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SLCC Pathophysiology Exam 3 GUIDE FROM CATT86 Questions With Complete Solutions
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SLCC Pathophysiology Exam 3 GUIDE FROM CATT86 Questions With Complete Solutions 3 Second Assessment Check wrist pulse ask them to take a deep breath and move their foot Airways Breathing Circulation Decrease in neurological function Arteriosclerosis An INFLAMMATORY PROCESS that causes ...

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  • October 13, 2024
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  • 2024/2025
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  • slcc pathophysiology exam 3 guide from catt86
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SLCC Pathophysiology Exam 3 GUIDE
FROM CATT86 Questions With Complete
Solutions

3 Second Assessment Check wrist pulse ask them to take a deep breath and move their

foot

Airways

Breathing

Circulation

Decrease in neurological function




Arteriosclerosis An INFLAMMATORY PROCESS that causes abnormal thickening and

hardening of arterial wall from deposition of collagen into vessel wall leads to diminished

distensability.




Atherosclerosis is the narrowing of the artery because of plaque build-up. It is a form of

arteriosclerosis resulting from fat being deposited in vessel wall that reduce lumen size.




Steps of Atherosclerosis Development 1. Ulceration of vessel-tear arterial wall


2. Fatty streak

, SLCC Pathophysiology Exam 3 GUIDE
FROM CATT86 Questions With Complete
Solutions
3. Fibrous plaque

4. Complicated lesion (Thrombosis)

5. Thrombus to Embolus-could result in everything from localized tissue necrosis to death from a

pulmonary embolism that lodges in the lungs




Coronary Artery Disease (CAD) Etiology: atherosclerosis, vasospasm, thrombi


Incidence: 50% all deaths (35% deaths in 35-65 year old)

Pathophysiology: coronary artery occlusion → decreased myocardial perfusion → myocardial

ischemia → angina → ischemia persists (>20 minutes) → ischemic cells are injured → injured

cells necrosis (Myocardial infarction) → myocardium scars over dead tissue dead tissue will not

conduct electrical current → NO HEART BEAT! (this results in abnormal EKG readings).




Risk Factors Coronary Artery Disease Non-modifiable - age >60, gender especially male,

genetic predisposition, Type I Diabetes

Modifiable - hyperlipidemia (high cholesterol), hypertension, smoking, obesity, sedentary

lifestyle, Type II Diabetes.

, SLCC Pathophysiology Exam 3 GUIDE
FROM CATT86 Questions With Complete
Solutions
DVT RISK FACTORS: Prior history of a DVT (**this is the #1 highest risk factor**);

Pregnancy;

sitting for long periods (i.e., driving/flying); desk job; Birth control pills; obesity; smoking.

Pathophysiology: hemostasis→ activation of intrinsic clotting cascade → thrombus formation

that is the source of blood clots that break off and travel to different parts of the body (emboli) -

usually to lungs and causing P.E. (pulmonary emboli).

S/S: tenderness, swelling, redness/warmth in affected leg (when compared to other leg)

Rx: anticoagulants (like Heparin, Coumadin, Plavix or Lovenox) and prevention




Angiotensin Receptor Blocker Drug Therapy: ARB


*end in -in




Angiotensin Converting Enzyme Drug Therapy: ACE Inhibitor




Beta blocker Drug Therapy: Blocks beta receptors I (heart) & II (lungs). Heart slows

down and lowers blood pressure. Causes lungs to bronco-dilate and causes coughing.

*End in -olol

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