Exam (elaborations)
NURS 5315 UTA EXAM 1 WITH QUESTIONS AND VERIFIED DETAILED ANSWERS
- Course
- Institution
NURS 5315 UTA EXAM 1 WITH QUESTIONS AND VERIFIED DETAILED ANSWERS ...
[Show more]
Content preview
NURS 5315 UTA EXAM 1 WITH QUESTIONS AND VERIFIEDDETAILED ANSWERS 2024-2025
Atrophy - Answer E. Shrinkage of cells in size
P. Still functional
Physiologic: thymus gland in early childhood
Pathological: disuse
Hypertrophy - Answer E. Increase in cell size
P. Increased workload
Physiologic: weightlifting
Pathologic: cardiomegaly from HTN
Hyperplasia - Answer E. Increase in cell number
P. Increased cellular division
Physiologic: liver regeneration
Pathologic: endometrial- usually r/t hormones
Dysplasia - Answer E. Cells change in size, shape, organization
P. AKA atypical hyperplasia, a disorderly proliferation
Physiologic: N/A
Pathologic: squamous dysplasia of cervix from HPV
Metaplasia - Answer E. one cell type replaced with another
P. reprogramming of stem cells, reversible
Physiologic: N/A
, Pathologic: stratified squamous cells in bronchial lining r/t cigarette smoke
Hypoxia injury - Answer E. inadequate oxygenation of tissues
P. decrease in mitochondrial function, decreased production of ATP increases
anaerobic metabolism. eventual cell death.
C.M. hypoxia, cyanosis, cognitive impairment, lethargy
Free radical and ROS - E. normal product of ATP production, will overwhelm the
mitochondria- use up intracellular antioxidants
P. cause lipid peroxidation, damage proteins, fragment DNA
C.M. development in Alzheimer's, heart disease, Parkinson's disease, Amyotrophic
Lateral Sclerosis
Ethanol - E. mood altering drug, long term effects on liver and nutritional status
P. metabolized by liver, produces free radicals
C.M. CNS depression, nutritional deficiencies-Mag, Vit B6, thiamine, PO4, inflammation
and fatty change of liver, hepatomegaly, progresses to liver failure irreversible
Oncosis - Response Na and H2O influx into cell and swelling of cell. Organ weight
increases, becomes distended and pale. Associated with high fever, hypocalcemia,
some infection
Fatty Infiltration - Response intracellular lipid deposition within the liver
liver unable to metabolize lipids. usually from ETOH or very greasy diet. can result in
cirrhosis
dystrophic calcification- Answer deposition of Ca in dead/damaged tissues
calcium salt precipitate and coalesce- cellular architecture and function impaired
r/t pulmonary TB, arteriosclerosis, traumatized heart valves, chronic pancreatitis
metastatic calcification- Answer deposition of Ca in normal tissue
result of hypercalcemia r/t hyperparathyroidism, hyperthyroidism, toxic levels of Vit D.
The benefits of buying summaries with Stuvia:
Guaranteed quality through customer reviews
Stuvia customers have reviewed more than 700,000 summaries. This how you know that you are buying the best documents.
Quick and easy check-out
You can quickly pay through credit card or Stuvia-credit for the summaries. There is no membership needed.
Focus on what matters
Your fellow students write the study notes themselves, which is why the documents are always reliable and up-to-date. This ensures you quickly get to the core!
Frequently asked questions
What do I get when I buy this document?
You get a PDF, available immediately after your purchase. The purchased document is accessible anytime, anywhere and indefinitely through your profile.
Satisfaction guarantee: how does it work?
Our satisfaction guarantee ensures that you always find a study document that suits you well. You fill out a form, and our customer service team takes care of the rest.
Who am I buying these notes from?
Stuvia is a marketplace, so you are not buying this document from us, but from seller Stetson. Stuvia facilitates payment to the seller.
Will I be stuck with a subscription?
No, you only buy these notes for $9.99. You're not tied to anything after your purchase.
Can Stuvia be trusted?
4.6 stars on Google & Trustpilot (+1000 reviews)
82215 documents were sold in the last 30 days
Founded in 2010, the go-to place to buy study notes for 14 years now
