100% satisfaction guarantee Immediately available after payment Both online and in PDF No strings attached
logo-home
NURS 5315 GI Module 9, exam 5 already graded A+ 2024/2025 $12.99   Add to cart

Exam (elaborations)

NURS 5315 GI Module 9, exam 5 already graded A+ 2024/2025

 75 views  2 purchases

NURS 5315 GI Module 9, exam 5 already graded A+ 2024/2025

Preview 2 out of 8  pages

  • January 28, 2024
  • 8
  • 2023/2024
  • Exam (elaborations)
  • Questions & answers
  • nurs 5315
All documents for this subject (204)
avatar-seller
Ashley96
NURS 5315 GI Module 9, exam 5

Upper GI organs - ANSmouth,pharynx, esophagus, stomach, and duodenum

Lower GI organs - ANSsmall intestine, large intestine, rectum and anus

Hepatoportal circulation - ANShepatic artery receives oxygenated blood from the inferior
mesenteric, gastric, and cystic veins. The hepatic portal vein receives deoxygenated blood from
the inferior and superior mesenteric vein and splenic vein and delivers nutrients that have been
absorbed from the intestinal system

Osmotic diarrhea - ANSCaused by the presence of a nonabsorbable substance in the
intestines. This pulls water by osmosis into the intestinal lumen and results in large volume
diarrhea. This is how mag citrate, lactulose and miralax work. Causes include: excessive
ingestion of nonabsorbable sugars, tube feedings, dumping syndrome, malabsorption,
pancreatic enzyme deficiency, bile salt deficiency, small intestine bacterial overgrowth or celiac
disease

Secretory diarrhea - ANSResults in large volume losses secondary to infectious causes such as
rotavirus, bacterial enterotoxins, or c-diff.

Motility diarrhea - ANSAKA short bowel syndrome. Results from resection of small intestine or
surgical bypass of small intestine, IBS, diabetic neuropathy, hyperthyroidism, and laxative
abuse. Fatty stools and bloating are common in malabsorption syndrome. Complications
include: dehydration, electrolyte imbalance, metabolic acidosis, weight loss and malabsorption.

Upper GI bleed - ANSbleeding that occurs in the esophagus, stomach or duodenum commonly
caused by bleeding varices, peptic ulcers or Mallory-Weiss tear(tearing of esophagus from
stomach) Characterized by frank, bright red or coffee ground emesis.

Lower GI bleed - ANSBleeding in the jejunum, ileum, colon or rectum from inflammatory bowel
disease, cancer, diverticula or hemorrhoids. Hematochezia, or the presence of bright red blood
in the stools, suggest what kind of bleed

Peptic Ulcer Disease - ANSIs a break in the integrity of the mucosa of the esophagus, stomach
or duodenum resulting in exposure of the tissue to gastric acid. Risk factors include smoking,
advanced age, NSAID use, ETOH, chronic disease, acute pancreatitis, COPD, obesity,
socioeconomic status, gastrinoma, and infection with Helicobacter pylori. S&S: Epigastric pain is
worse with eating, melena or hematemesis

Duodenal ulcers - ANSmost common and tend to develop in younger patients. S&S: epigastric
pain that is relieved by food. Patients may have melena(black and tarry stool) or hematemesis

, Ulcerative colitis (UC) - ANSInflammatory disease of the large instestine in persons 20-40y/o.
Less common in people who smoke. Has periods of remission and exacerbations.
Characterized by inflammation and ulcerations that remain superficial and in the small intestine.

UC S&S - ANSrecurrent diarrhea, bloody stools, febrile, polyarthritis, uveitis, sclerosing
cholangitis, erythema nodosum and pyoderma gangrenosum

UC complications - ANSfissures, hemorrhoids, perirectal abscess, toxic megacolon, colon
perforation, and colorectal adenocarcinoma. Increased risk of VTE and microthrombi, and colon
cancer

Crohn's disease - ANSChronic inflammatory disorders that can affect any portion of the GI tract
but most often in the ileum and proximal colon. Affects persons in their 20-30s and of jewish
decent. CARD15/NOD2 gene mutation commonly associated.

Crohn's disease risk factors - ANSsmoking, family history, Jewish decent, age less than 40,
slight predominance in women and altered gut microbiome.

Crohn's disease patho - ANSincludes trasmural involvement of the affected area(entire wall of
intestine is affected) and the presence of skip lesions. Disease progression may lead to abscess
formation in GI tract. Possible causes include infectious agents, autoimmune, psychosomatic,
impaired T-cell immunity

Crohn's disease S&S - ANSabd pain, diarrhea, dehydration, bloody stools, malabsorption,
malnutrition, weight loss, intestinal obstruction from chronic inflammation, fistulas and
perforation of the intestine

Diverticular disease - ANSCharacterized as the presence of diverticula in the large intestine.
Risk factors include older age, genetic predisposition, obesity, smoking, diet, lack of exercise,
ASA and other NSAIDS, altered DI microbiome and abnormal colonic peristalsis

Diverticulosis/Diverticulitis - ANSoutpouchings of mucosa from the muscle layer of the intestine
that protrude into the intestinal lumen most commonly in the sigmoid colon. Diverticulosis is the
presence of diverticula in an asymptomatic person. Diverticulitis is an inflammation of diverticula
and cause LLQ pain. Results in abscess formation, rupture and peritonitis

Appendicitis - ANSInflammation of the appendix usually in persons 10-19. S&S: periumbilical
pain, RLQ pain, nvd, anorexia. Pain may initially be epigastric or periumbilical then settle in
RLQ. Perforation, peritonitis and abscess formation are all potential complications

Small bowel obstruction - ANSMost commonly caused by postop adhesions, tumors, Crohn's
disease, hernias and intussusception. Causes distention 2ndary to impaired absorption and

The benefits of buying summaries with Stuvia:

Guaranteed quality through customer reviews

Guaranteed quality through customer reviews

Stuvia customers have reviewed more than 700,000 summaries. This how you know that you are buying the best documents.

Quick and easy check-out

Quick and easy check-out

You can quickly pay through credit card or Stuvia-credit for the summaries. There is no membership needed.

Focus on what matters

Focus on what matters

Your fellow students write the study notes themselves, which is why the documents are always reliable and up-to-date. This ensures you quickly get to the core!

Frequently asked questions

What do I get when I buy this document?

You get a PDF, available immediately after your purchase. The purchased document is accessible anytime, anywhere and indefinitely through your profile.

Satisfaction guarantee: how does it work?

Our satisfaction guarantee ensures that you always find a study document that suits you well. You fill out a form, and our customer service team takes care of the rest.

Who am I buying these notes from?

Stuvia is a marketplace, so you are not buying this document from us, but from seller Ashley96. Stuvia facilitates payment to the seller.

Will I be stuck with a subscription?

No, you only buy these notes for $12.99. You're not tied to anything after your purchase.

Can Stuvia be trusted?

4.6 stars on Google & Trustpilot (+1000 reviews)

83637 documents were sold in the last 30 days

Founded in 2010, the go-to place to buy study notes for 14 years now

Start selling
$12.99  2x  sold
  • (0)
  Add to cart