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NURS 617 Exam 1 blueprint Unit 9 TEST QUESTIONS WITH 100% VERIFIED CORRECT ANSWERS
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NURS 617 Exam 1 blueprint Unit 9 TEST QUESTIONS WITH 100% VERIFIED CORRECT ANSWERS
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NURS 617 Exam 1 blueprint Unit 9- ANS-Classifications
- ANS-Pathology of Kawasaki disease
(angina) Develops when there is diminished myocardial blood supply. Usually caused by
atherosclerotic plaque progression but can be caused by vasospasm. - ANS-Transient
myocardial ischemia
(infarction extends through the myocardium all the way from the endocardium to epicardium).
Results in severe cardiac dysfunction. Usually presents with ST elevations on ECG → STEMI -
ANS-Transmural MI
(only involves the area directly beneath the endocardium). This usually presents with ST
depression and T-wave inversion: non-STEMI. It is important to distinguish this type because
the likelihood of recurrent clot formation is high: intervention is needed. - ANS-Subendocardial
MI
1.placement of shunt in PA to increase pulmonary blood flow 2.corrective repair of VSD -
ANS-Repair of TOF
24-hour blood pressure monitoring to assess episode frequency, severity, and correlation with
symptoms, ECG, Blood electrolyte labs, Autonomic testing: catecholamine measurements -
ANS-Diagnosis OH
A blood clot that remains attached to a vessel wall, usually in a single side of a lower extremity.
More common in veins than arteries because flow and pressure are lower in the veins than in
the arteries - ANS-Deep vein thrombosis
A condition in which the aorta rises from the RV and the PA rises from the LV. This means that
unoxygenated blood is constantly flowing through systemic circulation and oxygenated blood is
constantly going through pulmonary circulation. INCOMPATIBLE WITH LIFE UNLESS OTHER
DEFECTS EXIST TO ALLOW MIXING. PDA= Aorta to PA. ASD= RA to LA. VSD=RV to LV -
ANS-Transposition of the great arteries
A decrease in systolic blood pressure of at least 20mmHg or a decrease in diastolic blood
pressure of at least 10mmHg within 3 minutes of moving to a standing position. Often called
neurogenic and is usually the result of neurologic disorders at affecting autonomic function. -
ANS-Orthostatic (Postural) hypotension
, A diverse group of mostly idiopathic diseases that affect the myocardium -
ANS-Cardiomyopathies
A localized dilation or outpouching of a vessel wall of cardiac chamber - ANS-Aneurysm
Abnormal communication between the atria. Can occur at the base (ostium primum), in the
center (ostium secundum) and high up (sinus venosus defect) - ANS-Atrial septal defect (ASD)
Abnormal communication between the ventricles. 4 types: perimembranous→ immediately
below the aortic valve; Muscular → low or anterior in the septum; Supracristal VSDs → right
ventricular outflow tract, below the pulmonary valve; AV canal → posterior and inferior to the
membranous septum, beneath the tricuspid valve - ANS-Ventricular septal defect (VSD)
Accumulation of clotting factors and platelets leas to thrombus formation, Inflammation around
the thrombus promotes further platelet aggregation and the thrombus grows proximally. Most
dissolve without treatment → but can develop into VTE!!! - ANS-process of DVT
Accurate blood pressure that should be obtained in the right art with the arm supported at the
level of the heart (3 separate measurements should be taken). White coat hypertension may be
related to fear and anxiety. Ambulatory blood pressure monitoring (ABPM) over a 24 hour period
may be needed - ANS-Evaluation of systemic HTN
Action potential from the SA node causes both atria to contract and begin systole, It then
conducts the action potential onward toward the ventricles, Innervated by nerves from the
autonomic parasympathetic ganglia that serve as receptors for the vagus nerve, From this node,
conducting fibers converge to form the bundle of His, These give rise to the right and left bundle
branches, Terminal branches of the RBB and LBB are the Purkinje fibers - ANS-what does AV
node do
Acute, self-limiting vasculitis, seen mostly in children less than 5 years old, with a higher
incidence in Japan; males affected more than females. Etiology: idiopathic (limited
understanding at this time) - ANS-Kawasaki disease
Acute: fever, conjunctivitis, oral changes ("Strawberry tongue"), rash, and lymphadenopathy.
Acute myocarditis may develop. Subacute: begins when the fever ends and continues until the
clinical signs have resolved. Desquamation of the palms and soles as well as marked
thrombocytosis. Risk for coronary artery aneurysm development. Convalescent: marked by
continued elevation of the erythrocyte sedimentation rate and platelet count. Arthritis may be
present. This phase continues until lab values return to normal- usually about 6 to 8 weeks -
ANS-Clinical manifestations kawasaki disease
Admit to hospital, Place on oxygen, Administer aspirin, Morphine for pain (which also acts as a
vasodilator to coronary arteries), Antithrombotics if needed, Hyperglycemia treated with insulin,
Bed rest - ANS-Treatment of MI
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