NCC EFM Need to Know (Fetal Monitoring) Exam Questions with Correct Answers
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Course
NCC Fetal Monitoring
Institution
NCC Fetal Monitoring
Normal arterial blood pH - Answer-> 7.10
Normal arterial blood pH mean value range - Answer-7.2-7.29
Metabolic Acidemia pH - Answer-< 7.10
Respiratory Acidemia pH - Answer-< 7.10
Normal arterial blood PO2 - Answer-> 20
Normal arterial blood PO2 mean value range - Answer-1...
NCC EFM Need to Know (Fetal
Monitoring) Exam Questions with
Correct Answers
Normal arterial blood pH - Answer-> 7.10
Normal arterial blood pH mean value range - Answer-7.2-7.29
Metabolic Acidemia pH - Answer-< 7.10
Respiratory Acidemia pH - Answer-< 7.10
Normal arterial blood PO2 - Answer-> 20
Normal arterial blood PO2 mean value range - Answer-15-24
Metabolic Acidemia PO2 - Answer-< 20
Respiratory Acidemia PO2 - Answer-variable
Normal arterial blood PCO2 - Answer-< 60
Normal arterial blood PCO2 mean value range - Answer-49.2-56.3
Metabolic Acidemia PCO2 - Answer-< 60
Respiratory Acidemia PCO2 - Answer-> 60
Normal arterial blood HCO3 - Answer-> 22
Normal arterial blood HCO3 mean value range - Answer-22-24
Metabolic Acidemia HCO3 - Answer-< 22
Respiratory Acidemia HCO3 - Answer-> 22
Normal arterial blood Base Deficit - Answer-< 12
Normal arterial blood Base Deficit mean value range - Answer-2.7-8.3
Metabolic Acidemia Base Deficit - Answer-> 12
Respiratory Acidemia Base Deficit - Answer-< 12
, Normal arterial blood Base Excess - Answer-> -12
Metabolic Acidemia Base Excess - Answer-< -12
Respiratory Acidemia Base Excess - Answer-> -12
Parasympathetic Nervous System Physiology - Answer--originates in vagus nerve that
extends from the medulla oblongata
-stimulates release of acetylcholine
pathway from transmission of FHR variability
Parasympathetic Nervous System Effect on FHR - Answer--decreases FHR
-increasing gestational age, slow, gradual decrease in FHR
-Moderate variability indicates absence of metabolic acidemia
-modulates baseline FHR with sympathetic branch
Sympathetic Nervous System Physiology - Answer--widely distributed throughout
myocardium
-stimulates release of catecholamines (norepinephrine and epinephrine)
-reserve mechanism that improves heart's pumping ability during intermittent
hypoxemia/stress
-may cause fetal vasoconstriction and hypertension
-blocking with propranolol results in 10bpm decrease in FHR
Sympathetic Nervous System Effect on FHR - Answer--increases FHR
-intermittent hypoxemia causes initial normal fetal compensatory response of FHR
increase or brief tachycardia
-at term, tachycardia is not normal
-in early gestation, SNS dominance results in slightly higher FHR and decreased
variability
Cardiac Output - Answer--Fetal CO is dependent on HR
-CO = HR ~ SV
-small changes in HR have minimal effect on CO
-fetal tachycardia greater than 240 bpm or bradycardia less than 60 bpm may cause a
decrease in fetal CO and umbilical blood flow
Baroreceptors - Answer--protective stretch receptors
-located in aortic arch and carotid sinuses
-when arterial BP increases, baroreceptors detect stretch and send impulses via vagus
nerve to midbrain
-causes an abrupt decrease in FHR, CO, and BP
-causes variable decelerations
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