Summary
NURS 4540 HYPOTHYROIDISM VS HYPERTHYROIDISM SUMMARY
- Course
- Nurs 4540
- Institution
- Sam Houston State University
This is a comprehensive and detailed summary on HYPOTHYROIDISM VS HYPERTHYROIDISM.
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HYPOTHYROIDISM VS HYPERTHYROIDISMTHYROID UNDERGOES MANY CHANGES IN PREGNANCY. MANY OF THE SIGNS &
SYMPTOMS OF THYROID DISORDERS ARE ALSO COMMON COMPLAINTS DURING
PREGNANCY
HYPO HYPER
OFTEN ASSOCIATED WITH INFERTILITY AND AN INCREASED RISK OF
•RARE IN PREGNANCY: 2 TO 17 OF EVERY 1000
MISCARRIAGE
OCCURS IN 2 TO 12 PREGNANCIES PER 1000 BIRTHS
SYMPTOMS: WEIGHT GAIN, LETHARGY, DECREASE IN EXERCISE CAPACITY •90% TO 95% CASES IN PREGNANCY ARE CAUSED
COLD INTOLERANCE BY GRAVES’ DISEASE
MEDICATION:
LEVOTHYROXINE/SYNTHROID (E.G., T4)
CLINICAL MANIFESTATIONS
CAUSES: •HEAT INTOLERANCE, DIAPHORESIS, FATIGUE,
NOT ENOUGH IODINE ANXIETY, EMOTIONAL LABILITY, AND
HASHIMOTOS: (AUTOIMMUNE DISORDER WHERE THE BODY ATTACKS THE
THYROID GLAND)
TACHYCARDIA
ANTITHYROID MEDICATION TOXICITY USED TO TREAT HYPERTHYROIDISM •MAY INCLUDE WEIGHT LOSS, GOITER, AND A
THYROIDECTOMY PULSE RATE GREATER THAN 100 BEATS/MINUTE
PITUITARY TUMOR (STOP THE ANTERIOR PITUITARY GLAND FROM SECRETING
•PRIMARY TREATMENT DURING PREGNANCY IS
TSH WHICH STIMULATES THE THYROID TO RELEASE T3 AND T4)
SIGNS AND SYMPTOMS:
WEIGHT GAIN
DRUG THERAPY
MODULE
THYROID HORMONES PLAY A ROLE IN BURNING
UNABLE TO TOLERATE COLD
POSSIBLE GOITER FROM CONSTANT THYROID STIMULATION TO GET THE
CALORIES, STIMULATING SYMPATHETIC
NERVOUS SYSTEM (REFLEX, RESPONSES),
5
THYROID GLAND TO PRODUCE T3 AND T4 MOST COMMON SIGN IN
HASHIMOTO’S INCREASING HEART RATE/BLOOD PRESSURE,
AND HOW FAST WE DIGEST FOOD
SLIDES
EXTREMELY TIRED AND FATIGUED
18-19
SLOW HEART RATE
THINNING AND BRITTLE HAIR
CONSTIPATION CAUSES:
MEMORY LOSS TOO MUCH IODINE
MYXEDEMA: SWELLING OF THE SKIN (EYES AND FACE) THAT GIVES IT A WAXY GRAVE’S DISEASE (AUTOIMMUNE DISORDER
APPEARANCE
WHERE THE BODY PRODUCES TSI WHICH THE
DRY SKIN
DEPRESSION BODY THINKS IS TSH…AND THIS CAUSES THE
MENSTRUAL PROBLEMS (IRREGULAR OR HEAVY PERIODS) THYROID GLAND TO RELEASE EXCESSIVE
AMOUNTS OF T3 AND T4)
TOXIC NODULAR GOITER (NODULAR GROWTHS
THAT PRODUCE EXCESSIVE THYROID HORMONE)
THYROID REPLACEMENT MEDICATION TOXICITY
TREATMENT
SIGNS & SYMPTOMS
WEIGHT LOSS
HYPOTHYROIDISM: THYROID HORMONE REPLACEMENT (SYNTHROID, THYROLAR, HEAT INTOLERANCE
CYTOMEL) GOITER (BOTH)
AVOID SEDATIVES AND NARCOTICS BECAUSE THESE PATIENTS ARE VERY RESTLESS, IRRITABLE
FAST HEART RATE
SENSITIVE TO THEM AND THEY INCREASE THE CHANCES OF MYXEDEMA COMA
HAIR LOSS
HYPERTHYROIDISM: DIARRHEA
ANTITHYROID MEDICATIONS SUCH AS: UNABLE TO FOCUS MENTALLY
TAPAZOLE “METHIMAZOLE” OILY SKIN
PTU NERVOUSNESS
IODIDE SOLUTION “LUGOL’S SOLUTION MENSTRUAL PROBLEMS (BOTH)
BETA BLOCKERS: INDERAL
RADIOACTIVE IODINE THERAPY
THYROIDECTOMY
CHAPTER 29 - ENDOCRINE & METABOLIC DISORDERS
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