Exam (elaborations)
CSC Study Questions with Correct Answers 100% Pass
CSC Study Questions with Correct
Answers 100% Pass
Following surgical repair of a thoracic aneurysm with an endoluminal graft, the patient is unable to move
his lower extremities. The nurse should first
a. Activate stroke team and prepare to do an urgent (STAT) computed tomography (CT) scan
b....
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CSC Study Questions with Correct
Answers 100% Pass
Following surgical repair of a thoracic aneurysm with an endoluminal graft, the patient is unable to move
his lower extremities. The nurse should first
a. Activate stroke team and prepare to do an urgent (STAT) computed tomography (CT) scan
b. Prepare to return to surgery for exploration of femoral artery occlusion
c. Call surgeon to obtain a neurology consultation in the morning
d. Prepare for lumbar drain insertion to remove cerebrospinal fluid (CSF) - ANSWER✔✔-D
Spinal cord ischemia is a complication from thoracic aneurysm repair for both open and endoluminal
repair. Spinal cord ischemia results from increased cerebral spinal pressure that compresses the spinal
nerves. Untreated spinal cord ischemia can lead to paraplegia. It is important to recognize the signs of
spinal cord ischemia (loss of lower extremity movement) promptly. Immediate insertion of a spinal drain
can reverse the spinal cord ischemia and prevent paraplegia. Bilateral leg paralysis is not typically an
initial symptom of stroke (A). Signs of femoral artery occlusion (B) are the 5 Ps: pulselessness, pallor,
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pain, paresthesias, and paralysis. These symptoms would be unilateral, not bilateral. Immediate insertion
of a spinal drain is needed; waiting until morning for the consultation (C) would be inappropriate.
A postoperative patient who had undergone coronary artery bypass graft and ventricular aneurysm
repair has a 15-beat run of ventricular tachycardia. The most likely cause of the dysrhythmia is
a. Irritability of the ventricle from the aneurysm repair
b. Spasm of the right coronary artery graft
c. Potassium 4.8 mEq/dL and magnesium 2.1 mEq/L
d. Did not restart administration of the angiotensinconverting enzyme (ACE) inhibitor (taken
preoperatively) - ANSWER✔✔-A
Patients with left ventricular aneurysm typically have depressed left ventricular (LV) function. Depressed
LV function may lead to increased ventricular arrhythmias. LV aneurysm repair is indicated to improve
symptoms of angina, heart failure, systemic thromboembolism, or malignant arrhythmias. In the
immediate postoperative phase, the repaired ventricle continues to be depressed and has the added
trauma of surgery on the left ventricle. Both of these increase the irritability of the LV, leading to
ventricular arrhythmias. Spasm (B) or occlusion of the RCA leads to bradyarrhythmias, not ventricular
arrhythmias. The potassium and magnesium levels in (C) are normal. Low potassium and magnesium
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levels increase the risk for ventricular arrhythmias. ACE inhibitors (D) do not have any antiarrhythmic
effect.
The nurse admits a patient after aortic valve replacement and notes the following settings of the
temporary pacemaker: DDD rate, 80/min; atrial MA, 10; ventricular MA, 4. The underlying rhythm is
complete heart block with ventricular escape rhythm rate 30/min. The most likely cause of the complete
heart block is
a. Spasm of the right coronary artery (RCA) graft
b. Damage of the atrioventricular (AV) node during repair of the aortic valve
c. Low potassium and magnesium levels
d. Toxic effects of β-blocker - ANSWER✔✔-B
The AV node and the bundle of His are near the aortic valve. During aortic valve replacement,
hemorrhage, edema, suturing, or debridement near the AV node and the bundle of His may cause heart
blocks. Typically the epicardial pacing is only needed for a few days until the edema resolves. If complete
heart block persists after a few days, a permanent pacemaker may be required. The RCA supplies oxygen
to the sinoatrial (SA) and AV nodes and spasm of the RCA graft (A) may cause bradycardia and/or heart
blocks. This patient did not have bypass surgery, so RCA spasm would not be a postoperative
complication. Low potassium and magnesium levels (C) increase the risk for ventricular arrhythmias, not
AV conduction defects. If toxic effects of β-blockers (D) were present, the complete heart block would
have been the underlying rhythm preoperatively.
A postoperative coronary artery bypass graft and aortic valve replacement patient has been in a normal
sinus rhythm for 4 hours. The monitor is now showing P waves at a rate of 73 beats per minute with no
ventricular response. The best action would be to
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a. Administer atropine 0.5 mg intravenous bolus (IV push)
b. Connect transcutaneous pacing pads to patient
c. Connect epicardial pacing wires to a temporary pacemaker
d. Administer epinephrine 1 mg IV push and start epinephrine infusion - ANSWER✔✔-C
The AV node and the bundle of His are near the aortic valve. During aortic valve replacement,
hemorrhage, edema, suturing, or debridement near the AV node and the bundle of His may cause heart
blocks. Pacing is needed to treat the conduction defect caused by the surgery. Atropine (A) and
epinephrine (D) will not work because the conduction problem is with the AV node and/or the bundle of
His. Transcutaneous (external) pacing (B) would be the next best option if epicardial wires were not
present. The heart rate should be greater than 45/min and less than 80/min.
One hour after extubation, a diabetic coronary artery bypass surgery patient is becoming slightly
lethargic. Arterial blood gas (ABG) analysis yielded the following results: pH, 7.33; PaO2, 80 mm Hg;
PaCO2, 50 mm Hg; SaO2, 95%; HCO3, 28 mEq/L; base excess, 0.5. The nurse should first
a. Obtain a blood glucose level
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