Exam (elaborations)
NURS 5315 Module 1 Exam Questions and Answers 2024( A+ GRADED 100% VERIFIED).
- Course
- Institution
NURS 5315 Module 1 Exam Questions and Answers 2024( A+ GRADED 100% VERIFIED).
[Show more]
Seller
Follow
Content preview
NURS 5315 Module 1Cell membrane composed of - ANS phospholipids, cholesterol, glycolipid
Role of phospholipids - ANS repairing the cell by folding
Cell membrane is highly permeable to - ANS CO2, O2, fatty acids, steroid hormones
Resting membrane potential - ANS slight difference in charge between ICF and ECF. Charge is
-70 to -85mV
Action potential - ANS sudden impulse and rapid change in resting membrane potential.
Depolarization - ANS charge is moving closer to zero and becoming more positive
Threshold potential - ANS occurs when the inside of the cell does not change at least 15 to
20mV and the action potential is not successfully reached
Repolarization - ANS occurs when the charge reaches zero and then returns back to the
baseline of -70 to -85mV
Refractory period - ANS cell membrane resists repolarization and cannot depolarize
Absolute refractory period - ANS membrane does not respond to any stimulus
Relative refractory period - ANS repolarization only occurs to very strong stimulus
Hyperpolarized - ANS charge is greater than -85mV. Cell is less excitable
Hyperpolarized - ANS charge is less than -65mV. Cell is very excitable
Hypokalemia effects of resting membrane potential - ANS hyperpolarized, less excitable
Decrease in neuromuscular excitability, weakness, smooth muscle atony, paresthesias and
cardiac dysrhythmias
Hyperkalemia effects of resting membrane potential - ANS hyperpolarized, more excitable
Peak T waves, K+ increases, widening QRS, cardiac standstill
Hypercalcemia effects of resting membrane potential - ANS hyperpolarized, less excitable
Weakness, hyporeflexia, lethargy, confusion, encephalopathy, short QT, depressed T waves
, Hypocalcemia effects of resting membrane potential - ANS hyperpolarized, more excitable
Tetany, hyperreflexia, circumoral paresthesias, seizures, dysrhythmias
Atrophy - ANS E: shrinkage of cell size
P: imbalance between protein synthesis and degradation, less mitochondrial, myofilaments, and
endoplasmic reticulum. Autophagy is also present.
CM: shrinkage of thymus gland in childhood, skeletal muscle atrophy d/t being bedridden
Physiologic atrophy:
Pathologic atrophy: - ANS -shrinkage of thymus gland in childhood
-skeletal muscle shrinkage d/t being bedridden or paralyzed. Also brain shrinkage d/t age
Hypertrophy - ANS E: increase in size of cells
P: hormonal stimulation or increased functional demand which increases cellular protein in
mitochondria and endoplasmic reticulum
CM: skeletal muscle growth d/t lifting weights and working out, kidney enlargement d/t other
kidney being removed, cardiomegaly d/t increased workload
Physiologic hypertrophy:
Pathologic hypertrophy: - ANS -skeletal muscle enlargement d/t lifting weights, surgical removal
of one kidney causes the other kidney to enlarge to compensate
-cardiomegaly d/t increased workload, hypertension
Hyperplasia - ANS E: increase in number of cells
P: happens only in cells capable of mitosis d/t growth factors that stimulate the cells to multiply
CM: increase in tissue mass as damage or resection, removal of part of liver, uterine and
mammary enlargement in pregnancy, endometrial lining of the uterus d/t estrogen secretion
Physiologic hyperplasia:
Pathologic hyperplasia: - ANS compensatory hyperplasia d/t increase in tissue mass after
damage or resection, removal of part of the liver will result in it to regenerate, uterine and
mammary gland enlargement d/t pregnancy
abnormal increase in hormonal stimulation, endometrial lining undergoes hyperplasia as a result
of estrogen however progesterone will halt this. It becomes a problem when progesterone does
not halt it and it will lead to cancer
Metaplasia - ANS E: reversible, adult cell is replaced by another adult cell
The benefits of buying summaries with Stuvia:
Guaranteed quality through customer reviews
Stuvia customers have reviewed more than 700,000 summaries. This how you know that you are buying the best documents.
Quick and easy check-out
You can quickly pay through credit card or Stuvia-credit for the summaries. There is no membership needed.
Focus on what matters
Your fellow students write the study notes themselves, which is why the documents are always reliable and up-to-date. This ensures you quickly get to the core!
Frequently asked questions
What do I get when I buy this document?
You get a PDF, available immediately after your purchase. The purchased document is accessible anytime, anywhere and indefinitely through your profile.
Satisfaction guarantee: how does it work?
Our satisfaction guarantee ensures that you always find a study document that suits you well. You fill out a form, and our customer service team takes care of the rest.
Who am I buying these notes from?
Stuvia is a marketplace, so you are not buying this document from us, but from seller LECTSKYJAYDEN. Stuvia facilitates payment to the seller.
Will I be stuck with a subscription?
No, you only buy these notes for $11.49. You're not tied to anything after your purchase.
Can Stuvia be trusted?
4.6 stars on Google & Trustpilot (+1000 reviews)
75619 documents were sold in the last 30 days
Founded in 2010, the go-to place to buy study notes for 14 years now