100% satisfaction guarantee Immediately available after payment Both online and in PDF No strings attached
logo-home
BIO 669 STUDY GUIDE QUIZ Q&A $13.49   Add to cart

Exam (elaborations)

BIO 669 STUDY GUIDE QUIZ Q&A

 5 views  0 purchase
  • Course
  • BIO 669
  • Institution
  • BIO 669

BIO 669 STUDY GUIDE QUIZ Q&A

Preview 2 out of 8  pages

  • August 31, 2024
  • 8
  • 2024/2025
  • Exam (elaborations)
  • Questions & answers
  • BIO 669
  • BIO 669
avatar-seller
biggdreamer
BIO 669 STUDY GUIDE QUIZ Q&A
Varicose veins - answer-vein in which blood has pooled producing distended, tortous,
palpable vesseks
-caused by trauma or gradual vein distention
Risk factors-female, family hx, fat, pregnant, dvt, prior leg injury

Chronic venous insufficiency - answer-inadequate venous return over a long period due
to varicose veins or valvular incompetence
-can lead to venous stasis ulcer

Thrombus formation - answer-obstruction of venous flow leading to increased venous
pressure
-3 factors (triad of virchow) promote thrombosis=
1)venous stasis 2)venous endothelial damage
3)hypercoaguable states
-usually occurs in lower extremities & is asymptomatic

Superior vena cava syndrome - answer-progressive occlusion of svc that leads to
venous distention in upper extremities & head
-causes=tumors of lymphatics or bronchus
-a low pressure vessel

Htn - answer-consistent elevation of systemic arterial bp
-systolic >140
-diastolic > 90

Primary htn - answer-most common, aka essential or idiopathic
-genetic & environmental factors
-sns causes increased hr & vasoconstriction which increase bp
-overactive raas causes na & water retention, increased vascular resistance, insulin
resistance & platelet aggregation
-decreased renal excretion of salt=pressure-natriuresis relationship

Secondary htn - answer-caused my systemic disease process that raises peripheral
vascular resistance or cardiac output
Examples=renal disease, adrenocorticoid tumors, adrenamedullary tumors, or drugs

Complicated htn - answer-chronic damage to blood vessels & tissues leading to target
organ damage in heart, kidneys, brain, & eyes

Malignant htn - answer-htn crisis
-rapidly progressing htn when diastolic is >140
-will cause cerebral encephalopathy & life threatening organ damage if not fixed

Tx of htn - answer-loop or thiazide diuretics, ace or arb inhibitors, ca channel blockers

, -no longer beta blockers b/c they have a higher rate of stroke than those drugs

Orthostatic hypotension - answer-decrease in systolic & distolic bp among standing up
-lack normal bp compensation in response to gravitational changes on the circulation

Acute & chronic orthostatic hypotension - answer-acute=elderly at increased risk b/d
decreased thrist mechanisms. Causes=drugs, prolonged immoblilty, starvation,
exhaustion, dehydration/volume depletion, venous pooling

Chronic-causes=dm, endocrine d/o, metabolic d/o, cns or pns d/o, cerebral infarcts,
parkinsons, autonomic neuropathy=most common cause

Aneurysm - answer-localized dilation or outpouching of a vessel wall or cardiac
chamber
-can be thoracic or abdominal or can occur in cerebral circle of willis
-most common=abdominal
Causes=atherosclerosis or htn
-can lead to aortic dissection or rupture

True aneurysm - answer-involves all 3 layers of wall
-weakening of vessel wall
-can be fisuform or circumferential (circular) or saccular (spherical)

False aneurysm - answer-extravascular hematoma (outside of vessel) that penetrates &
communicated w/intravascular space

Thrombus formation - answer-blood clot that remains attached to the vessel wall
-risk factors=injury/inflammation, obstruction of flow, stasis/pooling
Can have arterial or venous thrombus

Embolism - answer-obstruction of blood flow by a blous of matter circulating in the
bloodstream
-can be dislodged thrombus, air bubble, fat embolism, bacteria, ca cell, or foreign
substance

Thromboangitis obliterans (buergers disease) - answer-inflammatory disease of
peripheral arteries
-strongly associated w/smoking
-formation of thrombi filled w/immune & inflammatory cells
-overtime thrombi become organized & fibrotic leading to permanent occlusion &
obliteration of portions of small & medium sized arteries of feet & hands
-autoimmune d/o

Raynaud phenomenon - answer-episodic vasospasm attacks in small arteries of fingers
& toes
Primary-unknown cause

The benefits of buying summaries with Stuvia:

Guaranteed quality through customer reviews

Guaranteed quality through customer reviews

Stuvia customers have reviewed more than 700,000 summaries. This how you know that you are buying the best documents.

Quick and easy check-out

Quick and easy check-out

You can quickly pay through credit card or Stuvia-credit for the summaries. There is no membership needed.

Focus on what matters

Focus on what matters

Your fellow students write the study notes themselves, which is why the documents are always reliable and up-to-date. This ensures you quickly get to the core!

Frequently asked questions

What do I get when I buy this document?

You get a PDF, available immediately after your purchase. The purchased document is accessible anytime, anywhere and indefinitely through your profile.

Satisfaction guarantee: how does it work?

Our satisfaction guarantee ensures that you always find a study document that suits you well. You fill out a form, and our customer service team takes care of the rest.

Who am I buying these notes from?

Stuvia is a marketplace, so you are not buying this document from us, but from seller biggdreamer. Stuvia facilitates payment to the seller.

Will I be stuck with a subscription?

No, you only buy these notes for $13.49. You're not tied to anything after your purchase.

Can Stuvia be trusted?

4.6 stars on Google & Trustpilot (+1000 reviews)

75632 documents were sold in the last 30 days

Founded in 2010, the go-to place to buy study notes for 14 years now

Start selling
$13.49
  • (0)
  Add to cart