100% satisfaction guarantee Immediately available after payment Both online and in PDF No strings attached
logo-home
Test Bank For Pathophysiology: A Practical Approach 4th Edition By Lachel Story 9781284205435 Chapter 1- 14 Complete Guide . $14.99   Add to cart

Exam (elaborations)

Test Bank For Pathophysiology: A Practical Approach 4th Edition By Lachel Story 9781284205435 Chapter 1- 14 Complete Guide .

 8 views  0 purchase
  • Course
  • Pathophysiology: A Practical Approach 4th Edition
  • Institution
  • Pathophysiology: A Practical Approach 4th Edition

Test Bank For Pathophysiology: A Practical Approach 4th Edition By Lachel Story 5435, 6 , 9967, 3 1 Cellular Function 2 Immunity 3 Hematopoietic Function 4 Cardiovascular Function 5 Respiratory Function 6 Fluid, Electrolyte, and Acid–Base Homeostasis 7 Urinary Function 8 Reproductive Function 9 G...

[Show more]

Preview 4 out of 120  pages

  • August 31, 2024
  • 120
  • 2024/2025
  • Exam (elaborations)
  • Questions & answers
book image

Book Title:

Author(s):

  • Edition:
  • ISBN:
  • Edition:
  • Pathophysiology: A Practical Approach 4th Edition
  • Pathophysiology: A Practical Approach 4th Edition
avatar-seller
Prose1
Test Bank For Applied Pathophysiology A
h h h h h


Conceptual Approach 4th Edition with
h h h h h


correct QAS 100% verified by experts 2023-
h h h h h h h


2024
h

,Pathophysiology: hA hPractical hApproach: hA hPractical hApproach h4th
hEdition hStory hTest hBank




Chapter h 1 h Cellular h Function
h 1. An h increase h in hcardiac h size h and h function h due h to h increased h workload h is
h termed
A) Atrophy.
B) Functional.
C) Hypertrophy.
D) Inflammation.

h 2. While h assessing h a h patient hdiagnosed h with h Marfan's h Syndrome, h the h nurse
h should
include h which h of hthe h following hthat h is hconsistent h with hthe h syndrome?
A) Cardiac h assessment h for h coarctation h of h the h aorta.
B) Genital h assessment h for h small h testicles.
C) Mental h assessment h for h impairment.
D) Oral h assessment h for h cleft h palate.


h 3. Children h with h PKU h must h avoid h phenylalanine h in hthe h diet. h Phenylalanine h is
h most
likely hto h be h a h component hof
A) Fat.
B) Sugar.
C) Protein.
D) Carbohydrate.


h 4. A h17-year-old h college-bound h student h receives h a h vaccine h against h an h organism
h that
causes hmeningitis. hThis his h an h example h of
A) primary h prevention.
B) secondary h prevention.
C) tertiary h prevention.
D) disease h treatment.


h 5. Metaplasia his
A) The h disorganization h of hcells h into h various h sizes, h shapes, h and h arrangements.
B) The h replacement h of h one h differentiated h cell htype h with h another.
C) The htransformation h of ha h cell h type h to h malignancy.
D) An h irreversible h cellular h adaptation.

, h 6. During ha h routine h ultrasound h of h a h 38 h year h old h women h 26 h weeks h gestation
h reveals h a
fetus hwith ha hsmall h square hhead, hupward h slant h of hthe heyes, hand h low hset
h ears. hThe hnurse h recognizes h that h these h findings h are h consistent h with h which
h of h the h following?
A) Fragile h X hSyndrome.
B) Monosomy hX h (Turner's h Syndrome).
C) Trisomy h21 h (Down's h Syndrome).
D) Trisomy h X h (Klinefelter's h Syndrome).


h 7. While h discussing h treatment h options h with h a h parent h of h a h newly h diagnosed
h Monosomy
X h (Turner's h Syndrome) h child, h the h nurse h should hinclude h which h of hthe
h following?
A) There h is hno htreatment h or hcure.
B) Symptoms h of hthe h condition h are h treated h with h estrogen.
C) Symptoms h of h the h condition h are h treated h with h testosterone.
D) Institutionalization h is h the h preferred h method h of h managing h care.


