Rasmussen University
(Minnesota)
NUR2063 Essentials of Pathophysiology
NUR2063 Patho Exam 1, 2 and Final Exams
Course Title and Number: Patient Care Technician Certification
(PCT) DaVita Exam
Exam Title: PCT DaVita ExamExam
Exam Date: Exam 2024- 2025
Instructor: [Insert Instructor’s Name]
Student Name: [Insert Student’s Name]
Student ID: [Insert Student ID]
Examination
180 minutes
Instructions:
1. Read each question carefully.
2. Answer all questions.
3. Use the provided answer sheet to mark your responses.
4. Ensure all answers are final before submitting the exam.
5. Please answer each question below and click Submit when you have completed
the Exam.
6. This test has a time limit, The test will save and submit automatically when the
time expires
7. This is Exam which will assess your knowledge on the course Learning
Resources.
Good Luck!
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NUR2063 Essentials of Pathophysiology (Rasmussen)
2025- 2026
NUR2063 Final Exam Prep Study Guide Questions and
Answers | 100% Pass Guaranteed | Graded A+ |
Read All Instructions Carefully and Answer All the
Questions Correctly Good Luck: -
hyperparathyroidism and etiology - =Answer>>
condition of excessive PTH production by parathyroid
glands
causes: tumors, hyperplasia, chronic hypocalcemia
(renal failure)
hyperparathyroidism manifestations/treatment -
=Answer>> osteoporosis, renal calculi, polyuria, abd.
pain, constipation, fatigue, weakness, flaccid muscles,
dysrhythmias, hypertension, depression, forgetfulness,
Cushing's syndrome - =Answer>> excessive cortisol
that results from the increased ACTH levels
Cushing's syndrome etiology - =Answer>> iatrogenic
from ingestion of glucocorticoid meds, adrenal tumors
that secrete glucocorticoids, pituitary tumors that
secrete ACTH and cortisol, and paraneoplastic
syndrome (rare disorders triggered by abnormal
immune response to a cancerous tumor called
neoplasm)
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Cushing's syndrome treatment - =Answer>> gradual
tapering of any glucocorticoids
tumors may need surgery and radiation
meds to control cortisol production
interventions to manage complications
Cushing's manifestations - =Answer>> moon face,
obesity, buffalo hump, muscle weakness/wasting,
delayed growth, acne, broad purple striae, thin
bruisable skin, delayed healing, hyperpigmentation
(due to ACTH), increased infections, osteoporosis,
hirsutism (abnormail hair growth), glucose intolerance,
hypertension, dyslipidemia, edema, hypokalemia, mood
changes
Growth Hormone (GH) - =Answer>> stimulates cell
growth and fat breakdown. primary targets are muscle
and bone, where GH stimulates amino acid uptake and
protein synthesis.
Anterior Pituitary gland
Type 1 diabetes mellitus, etiology - =Answer>>
develops when body's immune system destroys
pancreatic beta cells.
etiology-unknown, most likely viral or environmental
trigger in genetically susceptible people.
manifestations of diabetes mellitus - =Answer>>
polyuria, polydipsia, polyphagia
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hyperglycemia, glucosuria, weight loss, blurred vision,
fatigue
hypoglycemia manifestations - =Answer>> weak,
double or blurred vision, hunger, tachycardia,
palpitations
hyperglycemia manifestations - =Answer>> -urinary
frequency, anorexia, dry mouth
long term complications of diabetes mellitus -
=Answer>> hyperglycemia, DKA, hypoglycemia, heart
disease, stroke, HTN, diabetic retinopathy, blindness,
kidney disease, amputations, periodontal disease, preg
complications, increase susceptibility to infections,
erectile dysfunction.
high blood glucose can harden vasculature
Type 2 diabetes causes - =Answer>> starts out as
insulin resistance. ofter overweight
treatment for diabetes mellitus - =Answer>> Type 1
Insulin therapy
Type 2
Lifestyle changes
Oral drug therapy
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