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Test Bank For Goulds Pathophysiology for the Health Professions 7th Edition by VanMeter and Hubert 9780323792882 Chapter 1-28 All Chapters with Answers and Rationals $17.99
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Test Bank For Goulds Pathophysiology for the Health Professions 7th Edition by VanMeter and Hubert 9780323792882 Chapter 1-28 All Chapters with Answers and Rationals

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Test Bank For Goulds Pathophysiology for the Health Professions 7th Edition by VanMeter and Hubert 9780323792882 Chapter 1-28 All Chapters with Answers and Rationals

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  • January 22, 2025
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  • Gould's Pathophysiology For The Health Profession
  • Gould's Pathophysiology For The Health Profession
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Test Bank For Gould's Pathophysiology for the Health
cr cr cr cr cr cr cr




Professions 7th Edition VanMeter
cr cr cr cr

, 1

Chapter 01: Introduction to Pathophysiology
cr cr cr cr


VanMeter and Hubert: Gould’s Pathophysiology for the Health Professions, 7th
cr cr cr cr cr cr cr cr cr


Edition
cr




MULTIPLE crCHOICE

1. Which crof crthe crfollowing crwould crbe crthe crmost crlikelycrcause crof
an criatrogenic crdisease?
cr

a. An crinherited crdisorder
b. A crcombination crof crspecific cretiological crfactors
c. An crunwanted creffect crof cra crprescribed crdrug
d. Prolonged crexposure crto crtoxic crchemicals crin crthe crenvironment
ANS: c r C

2. The crmanifestations crof cra crdisease crare crbest crdefined cras crthe
a. subjective crfeelings crof crdiscomfort crduring cra crchronic crillness.
b. signs crand crsymptoms crof cra crdisease.
c. factors crthat crprecipitate cran cracute crepisode crof cra crchronic crillness.
d. earlycrindicators crof crthe crprodromal crstage crof crinfection.
ANS: c r B

3. The crbest crdefinition crof crthe crterm crprognosis cris crthe
a. precipitating crfactors crcausing cran cracute crepisode.
b. number crof crremissions crto crbe crexpected crduring crthe crcourse crof cra crchronic crillness.
c. predicted croutcome cror crlikelihood crof crrecovery crfrom cra crspecific crdisease.
d. exacerbations croccurring crduring crchronic crillness.
ANS: c r C

4. Which crof crthe crfollowing cris crconsidered cra crsystemic crsign crof crdisease?
a. Swelling crof crthe crknee
b. Fever
c. Pain crin crthe crneck
d. Red crrash cron crthe crface
ANS: c r B

5. Etiologycris crdefined cras crthe crstudycrof crthe
a. causes crof cra crdisease.
b. course crof cra crdisease.
c. expected crcomplications crof cra crdisease.
d. manifestations crof cra crdisease.
ANS: c r A

6. A crtype crof crcellular cradaptation crin crwhich crthere cris cra crdecrease crin crcell crsize cris crreferred crto cras
a. hypertrophy.
b. metaplasia.
c. anaplasia.
d. atrophy.

ANS: c r D

7. A crchange crin cra crtissue crmarked crby crcells crthat crvary crin crsize crand crshape crand crshow crincreased
cr mitotic crfigures crwould crbe crcalled

,Test crBank crFor crGould's crPathophysiology crfor crthe crHealth crProfessions cr7th crEdition 2
VanMeter

a. metaplasia.
b. atrophy.
c. dysplasia.
d. hypertrophy.
ANS: c r C

8. A crdeficit crof croxygen crin crthe crcells crusually crdue crto crrespiratory cror crcirculatory crproblems cris crcalled
a. apoptosis.
b. ischemia.
c. hypertrophy.
d. necrosis.
ANS: c r B

9. When cra crgroup crof crcells crin crthe crbodycrdies, crthe crchange cris crcalled
a. ischemia.
b. gangrene.
c. hypoxia.
d. necrosis.
ANS: c r D

10. Rigorous crweightlifting/body crbuilding crregimens crmay crresult crin crthe crskeletal crmuscle
cells crundergoing
cr

a. hypertrophy.
b. dysplasia.
c. atrophy.
d. regeneration.
ANS: c r A

11. The crterm crcancer crrefers crto
a. dysplasia.
b. hyperplasia.
c. metaplasia.
d. malignant crneoplasm.
ANS: c r D

12. To crwhich crof crthe crfollowing crdoes crthe crterm crapoptosis crrefer?
a. Increased crrate crof crmitosis crbycrcertain crcells
b. Ischemic crdamage crto crcells
c. Liquefaction crof crnecrotic crtissue
d. Preprogrammed crcell crself-destruction
ANS: c r D
13. Which crof crthe crfollowing crstatements cris crTRUE?
a. Alteration crof crDNA crdoes crnot crchange crcell crfunction.
b. Damaged crcells crmaycrbe crable crto crrepair crthemselves.
c. All crtypes crof crcells crdie crat crthe crsame crrate.
d. Mild crischemia crcauses crimmediate crcell crdeath.
ANS: c r B

14. Caseation crnecrosis crrefers crto cran crarea crwhere
a. cell crproteins crhave crbeen crdenatured.

, Test crBank crFor crGould's crPathophysiology crfor crthe crHealth crProfessions cr7th crEdition 3
VanMeter

b. cells crare crliquefied crbycrenzymes.
c. dead crcells crform cra crthick crcheesycrsubstance.
d. bacterial crinvasion crhas croccurred.
ANS: c r C

15. Routine crapplication crof crsun crblock crto crskin crwould crbe cran crexample crof
a. an criatrogenic crcause crof crcancer.
b. a crpreventive crmeasure.
c. a crprecipitating crfactor.
d. a crpredisposing crcondition.
ANS: c r B

16. A crcircumstance crthat crcauses cra crsudden cracute crepisode crof cra crchronic crdisease crto croccur cris crtermed
a. latent crstage.
b. predisposing crfactor.
c. incidence.
d. precipitating cr factor.
ANS: c r D

17. The crterm crhomeostasis crrefers crto
a. the crcausative crfactors crin cra crparticular crdisease.
b. maintenance crof cra crstable crinternal crenvironment.
c. a crcondition crthat crtriggers cran cracute crepisode.
d. a crcollection crof crsigns crand crsymptoms.
ANS: c r B

18. Which crterm cris crused crto crdescribe cra crnew crand crsecondary cror cradditional crproblem crthat crarises
after crthe croriginal crdisease crhas crbeen crestablished?
cr

a. Symptoms
b. Occurrence
c. Manifestations
d. Complication
ANS: c r D

19. Pathophysiologycrinvolves crthe crstudycrof
a. the crstructure crof crthe crhuman crbody.
b. the crfunctions crof crvarious crorgans crin crthe crbody.
c. functional cror crstructural crchanges crresulting crfrom crdisease crprocesses.
d. various crcell crstructures crand crrelated crfunctions.
ANS: c r C

20. Which crof crthe crfollowing cris crthe crbest crdefinition crof crepidemiology?
a. The crscience crof crtracking crthe croccurrence crand crdistribution crof crdiseases
b. The crrelative crnumber crof crdeaths crresulting crfrom cra crparticular crdisease
c. Identification crof cra crspecific crdisease crthrough crevaluation crof crsigns crand crsymptoms
d. The crglobal crsearch crfor cremerging crdiseases
ANS: c r A

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