100% satisfaction guarantee Immediately available after payment Both online and in PDF No strings attached
logo-home
Previously searched by you
Previously searched by you
logo
NURSING NSG5003 Week 6 Mid-term $7.99   Add to cart
Quickly navigate to
Preview
  2.  
  3. NURSING NSG5003
  4.  
  5. NURSING NSG5003

Exam (elaborations)

NURSING NSG5003 Week 6 Mid-term
 20 views  0 purchase
  • Course
  • NURSING NSG5003
  • Institution
  • NURSING NSG5003

NURSING NSG5003 Week 6 Mid-term Why does tissue damage occurs in acute rejection after organ transplantation? a. Th1 cells release cytokines that activate infiltrating macrophages, and cytotoxic T cells directly attack the endothelial cells of the transplanted tissue. b. Circulating immune comp...

[Show more]

Preview 2 out of 10  pages

Document information

  • January 23, 2024
  • 10
  • 2023/2024
  • Exam (elaborations)
  • Questions & answers

Subjects

  • nursing nsg5003 week 6 mid term why does tissue d

Written for

  • NURSING NSG5003
All documents for this subject (21)

Seller

avatar-seller
NurseAdvocate

Reviews received

Content preview

NURSING NSG5003 Week 6 Mid-term
These are all from the test bank for the textbook.

What causes the rapid change in the resting membrane potential to initiate an action potential?

a. Potassium gates open, and potassium rushes into the cell, changing the membrane potential
from negative to positive.

b. Sodium gates open, and sodium rushes into the cell, changing the membrane potential from
negative to positive.

c. Sodium gates close, allowing potassium into the cell to change the membrane potential from
positive to negative.

d. Potassium gates close, allowing sodium into the cell to change the membrane potential from
positive to negative.



What is a consequence of leakage of lysosomal enzymes during chemical injury?

a. Enzymatic digestion of the nucleus and nucleolus occurs, halting DNA synthesis.

b. Influx of potassium ions into the mitochondria occurs, halting the ATP production.

c. Edema of the Golgi body occurs, preventing the transport of proteins out of the cell.

d. Shift of calcium out of the plasma membrane occurs, destroying the cytoskeleton.



In hypoxic injury, sodium enters the cell and causes swelling because:

a. The cell membrane permeability increases for sodium during periods of hypoxia.

b. ATP is insufficient to maintain the pump that keeps sodium out of the cell.

c. The lactic acid produced by the hypoxia binds with sodium in the cell.

d. Sodium cannot be transported to the cell membrane during hypoxia.



What mechanisms occur in the liver cells as a result of lipid accumulation?

a. Accumulation of lipids that obstruct the common bile duct, preventing flow of

bile from the liver to the gallbladder

b. Increased synthesis of triglycerides from fatty acids and decreased synthesis of apoproteins

c. Increased binding of lipids with apoproteins to form lipoproteins

d. Increased conversion of fatty acids to phospholipids

, During an IgE-mediated hypersensitivity reaction, the degranulation of mast cells is a result of which
receptor action?

a. Histamine bound to H2

b. Chemotactic factor binding to the receptor

c. Epinephrine bound to mast cells

d. Acetylcholine bound to mast cells



What is the mechanism that results in type II hypersensitivity reactions?

a. Antibodies coat mast cells by binding to receptors that signal its degranulation, followed by
a discharge of preformed mediators.

b. Antibodies bind to soluble antigens that were released into body fluids, and the immune
complexes are then deposited in the tissues.

c. Cytotoxic T lymphocytes or lymphokine-producing helper T 1 cells directly attack and destroy
cellular targets.

d. Antibodies bind to the antigens on the cell surface



Type III hypersensitivity reactions are a result of which of the following?

a. Antibodies coating mast cells by binding to receptors that signal its degranulation, followed by
the discharge of preformed mediators

b. Antibodies binding to soluble antigens that were released into body fluids and the immune
complexes being deposited in the tissues

c. Tc cells or lymphokine-producing Th1 cells directly attacking and destroying cellular targets

d. Antibodies binding to the antigen on the cell surface.



Tissue damage caused by the deposition of circulating immune complexes containing an antibody
against the host DNA is the cause of which disease?

a. Hemolytic anemia

b. Pernicious anemia

c. Systemic lupus erythematosus

d. Myasthenia gravis

The benefits of buying summaries with Stuvia:

Guaranteed quality through customer reviews

Guaranteed quality through customer reviews

Stuvia customers have reviewed more than 700,000 summaries. This how you know that you are buying the best documents.

Quick and easy check-out

Quick and easy check-out

You can quickly pay through credit card or Stuvia-credit for the summaries. There is no membership needed.

Focus on what matters

Focus on what matters

Your fellow students write the study notes themselves, which is why the documents are always reliable and up-to-date. This ensures you quickly get to the core!

Frequently asked questions

What do I get when I buy this document?

You get a PDF, available immediately after your purchase. The purchased document is accessible anytime, anywhere and indefinitely through your profile.

Satisfaction guarantee: how does it work?

Our satisfaction guarantee ensures that you always find a study document that suits you well. You fill out a form, and our customer service team takes care of the rest.

Who am I buying these notes from?

Stuvia is a marketplace, so you are not buying this document from us, but from seller NurseAdvocate. Stuvia facilitates payment to the seller.

Will I be stuck with a subscription?

No, you only buy these notes for $7.99. You're not tied to anything after your purchase.

Can Stuvia be trusted?

4.6 stars on Google & Trustpilot (+1000 reviews)

67096 documents were sold in the last 30 days

Founded in 2010, the go-to place to buy study notes for 14 years now

Start selling
$7.99
  • (0)
  Add to cart