100% satisfaction guarantee Immediately available after payment Both online and in PDF No strings attached
logo-home
Summary NURS 172 Unit 2 Study Guide $12.99
Add to cart

Summary

Summary NURS 172 Unit 2 Study Guide

 4 views  0 purchase

This is a comprehensive and detailed study guide on unit 2 for Nurs 172. *Essential Study Material!!

Preview 3 out of 17  pages

  • October 6, 2024
  • 17
  • 2021/2022
  • Summary
All documents for this subject (11)
avatar-seller
anyiamgeorge19
Unit 2 exam study guide – circulation and perfusion
*friends note: some things not included b/c I already understand the concept. Everything on here is
things I need reminding of or have no idea wtf it is.

Week 5-
Wilkinson p.490-99:
Pulmonary circulation: O2 depleted blood goes from heart to lungs, becomes oxygenated by
alveoli then goes back to heart.
Systemic circulation: L ventricle pumps blood via arterial system
* Coronary circulation: circulates blood through heart (part of systemic circulation)
Capillaries: Arteries that are subdivided into tissues and organs. How O2 goes into tissues.
Venous system: collects O2 depleted blood and returns it to R atrium to begin circulation again
Diastole: relaxation (filling) DUB systole: contraction (emptying) LUB
Pulmonary arteries O2 depleted blood from R ventricle to lungs
Systemic arteries O2 enriched blood from L ventricle to body periphery
Pulmonary veins O2 enriched blood from lungs to L atrium
Systemic veins O2 depleted blood from periphery to R atrium


JVD- R side of heart is congested due to inadequate pump function (see more detail later)
Wilkinson p.955-89
R (lung) lobe=3 L (lung) lobe=2
Pathway of airflow: nosenasal cavity
pharynxlarynxtracheabronchusbronchiolesalveoliblood (back to alveoli, then
bronchioles, etc.*process reverses)
Ventilation: physical breathing respiration: gas exchange (O2+CO2)
External respiration Internal respiration
O2- diffuses across alveolar capillary membrane into O2- diffuses from blood through capillary cellular
blood of pulmonary capillaries membrane into tissue cells (used for metabolism)

CO2- diffuses out of blood into alveoli to be exhaled CO2- waste product, transported to lungs and exhaled.
(Capillary membrane blood  lungs  out)


TABLE 36-1 (Wilkinson p. 964)  respiratory medications to be reviewed
Kussmaul’s respirations: regular, but increased in rate and abnormally deep respirations (think of
as a form of hyperventilation)

,Biots respirations: irregular respirations of variable depth (usually shallow), alternating with
periods of apnea
Cheyne- Stroke: gradual increase in depth, then decrease in depth, then apnea.
Orthopnea: difficulty breathing laying down.
Stridor: high pitched, harsh, crowing- PARTIAL OBSTRUCTION of larynx/trachea. Hear w/o
stethoscope. (MORE SOUNDS FOUND HOFFMAN P.450)
ABG-P.977 wilkinson (to be reviewed later) – see hoffman notes below
Hoffman 439-56
Perfusion: oxygenated blood moving into tissues.
Acid base balance: exchange of CO2 for O2 in lungs + renal secretion of HCO3 (bicarb)
maintain body ph between 7.35-7.45
Surfactant: substance secreted by alveoli that prevent collapse (atelectasis)
Visceral pleura: thin membrane covering lungs Parietal pleura: membrane covering chest cavity
Pleural space: space between two membranes (V & P) has fluid that acts as a lubricant that
allows lungs to move freely as ventilation occurs.
Phrenic nerve- responsible for movement/contraction of diaphragm
Shunt: blood that returns to L heart without being oxygenated. can occur as result of atelectasis
or collapsed alveoli. 2 types:
Anatomical: blood moving from R to L heart w/o traveling through lungs.
physiological: alveoli are not functioning (returns to L heart w/o being oxygenated)
Crepitus: palpation finding. Air trapped under the skin – “Crackling feeling”. Can be caused by
pneumothorax or chest trauma.
Respiratory alkalosis Respiratory acidosis
Ph: >7.45 Ph: <7.35
PaCO2: <35mmHg PaCO2: >45mmHg
Associations: Associations:
Hyperventilation, Anxiety, +/- early asthma or COPD, Pneumonia, Respiratory failure
pneumonia
>7.45 alkalosis <7.35 acidosis PaCO2: 35-45 mmhg PaO2: 80-95 mmhg HCO3: 22-26meq/l
Diagnostic studies (ABG above):
Pulse ox: utilizes wave lengths to measure saturation of hemoglobin with O2
Pulse OX: 91-94%: mild hypoxemia, 86-90%: moderate hypoxemia, <85%: severe hypoxemia
Capnography: monitors the PaCO2 in airway during inhalation and exhalation- provides written
tracing.