h 8. Which h of h the h following h assessment h findings h indicates h an h alteration h in
h homeostatic
control h mechanisms?
A) Fever
B) Throat h pain
C) Joint hstiffness
D) Positive h throat h culture


h 9. What h information h should h parents h be h given h about h the h consequences h of
phenylketonuria h (PKU)?
A) Mental h retardation h is h inevitable.
B) PKU h is h commonly h associated h with h other h congenital h anomalies.
C) High h dietary h tyramine h may h help h induce h enzyme h production.
D) Failure h to h treat h properly h results h in h progressive h mental h retardation.


h 10. Injury hthat h occurs h when h blood h flow his h diminished h to htissue h is hcalled injury.
A) hypoxic
B) ischemic
C) hyperemic
D) neoplastic


h 11. Tay hsachs h is h caused h by hwhich h of hthe h following?
A) A hdeficiency h or h absence h of h hexosaminidase hA
B) A hdefect h on h chromosome h 17 h or h 22
C) A hmutation h on h chromosome h 15

, D) An h error h in h converting h phenylalanine h to h tyrosine


h 12. An h obese h but h otherwise h healthy hteen h goes h to ha h health h fair h and h has hher
h blood
pressure h checked. hThis h is han h example h of
A) primary h prevention.
B) secondary h prevention.
C) tertiary h prevention.
D) disease h treatment.


h 13. Characteristics h of h X-linked h recessive h disorders h include h which h of h the
h following?
A) The h son h of h a hcarrier h mother h has ha h 25% hchance h of hbeing haffected.
B) Affected h fathers h transmit h the h gene hto hall h of htheir h sons.
C) All h daughters h of h affected h fathers h are h carriers.
D) Boys h and h girls h are h equally haffected.


h 14. A hfactor h associated h with h risk h of hDown h syndrome h is
A) Maternal hage.
B) Maternal halcohol h intake.
C) Family h history h of h heritable h diseases.
D) Exposure h to hTORCH h syndrome h organisms.


h 15. Which h type h of h gangrene h is h usually h a h result h of harterial h occlusion?
A) Necrosis.
B) Dry.
C) Wet.
D) Gas.


h 16. The h cancer h growth h continuum h is h divided hinto h the h following h stages.
A) Stage h1, h Stage h 2, hStage h3
B) Initiation, h Progression, h Promotion
C) Preliminary, hEvolutionary, hMetastasis
D) Initiation, h Promotion, h Progression


h 17. A hdisease h in h which h the h principal h manifestation h is han h abnormal h growth h of
h cells
leading hto h formation h of htumors his hcalled h a disease.
A) congenital
B) degenerative
C) metabolic

The benefits of buying summaries with Stuvia:

Guaranteed quality through customer reviews

Guaranteed quality through customer reviews

Stuvia customers have reviewed more than 700,000 summaries. This how you know that you are buying the best documents.

Quick and easy check-out

Quick and easy check-out

You can quickly pay through credit card or Stuvia-credit for the summaries. There is no membership needed.

Focus on what matters

Focus on what matters

Your fellow students write the study notes themselves, which is why the documents are always reliable and up-to-date. This ensures you quickly get to the core!

Frequently asked questions

What do I get when I buy this document?

You get a PDF, available immediately after your purchase. The purchased document is accessible anytime, anywhere and indefinitely through your profile.

Satisfaction guarantee: how does it work?

Our satisfaction guarantee ensures that you always find a study document that suits you well. You fill out a form, and our customer service team takes care of the rest.

Who am I buying these notes from?

Stuvia is a marketplace, so you are not buying this document from us, but from seller Prose1. Stuvia facilitates payment to the seller.

Will I be stuck with a subscription?

No, you only buy these notes for $14.99. You're not tied to anything after your purchase.

Can Stuvia be trusted?

4.6 stars on Google & Trustpilot (+1000 reviews)

80461 documents were sold in the last 30 days

Founded in 2010, the go-to place to buy study notes for 14 years now

Start selling
$14.99
  • (0)
  Add to cart