Chapterv 1vCellularv Function
1. An increase in cardiac size and function due to increased workload is termed
A) Atrophy.
B) Functional.
C) Hypertrophy.
D) Inflammation.
v 2. Whilev assessingv av patientvdiagnosedv withv Marfan'sv Syndrome,v thev nursev should
includevwhichv ofvthev followingvthatv isvconsistentv withvthev syndrome?
A) Cardiacv assessmentv forv coarctationv ofvthev aorta.
B) Genitalv assessmentv forv smallv testicles.
C) Mentalv assessmentv forv impairment.
D) Oralvassessmentv forv cleftv palate.
v 3. Childrenv withv PKUv mustv avoidv phenylalaninev invthev diet.v Phenylalaninev isv most
likelyvtov bev avcomponentvof
A) Fat.
B) Sugar.
C) Protein.
D) Carbohydrate.
v 4. Av17-year-oldv college-boundv studentv receivesv av vaccinev againstv anv organismv that
causesvmeningitis.vThisvisv anvexamplev of
A) primaryv prevention.
B) secondaryv prevention.
C) tertiaryv prevention.
D) diseasev treatment.
v 5. Metaplasiavis
A) Thev disorganizationv ofvcellsv intov variousv sizes,v shapes,v andv arrangements.
B) Thevreplacementv ofvonevdifferentiatedv cellvtypevwithv another.
C) Thevtransformationv ofvavcellvtypevtovmalignancy.
D) Anv irreversiblev cellularv adaptation.
, v 6. Duringvav routinev ultrasoundv ofvav 38v yearv oldv womenv26v weeksv gestationv revealsva
fetusvwithvavsmallvsquarevhead,vupwardvslantvofvtheveyes,vandvlowvsetvears.vThevnur
sevrecognizesv thatvthesev findingsvarev consistentv withv whichvofvthev following?
A) FragilevXvSyndrome.
B) MonosomyvXv (Turner'sv Syndrome).
C) Trisomyv21v (Down'sv Syndrome).
D) TrisomyvXv (Klinefelter'sv Syndrome).
v 7. Whilev discussingv treatmentv optionsv withv av parentv ofv av newlyvdiagnosedv Monosomy
Xv(Turner'sv Syndrome)v child,v thevnursev shouldvincludev whichv ofvthev following?
A) Therev isvnovtreatmentvorvcure.
B) Symptomsv ofvthev conditionv arev treatedv withv estrogen.
C) Symptomsv ofvthev conditionv arev treatedv withv testosterone.
D) Institutionalizationv isv thev preferredv methodv ofv managingv care.
v 8. Whichv ofv thev followingv assessmentv findingsv indicatesv anv alterationv inv homeostatic
controlv mechanisms?
A) Fever
B) Throatv pain
C) Jointvstiffness
D) Positivev throatv culture
v 9. Whatv informationv shouldv parentsv bev givenv aboutv thev consequencesv of
phenylketonuriav (PKU)?
A) Mentalv retardationv isv inevitable.
B) PKUv isv commonlyv associatedv withv otherv congenitalv anomalies.
C) Highv dietaryvtyraminev mayv helpv inducev enzymev production.
D) Failurev tov treatv properlyvresultsv inv progressivev mentalv retardation.
v 10. Injuryvthatv occursvwhenvbloodv flowvisvdiminishedvtov tissuev isvcalled injury.
A) hypoxic
B) ischemic
C) hyperemic
D) neoplastic
v 11. Tayvsachsvisvcausedvbyvwhichvofvthev following?
A) Avdeficiencyvorv absencev ofvhexosaminidasevA
B) Avdefectv onv chromosomev 17v orv22
C) Avmutationv onv chromosomev 15
, D) Anv errorv inv convertingv phenylalaninev tov tyrosine
v 12. Anvobesev butvotherwisev healthyvteenv goesvtovav healthv fairv andvhasvhervblood
pressurev checked.vThisv isv anv examplev of
A) primaryv prevention.
B) secondaryv prevention.
C) tertiaryv prevention.
D) diseasev treatment.
v 13. Characteristicsv ofv X-linkedv recessivev disordersv includev whichv ofv thev following?
A) Thevsonvofvavcarriervmotherv hasvav 25%vchancev ofvbeingvaffected.
B) Affectedvfathersvtransmitv thev genevtovallvofvtheirvsons.
C) Allv daughtersv ofvaffectedvfathersv arevcarriers.
D) Boysvandv girlsv arevequallyvaffected.
v 14. Avfactorv associatedv withvriskvofvDownv syndromev is
A) Maternalvage.
B) Maternalvalcoholv intake.
C) Familyvhistoryvofv heritablev diseases.
D) Exposurev tovTORCHv syndromev organisms.
v 15. Whichv typev ofv gangrenev isv usuallyvav resultv ofvarterialv occlusion?
A) Necrosis.
B) Dry.
C) Wet.
D) Gas.
v 16. Thev cancerv growthv continuumv isv dividedvintov thev followingv stages.
A) Stagev1,vStagev2,vStagev3
B) Initiation,v Progression,v Promotion
C) Preliminary,vEvolutionary,vMetastasis
D) Initiation,v Promotion,v Progression
v 17. Avdiseasev inv whichv thev principalv manifestationv isvanvabnormalv growthv ofvcells
leadingvtov formationvofvtumorsvisvcalledva disease.
A) congenital
B) degenerative
C) metabolic
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