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NUR 206 Exam 3 Study Guide with complete solutions |Latest 2024/2025- Fortis.

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Stenosis vs. Regurgitation *Stenosis* •narrowing of valve or artery *Regurgitation* •backflow of blood into atria during ventricular systole Bipolar Comorbidities •anxiety •ADHD •personality disorder •CVD •DM2 •family hx •drug use •high stress •major life-alt...

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  • November 12, 2024
  • 39
  • 2024/2025
  • Exam (elaborations)
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NUR 206 Exam 3 Study Guide with complete
i.- i.- i.- i.- i.- i.- i.- i.-




solutions- Fortis. i.- i.-




Stenosis vs. Regurgitation
i.- i.- i.-i.- i.- *Stenosis*
•narrowing of valve or artery i.- i.- i.- i.-




*Regurgitation*
•backflow of blood into atria during ventricular systole
i.- i.- i.- i.- i.- i.- i.-




Bipolar Comorbidities
i.- i.-i.- i.- •anxiety
•ADHD
•personality disorder i.-




•CVD
•DM2
•family hx i.-




•drug usei.-




•high stress
i.-




•major life-altering event
i.- i.-




Bipolar Cyclical Features
i.- i.- i.-i.- i.- *Rapid Cycling*i.-




•4 episodes w/in 1 year w/ >2mo of partial or full remission
i.- i.- i.- i.- i.- i.- i.- i.- i.- i.- i.-




*Cyclothymic Disorder* i.-

,•when pts sustain ≥ 2 years of chronic fluctuating mood disturbance
i.- i.- i.- i.- i.- i.- i.- i.- i.- i.-




Conditions that Lead to Valvular Heart Disease
i.- i.- i.- i.- i.- i.- i.-i.- i.- •congenital
•valvular stenosis i.-




•valvular insufficiency i.-




•HTN
•age
•atherosclerosis
•infection
•MI


Clinical Manifestations of Valvular Heart Disease
i.- i.- i.- i.- i.- i.-i.- i.- •SOB i.-



(mitral/aortic)
•weakness/lightheadedness (mitral/aortic) i.-




•chest discomfort (any?)
i.- i.-




•edema of LE (tricuspid/pulmonic)
i.- i.- i.-




•palpitations (any?) i.-




•rapid weight gain (tricuspid/pulmonic)
i.- i.- i.-




Cardiomyopathy i.-i.- i.- •disease that affects heart muscle's ability to
i.- i.- i.- i.- i.- i.- i.-



pump effectively
i.- i.-

,•primary abnormality of heart muscle that affects it's structural or
i.- i.- i.- i.- i.- i.- i.- i.- i.- i.-



functional characteristics i.-




Dilated Cardiomyopathy
i.- i.-i.- i.- •most common i.-




•inherited (1/3) i.-




•chemical exposure i.-




•common occurrence in 20-60 yo i.- i.- i.- i.-




•men > women
i.- i.-




•muscle becomes thinner and weaker as it dilates
i.- i.- i.- i.- i.- i.- i.-




•SOB, fatigue, LE edema, JVD
i.- i.- i.- i.-




Hypertrophic Cardiomyopathy (HCM) i.- i.- i.-i.- i.- •↓ compliance of LV and
i.- i.- i.- i.- i.-



hypertrophy
•usually inherited i.-




•also sec to HTN
i.- i.- i.-




•muscle mass enlarges, leading to small LV volume
i.- i.- i.- i.- i.- i.- i.-




•septal hypertrophy may inhibit outflow
i.- i.- i.- i.-




Restrictive Cardiomyopathy (RCM) •rigid ventricular walls that
i.- i.- i.-i.- i.- i.- i.- i.- i.-



restrict heart's ability to stretch and fill w/ blood
i.- i.- i.- i.- i.- i.- i.- i.-




•usually acquired as result of amyloidosis (build up of protein)
i.- i.- i.- i.- i.- i.- i.- i.- i.-




•↓ compliance (expansion)
i.- i.-




•diastolic dysfunction and HF i.- i.- i.-

, Arrythmogenic Right Ventricular Dysplasia (ARVD) i.-•↑ risk of i.- i.- i.- i.-i.- i.- i.- i.- i.-



ventricular dysrhythmias sec to thickening of cardiac muscle
i.- i.- i.- i.- i.- i.- i.-




•usually hereditary
i.-




•can cause sudden cardiac death in young adults and athletes
i.- i.- i.- i.- i.- i.- i.- i.- i.-




Unclassified Cardiomyopathy i.- i.-i.- i.- •peripartum cardiomyopathy (PPCM) i.- i.-




•rare but serious
i.- i.-




•usually seen in last months of pregnancy or up to 5 months PP
i.- i.- i.- i.- i.- i.- i.- i.- i.- i.- i.- i.-




•risk factors = multiple pregnancies, obesity, poor nutrition, HTN, some
i.- i.- i.- i.- i.- i.- i.- i.- i.- i.-



meds


Valvular Heart Disease Treatment and Interventions
i.- i.- i.- i.- i.- i.-i.- i.- *Medication*
•BB
•ACE inhibitors
i.-




•nitrates
•diuretics
•calcium channel blockers
i.- i.-




•statins
•hydrazeline
*Interventions*
•low Na+ diet
i.- i.-

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