TAMU NURS 617 EXAM 2 Questions and Correct Answers the Latest Update
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Course
NURS 617
Institution
NURS 617
During the cardiac cycle, which structure directly delivers action potential to
the ventricular myocardium?
a. Sinoatrial (SA) node
b. Atrioventricular (AV) node
c. Purkinje fibers
d. Bundle branches
c. Purkinje fibers
Each cardiac action potential travels from the SA node to the AV no...
TAMU NURS 617 EXAM 2 Questions and
Correct Answers the Latest Update
During the cardiac cycle, which structure directly delivers action potential to
the ventricular myocardium?
a. Sinoatrial (SA) node
b. Atrioventricular (AV) node
c. Purkinje fibers
d. Bundle branches
✓ c. Purkinje fibers
✓
✓ Each cardiac action potential travels from the SA node to the AV node to the bundle
of His (AV bundle), through the bundle branches, and finally to the Purkinje fibers
and the ventricular myocardium, where the impulse is stopped.
The cardiac electrical impulse normally begins spontaneously in the sinoatrial
(SA) node because it:
a. Has a superior location in the right atrium.
b. Is the only area of the heart capable of spontaneous depolarization.
c. Has rich sympathetic innervation via the vagus nerve.
d. Depolarizes more rapidly than other automatic cells of the heart
✓ d. Depolarizes more rapidly than other automatic cells of the heart
✓
✓ The electrical impulse normally begins in the SA node because its cells depolarize
more rapidly than other automatic cells.
What can shorten the conduction time of action potential through the
atrioventricular (AV) node?
a. Parasympathetic nervous system
b. Catecholamines
c. Vagal stimulation
d. Sinoatrial node (SA)
✓ b. Catecholamines
✓
✓ Catecholamines speed the heart rate, shorten the conduction time through the AV
node, and increase the rhythmicity of the AV pacemaker fibers.
If the sinoatrial (SA) node fails, then at what rate (depolarizations per minute)
can the atrioventricular (AV) node depolarize?
a. 60 to 70
b. 40 to 60
c. 30 to 40
d. 10 to 20
✓ b. 40 to 60
✓
✓ If the SA node is damaged, then the AV node will become the heart's pacemaker at
a rate of approximately 40 to 60 spontaneous depolarizations per minute.
Within a physiologic range, what does an increase in left ventricular end-
diastolic volume (preload) result in?
a. Increase in force of contraction
b. Decrease in refractory time
c. Increase in afterload
d. Decrease in repolarization
✓ a. Increase in force of contraction
✓
✓ This concept is expressed in the Frank-Starling law; the cardiac muscle, like other
muscles, increases its strength of contraction when it is stretched.
As stated in the Frank-Starling law, a direct relationship exists between the
_____ of the blood in the heart at the end of diastole and the _____ of
contraction during the next systole.
a. Pressure; force
b. Volume; strength
c. Viscosity; force
d. Viscosity; strength
✓ b. Volume; strength
✓
✓ As stated in the Frank-Starling law, the volume of blood in the heart at the end of
diastole (the length of its muscle fibers) is directly related to the force (strength) of
contraction during the next systole.
What physical sign is the result of turbulent blood flow through a vessel?
a. Increased blood pressure during periods of stress b. Bounding pulse felt on
palpation
c. Cyanosis observed on excretion
d. Murmur heard on auscultation
✓ d. Murmur heard on auscultation
✓
✓ Where flow is obstructed, the vessel turns or blood flows over rough surfaces. The
flow becomes turbulent with whorls or eddy currents that produce noise, causing a
murmur to be heard on auscultation, such as occurs during blood pressure
measurement with a sphygomanometer.
What is the initiating event that leads to the development of atherosclerosis?
a. Release of the inflammatory cytokines
b. Macrophages adhere to vessel walls.
c. Injury to the endothelial cells that line the artery walls
d. Release of the platelet-deprived growth factor
✓ c. Injury to the endothelial cells that line the artery walls
✓
✓ Atherosclerosis begins with an injury to the endothelial cells that line the arterial
walls. Possible causes of endothelial injury include the common risk factors for
atherosclerosis, such as smoking, hypertension, diabetes, increased levels of low-
density lipoprotein (LDL), decreased levels of high-density lipoprotein (HDL), and
autoimmunity.
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