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NR507 Advanced Pathophysiology NR 507 Advanced Pathophysiology week 7 Final Quiz Review with Correct Answers | 100% Pass Guaranteed | Graded A+ | $14.69
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NR507 Advanced Pathophysiology NR 507 Advanced Pathophysiology week 7 Final Quiz Review with Correct Answers | 100% Pass Guaranteed | Graded A+ |

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NR507 Advanced Pathophysiology NR 507 Advanced Pathophysiology week 7 Final Quiz Review with Correct Answers | 100% Pass Guaranteed | Graded A+ |

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  • November 21, 2024
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NR507 Advanced Pathophysiology
NR 507 Midterm and Final Exam

Course Title and Number: NR507 Advanced Pathophysiology
Exam
Exam Title: NR 507 Exam
Exam Date: Exam 2024- 2025
Instructor: [Insert Instructor’s Name]
Student Name: [Insert Student’s Name]
Student ID: [Insert Student ID]

Examination
180 minutes
Instructions:
1. Read each question carefully.
2. Answer all questions.
3. Use the provided answer sheet to mark your responses.
4. Ensure all answers are final before submitting the exam.
5. Please answer each question below and click Submit when you have completed
the Exam.
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7. This is Exam which will assess your knowledge on the course Learning
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NR507 Advanced Pathophysiology 2024-2025
NR 507 Advanced Pathophysiology week 7 Final Quiz
Review with Correct Answers | 100% Pass Guaranteed |
Graded A+ |
Read All Instructions Carefully and Answer All the
Questions Correctly Good Luck: -

Gout - =Answer>> Inflammatory response to
excessive quantities of uric acid in the blood and other
body fluids, including the synovial fluid

Cause of pain in gout - =Answer>> Elevated uric acid
levels lead to formation of monosodium urate (MSU)
crystals in and around joints and eventually deposited
in connective tissues throughout the body-triggers
release of chemokines and interleukins—crystallization
in synovial fluid causes acute painful inflammation of
the joint which triggers macrophages-prolonged
accumulation results in joint damage

gouty arthritis - =Answer>> Prolonged accumulation
of uric acid resulting in joint damage

degenerative joint disease - =Answer>> characterized
by the wearing away of the articular cartilage within
the joints

osteophytosis - =Answer>> new bone formation of
joint margins

Primary pathogenesis of OA - =Answer>>
Degeneration and eventual loss and disordered repair
of articular cartilage-chondrocytes become damaged
earlier in disease process due to atypical load bearing
paired with genetic/epi genetic and biochemical factors
which lead to increased remodeling of articular

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