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NURA 221 FINAL EXAM

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NURA 221 FINAL EXAM

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  • August 23, 2024
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  • 2024/2025
  • Exam (elaborations)
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  • NURA 221
  • NURA 221
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NURA 221 FINAL EXAM 2024-2025


Antidysrhythmic Drugs

drugs used to treat abnormal heart rhythms

begins at the SA node,

descends to the AV node,

down to the bundle of HIS,

to both ventricles

What is the cardiac electrical system pathway?

Automaticity, conductivity

Cardiac dysrhythmias occur usually due to a problem with either __ or ___

Automaticity

The ability to generate an electrical impulse without outside stimulus

Decrease, sinus bradycardia, increase, sinus tachycardia

Cardiac dysrhythmias can come from either a ___ in automaticity causing ___ or an
___ in automaticity causing ___

Conductivity

The ability to conduct an impulse through an internal electrical system

Block, delay

Cardiac dysrhythmias can occur from a __ or __ in conductivity

Asystole, v-tach, V-fib

What are life-threatening dysrhythmias?

,New pattern

harmful, life-threatening

pre-life-threatening

future

The use of antidysrhythmics will depend on:

is this a ___ for this patient?

is this dysrhythmia ___ and/or ___?

is this a ___ rhythm?

is this rhythm that may lead to ___ (near or far) problems?

Resolution, cardiac output, HR, BP, capillary refill, normal

The expected outcome of antidysrhytmic therapy includes ___ of the dysrhythmia,
restoration of the __, __, __, and __, and return to __ state of being

Cardiac output

The amount of volume ejected from the LV over one minute

Class 1: sodium channel blockers

class II: beta-blockers

class III: potassium channel blockers

Class IV: calcium channel blockers

unclassified class: others

What are the antidysrhythmic classes?

1A, 1B, and 1C

Class 1 sodium channel blockers are divided into groups.

Long-term

Class 1A Na channel blockers are for ___ use (dysrhythmias that won't get better)

,Stabilizes, sodium, potassium

Class1A ___ the cell membrane by preventing the ready movement of __ and ___
across the cellular barrier

Impulse conduction, repolarization, atria, ventricles, purkinje fibers

Class 1A decreases __ and delays ___ in the __, ___, and ___

Widening, lengthened

Class 1A Na channel blockers cause ___ QRS and ___ PR-interval on the ECG

SVT, v-tach, a-flutter, a-fib

What are class 1A Na channel blockers used for?

Procainamide (Pronestyl)

What is a class 1A Na channel blocker?

Existing conduction delay

myasthenia gravis, weakness

toxic, >25%, abolition

hold, stop, provider

Class 1A Na Channel Blockers nursing management:

should not be utilized with ___ problems

avoid in ___ patients, may increase muscle ___

watch for ___ effects: widening of the QRS ___ or __ of the P wave

__ or ___ administration and inform ___ ASAP if these are noted

Short term

Class 1B Na channel blockers are for ___ use (have to fix the dysrhythmias right
away)

Electrical conduction, automaticity, repolarization

, Class 1B meds decrease ___ and ___. Clas 1B meds increase ___ rate

Ventricles, v-tach, v-fib, PVCs

Class 1B meds main effect is located within the ___. Helps with __, __, and ___

Lidocaine (Xylocaine)

What is the most popular class 1B Na channel blocker?

Cardiac blocks

levels, toxicity

ECG, IV

immediately, prolonged, prolonged, aggravation

Class 1B nursing management:

do not use with severe ___

check blood ___ often to monitor for ___

constant __ monitoring is necessary for __ admin

stop infusion ___ if any toxic signs are noted: __ PR interval, ___ QRS complex, or
___ of existing dysrhythmia




Conduction, atria, ventricles, purkinje, repolarization

Class 1C Na Channel blockers decrease ___ in __, __, and ___. Class 1C meds
delay ventricular ___




SVT (superventricular tachycardia)

Class 1C Na channel blockers treat and prevent ___

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