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Summary Histology Rapid Revision Krok 1

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  • Course
  • KROK 1, USMLE Step 1
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  • KROK 1, USMLE Step 1

In this file you will find a Rapid Revision Summary for the Histology part by Medic Mukul of Krok 1. A part of the summary is based on most frequent asked questions as well as ministery questions.

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Document information

  • May 2, 2023
  • 7
  • 2022/2023
  • Summary

Subjects

  • krok 1
  • usmle step 1
  • krok 1
  • ua medical exam
  • ukraine medical exam
  • krok histology summary
  • krok histology base
  • krok rapid revision histology

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  • KROK 1, USMLE Step 1
All documents for this subject (9)

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Histology
Rapid Revision KROK




Minna Mamik
@ Medic Mukul

, induced immune
Drug
hemolytic anomia
flare
(Cytexicsensitivityi
wheal a
a




S
Hypersensitivity -> Y
dypes
response
I
Type I
->
Anaphylatic, Immediate, Reagine
a
Adopy hemolytic disease in newban

by Rh-incompatibility
Cytotoxic) Type II -

Antibody Mediated, Complement
dependent
Immuno Complex
Type III ->
Hypersensitivity Thymus ->
Produce thymasin
Hasall's Capuscles present all).
Type E ->
Delayed a T-cell mediated
hypersensitively (degenerated
will shinkover deme
↳ & inducation
erythrema >Digeoge Syndrome -




thymosin not produced
>Undergo Age involution

of allengen with Absence Di-Geage Syndrome (Chromosome
Type I interaction
-




IgE, absorbed on -
22 defect)
(is-30 min)
mastocytes and basophils membeanes
↳
e.g bronchial astma, after
acine introduction
Red Bone Marrow ->
Produce RBC
↑



Type II
- Fixadion of alien
antigens fixed on the cells
Ezydheopoeitin
N
actson Redbone.

(B -

cell by prefamed andibodies with the subsequent kidney produce
mediated)
activation of complement
system by the cell-bound Aputation -> Removal of agans

Immune complexes ->
e.g bloodteansfusion, Rh phantom pain Amputadion

Type III -> Formation of cell-bound immune complexes causalgic pain -> Gun shot


fixing ondo tissues &
producing their
damage protopathic pain -> Glossitis, inflammation
->
e.g Systemic lupus eythomedus, Glomoulonephitis
(SLE)
Cellular Ganuloma of
Type
immune Combination bact &
-> response, Delayedin maccophage
-




T-cell=> > CM1
Granuloma
type It
hypes.
->

e.g
Lapzosy "/Sandwich
palisade Azragment
->

Mantcuxtes fatB
-




->




↳ Geanuloma cell's

(at
Poikilocytosis -


Change shape of RBC Tuberculosis -


Langerhans all skinbarrie)
dencritic alls

Anisocytosis ->
change Size of RBC
lepzosy - Vizchow
call/cigarete-like cells (LoVe)
Rhinoschecoma -
Mikuliz cell

Gumma Bodies
Immune
agains
-> Central &
Peripheral Syphilis
-




↓ *
(hymosin) thymus Tonsils -



B-lymp
(RBC&WBC) Red Bane Marrow Lymph noce

splee langs -
3
types of Cell

Antigen Indipendent) Antigen dependent) pneumocytes/ Prumocytes/ pneumocytes/
Alvedlan cell Alseolar Cell Alvedan Maczophage
Tonsils -
Only lymphoid organ
with epithelial lining Type I Type I
single

~ Nasopharynged ↳
Tonsils; ↳ Simple difussion produce Surfactant
surface
engulffonign body
Tonsil -numerous
cryptspresent (Gas exchange) to reduce
↳


