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ARDMS Abdomen Exam Questions And Answers Latest Update 2024/2025 $14.99   Add to cart

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ARDMS Abdomen Exam Questions And Answers Latest Update 2024/2025

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ARDMS Abdomen Exam Questions And Answers Latest Update 2024/2025

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  • September 3, 2024
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ARDMS Abdomen Exam Questions And
Answers Latest Update 2024/2025

Left lobe of the liver is divided into medial and lateral segments by what? Answer: Left hepatic vein



Left lobe of the liver is separated from the caudate lobe by what? Answer: ligamentum venosum



Right lobe of the liver is divided into anterior and posterior segments by what? Answer: Right
hepatic vein



Main lobar fissure Answer: The main lobar fissure separates the right and left lobes of the liver.

It is visualized sonographically as an echogenic linear structure extending from the portal vein to the
neck of the gallbladder

**landmark for identifying the gallbladder



Falciform ligament Answer: Attaches the liver to the anterior abdominal wall

Extends from the diaphragm to the umbilicus

Separate the right and left subphrenic spaces



Ligamentum venosum Answer: The ligamentum venosum is a remnant of the fetal ductus venosus.
It divides the caudate lobe from the left lobe.



Ligamentum teres Answer: Lies within the falciform ligament, remnant of the fetal umbilical
vein***, courses within the left intersegmental fissure, dividing the left lobe into medial and lateral
segments



Main Portal Vein diameter should not exceed: Answer: 13 mm or 1.3 cm

,Hepatomegaly Answer: Etiology: congestive heart failure, inflammatory processes, polycystic
disease, fatty infiltration, biliary obstruction, neoplasm, budd-chiari syndrome

Sonographic characteristics: Length exceeding 18 cm, Anterior-posterior diameter exceeding 15 cm



Alkaline Phosphatase Answer: An enzyme produced primarily by the liver, bone, and placenta and
excreted through the bile ducts

Marked elevation is associated with obstructive jaundice



Alpha-fetoprotein Answer: A protein normally synthesized by the liver, yolk sack, and GI tract of the
fetus

Nonspecific marker for malignancy



Alanine Aminotransferase (ALT) Answer: An enzyme found in large concentration in the liver and
lower concentrations in the heart, muscle, and kidneys

Remains elevated longer than AST

Elevation associated with cirrhosis, hepatitis, and biliary obstruction

Mild elevation associated with liver mets



Aspartate Aminotransferase (AST) Answer: An enzyme present in many types of tissues that is
released when cells are damaged or injured; levels will be proportional to the amount of damage and
time between cell injury and testing

Associated with hepatitis, cirrhosis, and Mononucleosis



Prothrombin time Answer: Normal clotting time is 10-15 seconds

enzyme produced by the liver

production depends on amount of Vitamin K

elevation associated with cirrhosis, malignancy, malabsorption of Vitamin K, and clotting failure

, Decreases with subacute or acute cholecystitis, internal biliary fistula, carcinoma of the GB, injury to the
bile ducts, and prolonged extrahepatic biliary obstruction



Hepatic Candidiasis Answer: Fungal infection

Immune-suppressed patients, Fever, chills, abdominal pain, palpable liver

Uniformly hypoechoic lesions within the liver parenchyma, thick wall margins, hepatomegaly, may
demonstrate a target or "wheel within a wheel," hyperechoic lesions with posterior shadowing



Echinococcal cyst Answer: Parasitic infection (Echinococcus granulosum)

***Sheep farmers

RUQ pain, fever, leukocytosis, hepatomegaly, elevated alkaline phosphatase

Septated cystic mass (honeycomb appearance)

Mobile internal echoes (snowflakes)

Cyst containing small cysts (daughter cysts)



Hepatitis Answer: Fatigue, loss of appetite, fever and chills, nausea, nonobstructive jaundice,
marked elevation in aspartate aminotransferase, alanine aminotransferase and bilirubin

Normal-appearing liver parenchyma

Hypoechoic liver parenchyma

Prominence of the portal veins (star effect)

Hepatomegaly

Splenomegaly

Increased echogenicity in chronic cases



Schistomiasis Answer: Parasite entering the skin or mucosa and traveling to the lung and then liver;
symptoms may take 4-6 weeks to appear, may even take several years to develop

Rash, fever, RUQ pain, diarrhea, lymphadenopathy

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