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