ETabralin
Tubal Acts
Tension as
phagocytosis
Waldeyer's
Tonsil >
hymphocytes arranged act as precuse prent
lungs from
paired het / as
lymphatic nodules fa type II
collapsing Emphysema
↳ Prevent
tendency to ↳ macrophage comes
paladine
Tonsil Tonsil
recede
Ccollapse)
~gese
& breaks walls,

baggy lungs,
Hal/gastzomucin alvedi damaged
Lymph noce ->>Bean shaped >Follicles in Contex
nodles - proten,producus
Medullary cads present aka-lymphatic
parital/oxyntic cell

2
It
Trabecal > cards in medulla
Rods
(duilight vision) contains maximum


Spleen -> Cential
artery Spleen consist of parts2 Alveolar
dype II ~ Mitochondria

↳> white pulp-ecentrically
tiven-> Central rein
placed acteride

LoVe ↳ Red pulp-made of
sinusoids & cads


Minna Mamik

, o Surfactant
Composition; o
Eyelens from Surface Ectodum * Redina from Estodem


Dipalmitoylphosphatidycholine (Dppc) chaloid ->
fom Mesodem


Sphingolipids S ↳ have blood veins


phospholipids CECell
Gganelles
lecithin Endoplasmic Redicutum

Ca2t SmoodhER
Rough ER
or




Sant Agranular
Glanclar


A
protein
W




B
Ribosomes(granules) pt.
Function
↓
Ribosome absent

Function:
Protein Synthesis lipid Synthesis
hydrophillic
-




hydrophobic Hydrophilic staage of Cant
#ate
water Detofication of Daugs
BK Be careful

Golgi Apparatus
St
Capillaries 1) post - translational modification
Non-continuous/Sinusoidal 2)
Continuous Fenestzated
sarding proteins
↳ NO Gaps, Nopores ↳ small Gaps ↳ Wider Gaps ↳
having (is &tzans end
kidney Red Bone Marrow
Brain
e.g eg he e.g;
lungs
Sheletal M.
I Liver

Spleen
↓RBC
WBC
-Expating & famation function
Smooth M. absorption platelets
ect,

hysosomes -> Suicide Bag/Residual Bodies

St Articles--Resistance Vessel Function:Destruction of unwanted Subst, bact, etc
Veins- Capacitance ↳ (produce hydedytic enzymes)
act as
phagocytosis
Lymphatic Capillaries (NoBasement or thin
-




Macula Densa ~membrane penoxisomes -> Destruction of te radicals by Catalase
↳ H202
Oxidation of of FA
long & very long
->
chain


St Ammonification. NHy is
dangerous in beain, heart. Cytoskeleton ->
Provides stength &steucture to cell
Muscles
bone of cell)
Brain, heart ↳ Microdubules -



Big filaments
Transport of NH, of NH3
Transport ↳ Intermediate filament -> in
most quanities present
by Glutamate
↓Livert by Alanine
↳ Microfilament -
Small filaments
lots of H20 NHs
↓
both
used fa Ureacycle -> in
cytoplasm
&
excretion of ammonia ↓ mitochondria 6 Epidhelium
wied

↓ simple epith. ->
Single layered
Albumin
most in
kidney Compound epith ->
Multilayered
Blood lange
~In quanidy
et
Collagen -> MostAbundant in
protein body .
Simple epithstium
Bone, Skin, Tendon epith
Type I Simple Squamos 3B- A (mnemonic
-> ->




if absent Osteogenesis imperfecta Bowman's capsule
-
, -




Blood vessels
Cardilage
-


Type II -


>Body Cavity
if absent Achondroplasia
>
> -> Alvedi


Type III - Redicular fibers, Blood Vessels, Aata -Simple Cuboidal epith. -> T-ok
Like Cube II
Ellen Danlos
Syndrome -
Thyzoid Follides

I -> Basement
Membrane ·7777, Germinal Epith. of Ovary
Type
-AlportSynchrome -
kidney Tubules


Minna Mamik poT & DCT (
proximal & Distal
convoluted Tubules

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