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NURS 5315 Endocrine Exam Questions Answers
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NURS 5315 Endocrine Exam Questions Answers
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NURS 5315 Endocrine Exam QuestionsAnswers
Thyroid iihormones ii- iiAnswers ii-T3, iiT4 iiand iicalcitonin
Pancreas iihormones ii- iiAnswers ii-Insulin iiand iiglucagon
Adrenal iiGlands iihormones ii- iiAnswers ii-Made iiup iiof iithe iicortex iiwhich iisecretes
iisteroids iisuch iias iicortisone iiand iialdosterone iiand iithe iimedulla iiwhich iisecretes
iicatecholamines iisuch iias iiepi iiand iinorepi
Primary iithyroid iidisorders ii- iiAnswers ii-result iiin iialterations iiof iithyroid iihormone ii(TH)
iilevels iiwith iisecondary iifeedback iieffects iion iipituitary iithyroid iistimulating iihormone
ii(TSH)
Subclinical iiThyroid iidisease ii- iiAnswers ii-Thyroid iidisease iithat iipresents iiwith iiminimal
iito iino iisymptoms iibut iiwith iiabnormal iilab iivalues
Secondary iiThyroid iidisorders ii- iiAnswers ii-conditions iithat iiresults iifrom iithe
iidysfunction iiof iithe iipituitary iigland iiTSH iiproduction
Thyrotoxicosis ii- iiAnswers ii-(hyperthyroidism) iia iicondition iithat iiresults iifrom iiany iicause
iiof iiincreased iiTH iilevels. iiWill iihow iilow iiTSH iilevels iiand iihigh iiT4 iilevel. iiS&S:
iiincreased iimetabolic iirate, iiheat iiintolerance, iigoiter, iimenstrual iiirrregularities, iiweight
iiloss, iidiaphoresis, iifine iitremor, iitachycardia, iifrequent iibowel iimovements,
iirestlessness, iishort iiattention iispan, iihair iiloss, iianorexia, iiexophthalmos, iipretibial
iiedema, iiand iiheart iifailure.
Thyrotoxic iiCrisis(thyroid iistorm) ii- iiAnswers ii-worsening iihyperthyroid iistate iitriggered
iiby iian iiigniting iieven iisuch iias iiinfection, iitrauma, iicardiopulmonary iidisorder, iiburns,
iiseizures iisurgery, iior iispontaneously. iiS&S: iiextreme iirestlessness iiand iiagitation,
iidelirium, iiseizures, iicoma, iisevere iitachycardia, iiheart iifailure, iihyperthermia, iidelirium,
iivolume iidepletion, iiNVD iiand iideath iiif iinot iitreated.
Grave's iidisease ii- iiAnswers ii-Most iicommon iicause iiof iihyperthyroidism iiand iiis iian
iiautoimmune iidisorder. iiAntibodies iiattach iito iithe iithyroid iicells iiand iimimic iithe iifunction
iiof iiTSH iiwhich iiresults iiin iian iiincreased iisecretion iiof iiT3 iiand iiT4 iiand iioverrides iithe
iinegative iifeedback iimechanisms iiwhich iiregulate iiTSH iisecretion. iiThe iistimulation iiof
iithe iireceptors iiby iithe iiantibodies iiresults iiin iithe iidevelopment iiof iigoiter. iiMay iialso
iiexperience iiexophthalmos, iiperiorbital iiedema, iiand iiextraocular iimuscle iiweakness
iileading iito iistrabismus iiand iidiplopia
, Hyperthyroidism iifrom iinodular iithyroid iidisease ii- iiAnswers ii-Follicular iihypertrophy iiof
iithe iithyroid iicells iiis iiresponsible iifor iithe iiformation iiof iithe iithyroid iinodules iiwhich
iisecrete iiextra iihormones. iiNodules iidevelop iibc iiof iinormal iichanges iiduring iipregnancy
iior iipuberty iior iias iia iiresult iiof iian iiautoimmune iiissue, iiviral iiinfection iior iigenetic
iiinfluence. iiSymptoms iidevelop iislowly iiand iiwill iinot iidisplay iiexophthalmos iior iipretibial
iimyxedema
Primary iihypothyroidism ii- iiAnswers ii-Defect iiis iiin iithe iithyroid iigland iiitself iiwhich
iicauses iiinsufficient iiamounts iiof iithyroid iihormone. iiCauses iiinclude iicongenital
iidefects, iithyroidectomy, iithyroid iiradiation, iiiodine iideficiency, iianti-thyroid iimedications,
iior iiimpairment iiin iithyroid iihormone iisynthesis
Secondary iihypothyroidism ii- iiAnswers ii-Malfunction iiin iithe iipituitary iior iihypothalamus
iiglands iiwhich iileads iito iia iilack iiof iiTSH. iiMost iicommon iicause iiis iipituitary iitumors.
iiOther iicauses iiinclude iiTBI, iisubarachnoid iihemorrhage, iior iipituitary iiinfarction
Subclinical iihypothyroidism ii- iiAnswers ii-mild iithyroid iifailure. iidefined iiby iielevated iiTSH
iilevel iiwith iinormal iiTH iilevel.
Hypothyroid iiS&S ii- iiAnswers ii-confusion, iisyncope, iislow iispeech iiand iithinking,
iianemia, iibradycardia, iireduced iistroke iivolume iiand iicardiac iioutput, iidyspnea,
iihypoventilation, iidecreased iiappetite, iiweight iigain, iidry iihair, iicold iiintolerant,
iiconstipation, iihyperlipidemia, iiperiorbital iiedema, iiperipheral iiedema, iimyxedema(puffy
iiface), iiincreased iitotal iibody iiwater, iihyponatremia, iireduced iirenal iiblood iiflow
Myxedema iicoma ii- iiAnswers ii-thyroid iiemergency iiwhich iihas iithe iiopposite iieffect iiof
iithyroid iistorm. iiResults iiin iidecreased iiLOC iiand iiis iiusually iiprecipitated iiby iian iievent
iisuch iias iiinfection, iidiscontinuation iiof iithyroid iimedications, iinarcotic iior iisedative iiuse.
iiother iiS&S iihypotension, iihypoventilation, iishivering, iihypothermia, iilactic iiacidosis,
iicoma, iiand iihypoglycemia.
Type ii1 iiDM ii- iiAnswers ii-3 iitypes: ii1A iiis iiautoimmune, ii1B iiis iiidiopathic iiand ii3c iiis
iiassociated iiwith iichronic iipancreatitis. iiin ii1A: iiautoimmune iiresponse iidestroys iithe
iibeta iicells iiin iithe iipancreas iiwhich iileads iito iiapoptosis. iiBeta iicell iidestruction iiis iiwhat
iicauses iia iilack iiof iiinsulin iito iibe iiproduced.
Type ii1 iiclinical iimanifestations ii- iiAnswers ii-polydipsia, iipolyuria, iipolyphagia, iiweight
iiloss iiand iifatigue.
Typer ii2 iiDM ii- iiAnswers ii-pathlogical iidefect iiis iiinsulin iiresistance. iiA iidecrease iiin
iinumber iiof iiinsulin iicell iireceptors iior iiinsufficient iiamounts iiof iiinsulin iisecretion iito
iimeet iimetabolic iineeds iiare iicharacteristic iiof iiDM ii2.
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