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Davis Advantage for Pathophysiology Introductory Concepts and Clinical Perspectives 3rd Edition By Theresa Capriotti
Davis Advantage for Pathophysiology Introductory Concepts and Clinical Perspectives 3rd Edition By Theresa Capriotti
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Davis Advantage for Pathophysiology Introductory Concepts
and Clinical Perspectives 3rd Edition By Theresa Capriotti
3 goals of inflammation - ANSWER:wall off injured area
effusion - ANSWER:accumulation of fluid in body cavity
Cellular Chemotaxis - ANSWER:a chemical signal from microbial agents, endothelial
cells, and WBCs attracts platelets and other WBCs to the site of injury
Margination - ANSWER:WBCs line up along endothelium
release inflammatory mediators
Leukocytosis - ANSWER:increase in the number of white blood cells
, leukemoid reaction - ANSWER:extreme elevation in WBC
WBC differential - ANSWER:measure of the percentage of different types of white
blood cells present in the blood
cytokines (2) - ANSWER:regulate immunity, released by WBC
Tumor necrosis factor alpha TNF
Interleukins ILs
chemokines - ANSWER:Attract leukocytes to site of inflammation
acute phase proteins - ANSWER:liver produces in response to cytokines
CRP, fibrinogen, serum amyloid A, Hepcidin
C-reactive protein (CRP), indicates? - ANSWER:marks foreign material for
phagocytosis
activates complement system
stimulates other inflammatory cytokines
elevation indicates active inflammation
High sensitivity C-reactive protein indicates - ANSWER:risk of heart attack
fibrinogen - ANSWER:binds to RBCs and fixes them into stacks that precipitate
process called rouleaux and sedimentation
erythrocyte sedimentation rate (ESR) - ANSWER:timed test that measures the rate at
which red blood cells settle through a volume of plasma
active inflammation
Amyloidosis, indicates? - ANSWER:prolonged secretion of serum amyloid a
indicates chronic inflammation
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