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TEST BANK FOR Davis Advantage for Pathophysiology Introductory Concepts and Clinical Perspectives 2nd Edition by Theresa Capriotti Chapter 1-46 $19.49   Add to cart
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TEST BANK FOR Davis Advantage for Pathophysiology Introductory Concepts and Clinical Perspectives 2nd Edition by Theresa Capriotti Chapter 1-46
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TEST BANK FOR Davis Advantage for Pathophysiology Introductory Concepts and Clinical Perspectives 2nd Edition by Theresa Capriotti Chapter 1-46

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  • August 31, 2024
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Test Bank for Davis Advantage for Pathophysiology:
Introductory Concepts and Clinical Perspectives, 2nd
Edition, Theresa Capriotti



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mahgitahi




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Chapters1,sThesCellsinsHealthsandsIllness

MultiplesChoice
Identifystheschoicesthatsbestscompletessthesstatementsorsanswerssthesquestion.

s 1.s Whichsstatementsregardingsthessodium–potassiumspumpsisscorrect?
1. Thescell’ssplasmasmembranesissmoressolublestossodiumsionssthanspotassiumsions.
2. Thesconcentrationsofssodiumsionssshouldsbeshighersinsidesthescellscompartment.
3. Thesconcentrationsofspotassiumsionssshouldsbeshighersoutsidesthescellsc
ompartment.
4. Thesactivestransportsinvolvesspumpingsoutsthreessodiumsionssandspumpingsinstwo
potassiumsions.
s

2.sInsthesabsencesofsoxygen,swhichscellularsfunctionscreatessthessamesamountsofsenergysassisscreateds
insthespresencesofsoxygen?
1. Dissipationsofspyruvicsacid
2. Initiationsofsthescitricsacidscycle
3. Activationsofsacetyl-coenzymesA
4. Creationsofsacidosissviaslacticsacid
s 3.s Howsmanysadenosinestriphosphatess(ATPs)saresproducedsinsaerobicsenergysmetabolism?
1.sss 2
2.sss 3
3.sss 34
4.sss 53

s 4.s Whichscellsorganellessdiffersinstheirsnumbersaccordingstosthescell’ssenergysneeds?
1. Ribosomes
2. Mitochondria
3. Ribonucleicsacids
4. Deoxyribonucleicsacids

s 5.s Whichsoptionsbestssupportssthesreasonsmoresenergysissproducedswhensaspersonsissexercising?
1. Exercisescausessansincreasesinsthessynthesissofsprotein.
2. Theresissansincreasesinsthesproductionsofspyruvicsacidsinsthescells.
3. Thesconversionsofspyruvicsacidstoslacticsacidsissincreasedsbysexercise.
4. Musclescellsshavesmoresmitochondriastosmeetsenergysdemands.

s 6.s Whensdoessribosomalsproteinssynthesisscease?
1. Duringsendoplasmicsreticulumsstress
2. Duringsthessynthesissofsadenosinestriphosphates(ATP)
3. Duringsassevereshypoxicsstate
4. Duringsthesprocessingsofsprohormone

s 7.s Whichscellularsorganellessaresresponsiblesforspropellingsmucussandsinhaledsdebrissoutsofstheslungs?
1. Cilia
2. Microfilaments


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3. Secretorysvesicles
4. Endoplasmicsreticula

s 8.s Whichsarestheskeysproteinssinsthescontractilesunitssofsthesmusclescells?
1. Actinsandsmyosin
2. Prohormonesandstubulin
3. Tubulinsandsactin
4. Myosinsandsprohormone

s 9.sssWhichsdeficiencyscausessTay–Sachssdisease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomalsenzymes

s ss 10.s Whichsissascharacteristicsofsadrenoleukodystrophy?
1. Accumulationsofsganglioside
2. Cessationsofsribosomalsproteinssynthesis
3. Accelerationsofscellularsproteasomesactivity
4. Accumulationsofslong-chainsfattysacidssinsthesnervousssystem

