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CCRN (CRITICAL CARE NURSING) CERTIFICATION EXAM 2024/2025 | 3 VERSIONS | LATEST UPDATED WITH STUDY GUIDE, COMMON TERMINOLOGIES AND QUANTIFIERS FOR KAPLAN $20.49   Add to cart

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CCRN (CRITICAL CARE NURSING) CERTIFICATION EXAM 2024/2025 | 3 VERSIONS | LATEST UPDATED WITH STUDY GUIDE, COMMON TERMINOLOGIES AND QUANTIFIERS FOR KAPLAN

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CCRN (CRITICAL CARE NURSING) CERTIFICATION EXAM 2024/2025 | 3 VERSIONS | LATEST UPDATED WITH STUDY GUIDE, COMMON TERMINOLOGIES AND QUANTIFIERS FOR KAPLAN

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  • August 6, 2024
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  • 2024/2025
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CCRN (CRITICAL CARE NURSING)
CERTIFICATION EXAM 2024/2025 | 3 VERSIONS | LATEST
UPDATED
WITH STUDY GUIDE, COMMON TERMINOLOGIES AND
QUANTIFIERS FOR KAPLAN
SIADH

too much water, dilutional hyponatremia. Decreased osmolarity=hypoosmolar. Decreased urinary
output.

CSF normal protein, glucose, WBCs, specific gravity,

Protein <100, Glucose: 70 WBCs: 4 cells/mm2 Specific gravity 1.007

Poikothermia

fluctuation of core body temperature of more than 2° C due to changes in ambient room
temperature

pathophysiology of a seizure

neurons in the cerebral cortex fire at the same time in a paroxysmal burst.
System driven outcome

include length of stay, readmission rate, and resource utilization.

Arterial supply to the brain: vertebrobasilar, common carotid, meningeal arteries

The vertebrobasilar arteries supply the posterior portion of the brain. The common carotid
arteries supply the anterior area of the brain. The meningeal arteries supply the superior portion
of the brain.

Pheochromocytoma

adrenal medulla,hi epi/norepi. s/s: hypertension, sweating, headache, palpitations, apprehension,
nausea/vomiting, tremor, pallor, abdominal pain, chest pain, and hyperglycemia.

Acute radiation syndrome

large doses of ionizing radiation , Circulatory collapse, increased intracranial pressure, vasculitis,
and meningitis causing death within 3 days

Complications of SIADH

seizure activity

,Treatment of SIADH (avoid what solutions?)

Fluid restriction
3% nacl (1500 osmolarity over 25cc/hr or less)
Dont do hypotonic solutions!
Asses for fluid overload

hypertonic solutions

D5LR; D5 1/2 NS; D5NS

hypotonic solutions

0.5% NS (HNS or 0.45% NS); 2.5% dextrose in 0.45% NS (D2.5 45% NS)

Osmolality and Sodium

275-295= normal osmolality. Sodium=135-145. Usually 2X of Na

Causes of SIADH
Viral PNA
Oat cell carcinoma
Head problems
Increased serum osmolality
Anesthesia and analgesics
Stress

Diabetes insipidus (urine specific gravity?)

No ADH, can't keep water, increased UOP. Hypernatremic, hyperosmolar, increased urinary
output (6-24L a day of clear urine)
urine specific gravity 1.001-1.005
Severe hypovolemia

Causes of diabetes insipidus (what medication?)
Head problem
Dilantin (DI)
Treatment of diabetes insipidus (medication, fluid, monitoring x2)

Pitressin/vasopressin (same as ADH)
Give fluids (increase intravascular volume)
Monitor urine specific gravity
EKG monitor for ischemia

Hypoglycemia s/s

, Tachycardia, palpitations, diaphoresis, irritable, restlessness
Confusion, lethargy, slurred speech, seizure, coma, death. IF YOU ARE IN A BETA
ADRENERGIC BLOCKER, you only see the CNS symptoms

DKA (BS, breathing, acid vs K)

Blood Sugar 400 to 900, Dehydration, No insulin, Ketones, Kussmaul breathing
Whenever high acid =hi K. For every drop of 0.1 in pH =increase by 0.6 of K

HHNK (who gets, BS, breaths)

old age, diet controlled diabetics, TPN patients, who get a lot of inteavascular sugar, and
pancreatitis as pancreas is eating itself, does not work properly.
Blood sugar 1000-2000, severe dehydration, (6 to 10 Liters behind.
Patient still makes insulin, so it can occur over months, preventing the breakdown of fats which
causes no acidosis, Shallow breaths.

Treatment DKA

insulin (a lot)
A fair amount fluids first saline and then D5 1/2 NS

Treatment HHNK
Only a little insulin
A lot if fluids

Leukopenia
Abnormally low WBC count < 5000.
Caused by viral illness, bone marrow disorder or medications such as chemotherapy, HIV
regimens, lupus and its meds, antibiotics such as bactrim and immunosuppressive meds. Patients
present with malaise, chills, fever.
Patients should have neutropenic precautions, assess root cause and delay treatment if necessary
until levels are higher, steroids and monitoring.

TIA

ischemic or hemorrhagic causes. Symptoms get better in 24 hours

RIND (stands for)

Reversible ischemic neurological deficit. Can be a result of ischemic or hemorrhagic causes.
Usually takes 3 months or more to improve

Cerebral infarct (which artery, consequences)

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