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Pharm test 3 Maryville NURS 615 -questions with correct answers
- Course
- Maryville NURS615
- Institution
- Maryville College
Pharm test 3 Maryville NURS 615 -questions with correct answers
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Pharm test 3 Maryville NURS 615 -questions with correctanswers
1.How do antigout medications work?: by inhibiting the infiltration and
phagocy- tosis of leukocytes, thus decreasing the breakdown of uric
acid to urate crystals.
2.Deposition of urate crystals causes: pain and inflammation
3.What is Colchicine: anti-gout medication
4.how does Colchicine work?: Works by inhibiting inflammation,
reducing pain and swelling
5.common side effects of Colchicine: GI problems (diarrhea) use with
caution in elderly
Also can cause blood dyscrasias & abdominal pain
6.patient education with Colchicine: Can be given with food & milk to
decrease GI issues
Avoid beer, ale, & wine: may cause gout attack
Increase fluid intake: increases excretion of uric
acid Avoid smoked meats & high-protein diets
7.Low dose of Colchicine: Low dose colchicine is 1.2mg followed by
0.6mg one hour later or 1.8 milligrams total
8.High dose of Colchicine: high dose colchicine is 1.2mg followed by
0.6mg every four to six hours; or 4.8mg total.
9.Difference between high dose and low dose Colchicine: The difference
be- tween the two is low dose is as effective as high dose with a lower
,side effect profile.
10.Lab values to monitor with Colchicine: Check renal function test, BUN,
Cre- atine
11.Patient education with Colchicine: Almost always causes some
degree of diarrhea, make sure patients are aware of this side effect
12.Patient education with Febuxostat (Uloric): Gout may worsen with
therapy initially
13.Dietary changes to decrease gout attacks and uric acid deposits.:
Avoid beer, ale, & wine
Increase fluid intake: increases excretion of uric
acid Avoid smoked meats & high-protein diets
14.WHO 3 step ladder for pain medication:
15.What is not a first line for pain medication?: Narcotics
16.Recommendations for pain treatment: You want to start with NSAIDs
first and then work your way up from there.
17.types of Corticosteroid treatment: Prednisone, Cortisone,
Dexamethasone
,18. how do Corticosteroids work?: Suppress the inflammatory & immune
systems by inhibiting the synthesis of chemical mediators.
19.what are the chemical mediators that Corticosteroids work on?: -
Prostaglandins, leukotrienes, & histamines
20.Corticosteroids and inflammation: Decreases inflammation which
decreases swelling, warmth, redness, & pain.
21.Uses for corticosteroid treatment: Addison's disease, hormone
replacement, cancer therapy
SLE, arthritis, IBD, & to suppress graft rejection
22.contraindications for corticosteroid treatment: Systemic fungal
infections & with live vaccine
23.Use corticosteroids cautiously with:: Pregnancy, kids, HTN, heart
failure, renal impairment, & with infections resistant to treatment
24.Patient education with corticosteroids: Don't discontinue abruptly,
doses may need increased during stress, symptoms of Cushing's and GI
bleeding
25.Problem with taking Corticosteroids for greater than 6 months: The main
thing you want to worry about is osteoporosis it can also worsen
diabetic control and patients should report any tarry black stools or
abdominal pain.
26.Other common side effects with Corticosteroids: Peptic ulcers, GI
bleeding, edema, hyperglycemia, delayed wound healing, fluid &
electrolyte imbalances
27.Why is it important to tapper the corticosteroid?: Tapering must be
, done carefully to avoid both recurrent activity of the underlying disease
process and possible cortisol deficiency resulting from the
hypothalamic-pituitary-adrenal axis or HPA suppression during the
period of steroid therapy
28.Black box warning: Increased risk of serious cardiovascular thrombotic
events, myocardial-infarction and stroke which can be fatal.
29.What increases risk of black box warning occurring with NSAIDs.:
Risk increases with duration of use.
Patients with cardiovascular disease or with risk factors for cardiovascula
disease may be a greater risk.
30.NSAIDs and GI system: Increased risk of serious gastrointestinal
adverse effects including: bleeding, ulceration, and perforation the
stomach or intestines can be fatal.
Can occur at any time during use and without warning symptoms.
Elderly patients are at greater risk for serious GI events.
31.Ibuprofen and mechanism of action: Exact mechanism of action is
unknown. A non-selective cox-2 inhibitor in that it inhibits two isoforms
of psychologic oxyge- nase cox-1 and cox-2.
A non-selective inhibitor of cyclooxygenase, believed to be due
inhibition of cox-2
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