s sss 11.s Whichsstatementsregardingsendoplasmicsreticulums(ER)sstresssisscorrect?
1. DuringsERsstress,sproteinssaresrapidlysdegraded.
2. DuringsERsstress,slipidsscannotstravelstostheirspropersintracellularslocations.
3. DuringsERsstress,slong-chainsfattysacidssaccumulatesinsthesnervousssystem.
4. DuringsERsstress,snondegradedssubstancessaccumulatesinsthescells.
s

ss 12.s Asclientsissdiagnosedswithstypes1sdiabetessmellitus.sAtsascellularslevel,swhichsfunctionsisslikelyst
osbesinvolved?
1. Inabilitysofsribosomesstosproducesasspecificstypesofsprotein
2. IncorrectsprocessingsofsasproteinsbysthesGolgisapparatus
3. Stagnationsofsaspreviouslysdynamicsactionsinsmicrotubules
4. Obstructionsofsthessmoothsendoplasmicsreticulum
s

ss 13.s Asnewbornspatientsexhibitsscharacteristicssofsseveresphysical sdeformities.sWhichscellularscomp

onentsissexaminedstosdeterminesthescausesandsprobabilitysofsthesdiseasesbeingsgeneticallystransferred?
1. TransfersRNA
2. RibosomalsRNA
3. DoubleshelixsofsDNA
4. MitochondrialsDNA
s

ss 14.s Ashikersexperiencessmusclespainsandsacidosisswhilesascendingsasmountainsduringsaslong,s
steepsclimb.sWhichsissthesreasonsforsthesesmanifestations?
1. Cellularshypoxia
2. Autolysis
3. Heterolysis
4. Cellularsedema



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s ss 15.s WhichsfactorsprovidessDNAsthesuniquesmolecularsabilitystosreplicate?
1. Thespairingsofsnitrogenoussbases
2. Thespresencesofspyrimidinesbases
3. Thespresencesofsnucleotides
4. Thesnitrogenoussbasesandsphosphatesbond

s ss 16.s Howsmanysnitrogenoussbasesscomposesassinglescodon?
1.sss 2
2.sss 3
3.sss 4
4.sss 5

s ss 17.s WhichscomponentssformsthesstructuresofsDNA?
1. Nucleotides
2. Aminosacids
3. Fattysacids
4. Phosphates

s ss 18.s Whichsfactorsissessentialsinsordersforsproteinssynthesisstosoccur?
1. Free-standingsribosomesswithinsthescell
2. ProteinsblueprintsfromsthescellsofsthesDNA
3. Specificsinformationsfromsthesnucleussofsthescell
4. TransfersRNAstosmovesthesproteinsoutsofsthescell
s

ss 19.s Tetracyclinesantibioticsissprescribedsforsansadultsclientswithschlamydiasinfection.sWhichsi

ssthesmechanismsofsactionsofsthesdrug?
1. Itspreventssthesreplicationsofsbacteria.
2. Itsalterssthesconfigurationsofsbacterialscytoplasm.
3. Itsinterferesswithsthesfunctionsofsbacterialsribosomes.
4. Itsinhibitssthesfunctionssofsbacterialsmitochondria.

s ss 20.s Wheresdoessthesconversionsofsasprohormonesintosashormonestakesplace?
1. Insthesribosomes
2. InsthesGolgisapparatus
3. Insthessecretorysgranules
4. Insthesendoplasmicsreticulum

s ss 21.s Whichsissthescell’ss“mastersmind”?
1. Nucleus
2. Ribosome
3. Golgisapparatus
4. Endoplasmicsreticulum


MultiplesResponse
Identifysonesorsmoreschoicessthatsbestscompletesthesstatementsorsanswersthesquestion.

s ss 22.s Whichsstatementssregardingsthesmicrotubulessarestrue?sSelectsallsthatsapply.


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