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CSU MIP 260 Exam Study Questions with 100% Correct Answers
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CSU MIP 260 Exam Study Questions with 100% Correct Answers
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CSU MIP 260 Exam Study Questionswith 100% Correct Answers
Tick disease - ✔✔Efficient carrier, popular vector, causes blood loss,
anemia, disturbance and annoyance
Mite morphology - ✔✔Carucles(suckers), pedicles(stalk),
chelicera(mouthparts), tarsal claws
Mite life cycle - ✔✔Eggs->larva->nymph-> adult on surface of skin, can be all
over body or burrow depending on species
Psoroptes spp. - ✔✔Long jointed pedicles, ears mane and tail on large animals
and rabbits, pruritus is absent or intense
Chorioptes spp. - ✔✔Short pedicle, non burrowing, utter and hind legs, suckers on
pedicles, nonsegmented, direct contact transmission, itching, crusty scaly, common in
winter can affect milk production
Cheyletiella spp. - ✔✔Walking dandruff, nonburrowing, feed on keratin of
epidermis, dorsum, direct contact transmission
Sarcoptes scabei - ✔✔Dogsn wild canids, burrows in skin, 6 legged larvae, doesn't
survive well in environment, long unsegmented pedicles, alopecia, hyperkeratosis,
ears elbows flanks and ventrum
,Demodex spp. - ✔✔Eggs to adult in a month, hair follicles and oil glands, direct
contact, mild signs on head or limbs, can resolve itself or overgrow with
weakened immune system
Immune Response - ✔✔host reacts to invader, diseases caused by parasite are
form host defense mechanism and parasite invasion mechanism
Susceptibility vs Resistance - ✔✔Susceptibility= can't eliminate parasite before
its established, resistance= parasite establishment and survival is prevented
Innate vs Adaptive immunity - ✔✔innate- already in body, don't need previous
exposure, adaptive- specific reaction to pathogen, takes time to develop, reaction
stronger after 2nd exposure
Premunition - ✔✔incomplete resistance, parasite is held in check, host
is asymptomatic
Concomitant immunity - ✔✔Parasite protects against reinfection, schistosomiasis
Innate defenses - ✔✔Surface and internal bariers, differentiate molecules
between self and non self
Cell signaling - ✔✔Cells have receptor that detect molecules, ligands=binding
molecules, can be detected on surface of cells in blood or surface of pathogens,
can induce apoptosis, phagocytosis or gene induction
, Cytokines/receptors - ✔✔Protein hormones, mechanism for cells to
communicate, can effect huge range of cells with specific receptor, ligation=cells are
attracted to receptor, causes intracellular cascade
Antimicrobial Peptides - ✔✔Low specificity, targeted toward category of
microbes (gram +/-, fungi)
Pattern Recognition Receptors - ✔✔on surface of cells, recognize molecular
patters, mediates secretion of antimicrobial peptides, can initiate phagocytosis or
complement, complement receptors
Complement - ✔✔Series of enzymes activated in a sequence, targets bacteria
fungi and helminths, cascade ends in cell lysis or destruction of invader, either start
by antibody binding to antigen or enzyme activated in blood and binds to cell surfaces
Scavenger receptors (PRR's) - ✔✔bind to many ligands and other molecules
produced by bacteria
Toll-like receptors (PRR's) - ✔✔Highly conserved, activated innate immunity and
initiates adaptive immunity, recognizes multiple different types of molecules, leads
to cascade and expression of antimicrobial peptides
GPIs - ✔✔Anchor proteins on eukaryotic cell membranes, variety of
pathogenic effects and immune response
Other chemical defenses - ✔✔pH extremes, antimicrobial enzymes in GI, mucus,
IgA and lysozyme, in saliva sweat and WBCs
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