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NUR 265 Exam 3 # 273 Questions & Answers: Latest Updated A+ Score Solution.
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NUR 265 Exam 3 # 273 Questions &Answers: Latest Updated A+ Score
Solution.
What causes Autonomic Dysreflexia - -Stimulation at level of T6
or above
-What are some examples of triggers of autonomic dysreflexia -
-restrictive clothing; full bladder/neurogenic bladder; fecal
impaction; directive pressure s/a sitting in the wheel chair
-signs and systems of Autonomic Dysreflexia - -hypertension,
flushed face, headaches, JVD, bradycardic, diaphoresis, pale ext
below the level of T6, nausea, dilated pupils, blurred vision,
restlessness
-What is the purpose of fluid resuscitation for a burn victim - -
maintain vital organ perfusion, reduce edema, minimize effects
of fluid shifts, prevent hypovolemic shock
-What IV solution is commonly used to resuscitate a pt with a
burn - -Lactated ringers
-Chemical burns should be irrigated until - -20 minutes or the
burn sensation continues after the 20 minute marker
-what would you use to remove hot tar or asphalt - -citrus
petroleum jelly ex; medisol
petroleum jelly
antibiotic ointment
-what kind of brain injury would you expect if an adult client is
positive for Palmer's infant reflexes - -cortical and premotor
cortex damage
-what kind of brain injury would you expect if an adult client is
positive for plantar infant reflexes - -upper motor neuron lesion
, -what kind of brain injury would you expect if an adult client is
positive for rooting infant reflex - -frontal lobe damage
-what kind of brain injury would you expect if an adult client is
positive for sucking infant reflex - -Advance dementia; cortical
brain damage
-what kind of brain injury would you expect if an adult client is
positive for glabella (persistent blinking) infant reflex - -diffuse
cortical dysfunction
-What is the consensus formula for burns - -2-4 ml X TBSA X KG
-What S&S are expected for a burn client who is receiving the
first 8 hours of fluid resuscitation - -Restlessness, anxiety,
Hypothermia
-how much fluid replacement are you going to give the first 8
hours - -1/2 of the fluid consensus
-What do you need to monitor when resuscitating fluids for burn
pt to make sure that it is working - -Urine output
-Besides a hyperbaric chamber how would you admin o2 to a
client with CO poisoning - -100% O2 with a non-rebreather
-How would you treat a circumferential trunk burn that is
swelling and why? - -Eschartomies R/T constriction of the chest
wall expansion
-what are the classification of shock - -Cardiogenic;
hypovolemic, neurogenic, and Disruptive
-All shock is caused by - -inadequate tissue perfusion
-Patho of hypovolemic shock - -Inadequate circulating blood
volume S/A burns, hemorrhage, dehydration
-Patho for cardiogenic shock - -Inadequate pumping action of
the heart S/A MI, CHF, PE
, -What are the 3 subclasses of Distributive shock - -Anaphylactic;
Septic; Neurogenic
-Patho for neurogenic shock - -interference of the nervous
system that controls the blood vessels
-Patho for septic shock - -Release of vasoactive substance from
the immune system
-How much blood loss is required for the patient to be at high
risk for hypovolemic shock - -15-25%, or 1/3 of the body blood,
or 5L
-clients who experience slow blood loss can - -Tolerate the blood
loss better then a client with rapid blood loss
-Signs and symptoms of compensatory shock - -hypotension,
tachycardia, tachypnea, hypothermia, decrease pulse pressure
-During compensatory stage of shock, why would you hear
hypoactive bowl sounds and cool and clammy skin - -Body
shunting blood from skin, kidneys and GI to provide adequate
blood volume to the brain and heart
-Why would urine output decrease during compensatory stage of
shock - -High production of aldosterone
-why do RR increase in compensatory stage of shock - -The body
is going into acidotic state trying to blow it off Kussmals
-What is the byproduct of anaerobic metabolism that is
developing in what stage of shock - -Lactic acid, Compensatory
-Why would you not give someone with impaired hepatic
function lactated ringers - -Because it does not convert lactic
acid into bicarb fast enough could go into acidosis
-why would you check for high levels of sodium nd glucose in the
compensatory stage of shock - -Because of the release of
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