, Capnometry: measures amount of CO2 exhaled w/o continuous tracing.
Sputum analysis: check for microorganisms +/- abnormal cell growth.
Pulmonary function test: evaluate lung volume to determine lung function.
Bronchoscopy: direct visualization of the respiratory tract down to secondary bronchi.
*needs to be NPO 8 hours prior for sedation. May get tissue specimens.
Asses for bleeding/hypoxia after procedure.
Thoracentesis: diagnostic/treatment (depending). Needle inserted into pleural space to remove
excess fluid/air.
*tell pt to deep breathe, cough. Nurse monitor vitals, SPO2, lung sounds. Pt report
elevated HR, palpitations, SOB/Dyspnea, angina, hemoptysis to MD STAT.
Lung biopsy: small piece of lung tissue to be removed & analyzed under microscope
*same nursing implications as bronchoscopy and thoracentesis!!


Week 6-
Hoffman p.460-75
LOWER AIRWAY DISORDERS
Alveolar macrophages: eat the bad things in the alveoli via phagocytosis
Mucociliary elevator: sneeze/cough to get bad debris out of respiratory tract.
Colonization: small number of bacteria not causing infection of resident bacteria and some
viruses *ex. staphylococcus, streptococcus
Bactericidal effect: surface epithelium secrete proteins to kill bacteria.
Influenza A, B, C: highly contagious, droplet, rapidly spreads, fomite transfer (objects)
Incubation period: 18-72 hours. Shedding: 2-5 days. INFECTIOUS for 7-10 days.
Severity of symptoms are dependent on number of viruses shed during replication phase.
Diagnostic: gold standard= sample respiratory secretions for viral culture (can take up to 10 days
for results). Common test= rapid flu test (RIDTs), swab test, results less then 30 min – not
always accurate
Tx/prevention: flu vaccine, medications use symptomatically. Severe cases: antivirals (does not
cure, only reduces symptoms)
*Primary influenza viral pneumonia= least common severest complication. Affects
mostly >65yrs. Progressive SOB, persistent fever, and cardiovascular compromise.
p. 464 hoffman for interventions/teaching

The benefits of buying summaries with Stuvia:

Guaranteed quality through customer reviews

Guaranteed quality through customer reviews

Stuvia customers have reviewed more than 700,000 summaries. This how you know that you are buying the best documents.

Quick and easy check-out

Quick and easy check-out

You can quickly pay through credit card or Stuvia-credit for the summaries. There is no membership needed.

Focus on what matters

Focus on what matters

Your fellow students write the study notes themselves, which is why the documents are always reliable and up-to-date. This ensures you quickly get to the core!

Frequently asked questions

What do I get when I buy this document?

You get a PDF, available immediately after your purchase. The purchased document is accessible anytime, anywhere and indefinitely through your profile.

Satisfaction guarantee: how does it work?

Our satisfaction guarantee ensures that you always find a study document that suits you well. You fill out a form, and our customer service team takes care of the rest.

Who am I buying these notes from?

Stuvia is a marketplace, so you are not buying this document from us, but from seller anyiamgeorge19. Stuvia facilitates payment to the seller.

Will I be stuck with a subscription?

No, you only buy these notes for $12.99. You're not tied to anything after your purchase.

Can Stuvia be trusted?

4.6 stars on Google & Trustpilot (+1000 reviews)

50843 documents were sold in the last 30 days

Founded in 2010, the go-to place to buy study notes for 14 years now

Start selling
$12.99
  • (0)
Add to cart
Added