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NURSING PATHOPHYSIOLOGY FINAL EXAM REVIEW 100+ QUESTIONS AND ANSWERS MODULE4 ||ALREADY GRADED A+ $12.99   Add to cart

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NURSING PATHOPHYSIOLOGY FINAL EXAM REVIEW 100+ QUESTIONS AND ANSWERS MODULE4 ||ALREADY GRADED A+

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NURSING PATHOPHYSIOLOGY FINAL EXAM REVIEW 100+ QUESTIONS AND ANSWERS MODULE4 ||ALREADY GRADED A+ Cellular characteristics: increased macrophages, lymphocytes instead of neutrophils, and fibroblasts Cause of an allergic reaction - ANSWER immune response to exogenous and endogen...

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  • October 3, 2024
  • 14
  • 2023/2024
  • Exam (elaborations)
  • Questions & answers
  • Cardiovascular Ultrasound
  • Cardiovascular Ultrasound
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SmartscoreAaron
NURSING PATHOPHYSIOLOGY FINAL
EXAM REVIEW
100+ QUESTIONS AND ANSWERS
MODULE4 ||ALREADY GRADED A+
Cellular characteristics: increased macrophages, lymphocytes instead of
neutrophils, and fibroblasts

Cause of an allergic reaction - ANSWER ✔ immune response to exogenous and
endogenous antigens that produce inflammation and cause tissue damage

Type I Immediate Hypersensitivity - ANSWER ✔ Reactions begin rapidly, often
within minutes after contact with the allergen; exposure by inhalation, ingestion,
injection, or skin contact. Reactions can range from atopic (local) to anaphylactic
(systemic) depending on portal of entry. IgE mediated response to mast cells
degranulate and release inflammatory mediators.

Type II Antibody Mediated Hypersensitivity - ANSWER ✔ Reaction mediated by
IgG and IgM antibodies binding to normal or altered antigens on cell surface and
connective tissue. Injury to self can result from destruction, inflammation, and cell
dysfunction
-ex: mismatched blood transfusion, hemolytic disease in newborn, and certain drug
reaction

Type III antibody Immune complex mediated hypersensitivity - ANSWER ✔
Antigen-antibody complexes are formed in the blood and get deposited in vessel
lining or tissues which attracts complements (IgG or IgM) and cause an
inflammatory response.
Ex: autoimmune disease, vasculitis, SLE, glomerulonephritis. Reactions range
from atopic to anaphylactic

Type IV T cell mediated Hypersensitivity - ANSWER ✔ Cytotoxic T cell kills
antigen presenting target cell. Helper T cells secrete cytokines who recruit tissue
damaging inflammatory cells
Ex: viral hepatitis TB test

, Opportunist infectious agents - ANSWER ✔ secondary invaders; an immune
deficiency provides the portal
ex. IV hyperailmentation, foley catheter (ecoli, UTI)

Prions - ANSWER ✔ proteins transmitted by injection, transplant, food-Mad Cow
disease

Virion - ANSWER ✔ the infectious particle; viruses are the most frequent cause
of human illness. Capsid-protein based coating

Bacteria - ANSWER ✔ spirochetes and mycoplasmas

Fungal/yeast - ANSWER ✔ live in soil, skin (ringworm), or GI tract

Rickettstiae - ANSWER ✔ transmitted to humans through the bite of the vector.
Rocky mountain spotted fever

Chlamydiae - ANSWER ✔ An elementary body attaches to and enters the host
cells and undergoes replication

Parasites - ANSWER ✔ burrow into skin or mucous membranes, produce an
allergic inflammatory response, release histamine and eosinophilia-kill parasites

Incubation period - ANSWER ✔ the phase during which the pathogen begins
active replication without producing recognizable symptoms in the host

Prodromal stage - ANSWER ✔ Most contagious stage. The initial appearance of
symptoms in the host although the clinical presentation during this time may only
be a vague sense of malaise. May experience mild fever, headache, and fatigue.

Acute stage - ANSWER ✔ Period during which the host experience the max
impact of the infectious process corresponding to rapid proliferation and
dissemination of the pathogen. During this phase, toxic byproducts of microbial
metabolism, cell lysis, and the immune response mounted by the host combines to
produce tissue damage and inflammation

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