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NSG 530 Exam: Comprehensive Review with Graded Questions and Detailed Explanations for Advanced Pathophysiology and Clinical Nursing Practice Preparation.$25.99
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NSG 530 Exam: Comprehensive Review with Graded Questions and Detailed Explanations for Advanced Pathophysiology and Clinical Nursing Practice Preparation.
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Course
NSG 530
Institution
NSG 530
NSG 530 Exam: Comprehensive Review with Graded Questions and Detailed Explanations for Advanced Pathophysiology and Clinical Nursing Practice Preparation.
Advanced Pathophysiology 530 - Exam 3, Advanced
Pathophysiology Exam 3, NSG 530 Exam 3
Guaranteed to Improve Your Academic
Performance Designed for Achievement in Every
Exam
elevated high sensitivity c reactive protein and CAD (hs-CRP) - CORRECT ANSWER- -acute
phase reactant or protein
made in liver
indirect measure of atherosclerotic plaque-related inflammation/progression
inflammatory marker
the more inflammation the more likely to have plaque ruptures
t/f lipoproteins increase risk for a cardiac event, thrombus, and stroke - CORRECT
ANSWER- -true
t/f high numbers of large and puffy LDL particles are associated with increased risk for CAD
- CORRECT ANSWER- -false
high numbers of small dense LDL
total cholesterol levels - CORRECT ANSWER- -desirable - <200
response to injury hypothesis - CORRECT ANSWER- -Atherosclerosis hypothesis where
plaque build up begins with the endothelial damage
changes in permeability
monocytes attach
move in
release oxygen free radicals
LDL becomes oxidized
monocyte becomes macrophage
cell dies
dead cells and oxidized LDLs form the plaque
cover by smooth muscle cell
fatty streak - CORRECT ANSWER- -first lesion of atherosclerosis
-thickening of intima
-inc. in smooth muscle cells
, -smooth muscle cells migrate and proliferate into intima
-lipid deposits accumulate
-macrophages cause damage
fibrous plaque - CORRECT ANSWER- -second lesion of atherosclerosis
-evolves from fatty streak
-accumulation of connective tissue
-inc. in number of smooth muscle cells
-deeper lipid pool
-results in further endothelial cell dysfunction, necrosis, narrowing lumen
-development of fibrous cap
advanced (complicated) lesion - CORRECT ANSWER- --elevated and protrudes into lumen
-smooth muscle, macrophages, t cells, associated with lipid core and necrotic material
-covered by fibrous cap as it protrudes into lumen
-thick cap - provides stability
-thin cap - macrophage rich - lesion is unsatlbe
-inflammation
thrombosis, hemorrhage, calcification
-rupture
stable plaque - CORRECT ANSWER- -graudally increases in size and may partially occlude
the lumen, limiting flow, causing ischemia
exists in the wall, attached
thick cap
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