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Nesters Microbiology A Human Perspective 8th Edition Anderson Solutions Manual $17.99   Add to cart

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Nesters Microbiology A Human Perspective 8th Edition Anderson Solutions Manual

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Nesters Microbiology A Human Perspective 8th Edition Anderson Solutions Manual

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  • September 14, 2024
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  • 2024/2025
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Nesters Microbiology A Human Perspective 8th Edition
Anderson Solutions Manual

Red throat with patches of pus and scattered tiny hemorrhages (not adenovirus) -
ANSWER: Strep Throat

Neck lymph nodes enlarged and tender (not adenovirus) - ANSWER: Strep Throat

Scarlet fever and roughening of the skin - ANSWER: Strep Throat

Many virulence factors that can alter structural components of the host or be
enzymes and toxins - ANSWER: Streptococcus pyogenes

80 subtypes of streptococcal protein that are adhesins and prevent phagocytosis due
to C3b - ANSWER: M protein

Streptococcal protein that helps in attachment to cells of the throat by adhering to
fibrin - ANSWER: Protein F

Produces DNase, hyaluronidase, and proteases to spread (does not cause
conjunctivitis) - ANSWER: Streptococcus pyogenes

Produces streptokinase to break clots - ANSWER: Streptococcus pyogenes

Produces protein G as an Fc receptor - ANSWER: Streptococcus pyogenes

Produces C5a peptidase, preventing migration of phagocytes - ANSWER:
Streptococcus pyogenes

Produces Streploysins O and S that destroy erythrocytes and leukocytes - ANSWER:
Streptococcus pyogenes

Produces SPEs, aka erythrogenic toxin, in lysogenized form, superantigens that cause
massive release of cytokines causing scarlet fever and roughening of the skin -
ANSWER: Streptococcus pyogenes

Respiratory pathogen that may also cause "flesh eating necrotizing fasciitis" -
ANSWER: Streptococcus pyogenes

Protein that prevents migration of phagocytes - ANSWER: C5a peptidase

Streptococcal protein that acts as an Fc receptor - ANSWER: Protein G

,Protein produced to break clots - ANSWER: Streptokinase

Proteins that destroy erythrocytes and leukocytes - ANSWER: Streploysins O and S

Superantigens that cause massive release of cytokines causing scarlet fever and
roughening of the skin - ANSWER: SPE

GAS - ANSWER: Group A Streptococcus

Criterion for Lancefield grouping - ANSWER: Cell wall carbohydrates

Produces a hyaluronic acid capsule - ANSWER: Streptococcus pyogenes

Type of antibodies to which Protein G bonds - ANSWER: IgG

Confirmed by diagnostic tests and throat culture - ANSWER: Strep Throat

Treat with penicillin and erythromycin (2) - ANSWER: Strep Throat, Pneumococcal
pneumonia

Long term carriers have strains that are deficient in a protein (agent and protein) -
ANSWER: Streptococcus pyogenes, M protein

Infection that leads to multiple sequelae due to immune response - ANSWER: Strep
Throat

Can occur after healing from strep throat - ANSWER: Post-Streptococcal Sequelae

Post-Streptococcal Sequelae (3) - ANSWER: Acute Rheumatic Fever, Chronic
Rheumatic Heart Disease (Subacute Endocarditis), Acute Post-Streptococcal
Glomerulonephritis

Joint pains, chest pains, rash, and nodules under the skin - ANSWER: Acute
Rheumatic Fever

Causes chorea - ANSWER: Acute Rheumatic Fever

Causes heart failure and damage to valves in 1/3 of patients - ANSWER: Acute
Rheumatic Fever

Only develops in people who are genetically predisposed due to MHC II alleles -
ANSWER: Acute Rheumatic Fever

Autoimmune response involving both humoral and cell mediated response with
antibodies against the pathogen reacting with host tissue such as cardiac myosin -
ANSWER: Acute Rheumatic Fever

, Synonym of chorea - ANSWER: Uncontrollable body movement

Synonym of uncontrollable body movement - ANSWER: Chorea

Follows Acute Rheumatic Fever - ANSWER: Chronic Rheumatic Heart Disease

Precedes Chronic Rheumatic Heart Disease - ANSWER: Acute Rheumatic Fever

Can follow Strep Throat but more commonly follows streptococcal skin infection -
ANSWER: Acute Post-Streptococcal Glomerulonephritis

Fluid retention, high BP, blood and proteins in urine (looks like Coca Cola) - ANSWER:
Acute Post-Streptococcal Glomerulonephritis

Damage due to inflammatory response to pathogen antigens that accumulate in the
kidney glomeruli - ANSWER: Acute Post-Streptococcal Glomerulonephritis

Antibodies bind to antigens of pathogen and initiate complement response -
ANSWER: Acute Post-Streptococcal Glomerulonephritis

Disease caused by a pleomorphic bacterium - ANSWER: Diphtheria

Bacterium that forms pallisades - ANSWER: Corynebacterium diphtheriae

Forms black colonies in medium containing potassium-tellurite - ANSWER:
Corynebacterium diphtheriae

Grown on Loeffler medium, enhancing volutin granules that can be seen with
methylene blue - ANSWER: Corynebacterium diphtheriae

Dramatic swelling of the neck - ANSWER: Diphtheria

Whitish-gray pseudomembrane on the tonsils, throat, or nasal cavity - ANSWER:
Diphtheria

Heart, kidney failure, and paralysis (nerve damage) may occur due to toxin in blood -
ANSWER: Diphtheria

Toxin in lysogenized strains - ANSWER: Corynebacterium diphtheriae

Structure made of dead cells, clotted blood, fibrin, and leukocytes - ANSWER:
Diphtheria Pseudomembrane

Structure that may come loose and obstruct passageway - ANSWER: Diphtheria
Pseudomembrane

Obstruction by a membrane that leads to suffocation - ANSWER: Diphtheria

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