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Test Bank for Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives, 2nd Edition, Theresa Capriotti
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Test Bank for Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives, 2nd Edition, Theresa Capriotti

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  • December 16, 2024
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  • test bank for davis advantage for pathophysiology

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Test Bank for Davis Advantage for Pathophysiology:
A2 A2 A2 A2 A2 A2




Introductory Concepts and Clinical Perspectives, 2nd
A2 A2 A2 A2 A2




Edition, Theresa Capriotti
A2 A2 A2

,Chapter 1, The Cell in Health and Illness
A2 A2 A2 A2 A2 A2 A2




Multiple A2Choice
Identify A2the A2choice A2that A2best A2completes A2the A2statement A2or A2answers A2the A2question.

1. Which A2statement A2regarding A2the A2sodium–potassium A2pump A2is A2correct?
1. The A2cell’s A2plasma A2membrane A2is A2more A2soluble A2to A2sodium A2ions A2than A2potassium A2ions.
2. The A2concentration A2of A2sodium A2ions A2should A2be A2higher A2inside A2the A2cell A2compartment.
3. The A2concentration A2of A2potassium A2ions A2should A2be A2higher A2outside
the A2cell A2compartment.
A2

4. The A2active A2transport A2involves A2pumping A2out A2three A2sodium A2ions A2and
A2pumping A2in A2two A2potassium A2ions.



2. In A2the A2absence A2of A2oxygen, A2which A2cellular A2function A2creates A2the A2same A2amount A2of A2energy A2as
is A2created A2in A2the A2presence A2of A2oxygen?
A2

1. Dissipation A2of A2pyruvic A2acid
2. Initiation A2of A2the A2citric A2acid A2cycle
3. Activation A2of A2acetyl-coenzyme A2A
4. Creation A2of A2acidosis A2via A2lactic A2acid
3. How A2many A2adenosine A2triphosphates A2(ATPs) A2are A2produced A2in A2aerobic A2energy A2metabolism?
1. A 2 A 2 2
2. A 2 A 2 3
3. A 2 A 2 34
4. A 2 A 2 53

4. Which A2cell A2organelles A2differ A2in A2their A2number A2according A2to A2the A2cell’s A2energy A2needs?
1. Ribosomes
2. Mitochondria
3. Ribonucleic A2acids
4. Deoxyribonucleic A2acids

5. Which A2option A2best A2supports A2the A2reason A2more A2energy A2is A2produced A2when A2a A2person A2is
A2 exercising?
1. Exercise A2causes A2an A2increase A2in A2the A2synthesis A2of A2protein.
2. There A2is A2an A2increase A2in A2the A2production A2of A2pyruvic A2acid A2in A2the A2cells.
3. The A2conversion A2of A2pyruvic A2acid A2to A2lactic A2acid A2is A2increased A2by A2exercise.
4. Muscle A2cells A2have A2more A2mitochondria A2to A2meet A2energy A2demands.
6. When A2does A2ribosomal A2protein A2synthesis A2cease?
1. During A2endoplasmic A2reticulum A2stress
2. During A2the A2synthesis A2of A2adenosine A2triphosphate A2(ATP)
3. During A2a A2severe A2hypoxic A2state
4. During A2the A2processing A2of A2prohormone

7. Which A2cellular A2organelles A2are A2responsible A2for A2propelling A2mucus A2and A2inhaled A2debris A2out A2of A2the
lungs?
A2

1. Cilia
2. Microfilaments

, 3. Secretory A2vesicles
4. Endoplasmic A2reticula

A2 A2 8. A 2 Which A2are A2the A2key A2proteins A2in A2the A2contractile A2units A2of A2the A2muscle A2cells?
1. Actin A2and A2myosin
2. Prohormone A2and A2tubulin
3. Tubulin A2and A2actin
4. Myosin A2and A2prohormone
A2 A2 9. A 2 Which A2deficiency A2causes A2Tay–Sachs A2disease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomal A2enzymes
A2 10. A 2 Which A2is A2a A2characteristic A2of A2adrenoleukodystrophy?
1. Accumulation A2of A2ganglioside
2. Cessation A2of A2ribosomal A2protein A2synthesis
3. Acceleration A2of A2cellular A2proteasome A2activity
4. Accumulation A2of A2long-chain A2fatty A2acids A2in A2the A2nervous A2system
A2 11. A 2 Which A2statement A2regarding A2endoplasmic A2reticulum A2(ER) A2stress A2is A2correct?
1. During A2ER A2stress, A2proteins A2are A2rapidly A2degraded.
2. During A2ER A2stress, A2lipids A2cannot A2travel A2to A2their A2proper A2intracellular A2locations.
3. During A2ER A2stress, A2long-chain A2fatty A2acids A2accumulate A2in A2the A2nervous A2system.
4. During A2ER A2stress, A2nondegraded A2substances A2accumulate A2in A2the A2cells.
A2 12. A2A A2client A2is A2diagnosed A2with A2type A21 A2diabetes A2mellitus. A2At A2a A2cellular A2level, A2which A2function
is A2likely A2to A2be A2involved?
A2

1. Inability A2of A2ribosomes A2to A2produce A2a A2specific A2type A2of A2protein
2. Incorrect A2processing A2of A2a A2protein A2by A2the A2Golgi A2apparatus
3. Stagnation A2of A2a A2previously A2dynamic A2action A2in A2microtubules
4. Obstruction A2of A2the A2smooth A2endoplasmic A2reticulum
A2 13. A 2 A A2newborn A2patient A2exhibits A2characteristics A2of A2severe A2physical A2deformities. A2Which A2cellular
A2component A2is A2examined A2to A2determine A2the A2cause A2and A2probability A2of A2the A2disease A2being
A2genetically A2transferred?
1. Transfer A2RNA
2. Ribosomal A2RNA
3. Double A2helix A2of A2DNA
4. Mitochondrial A2DNA

A2 14. A2A A2hiker A2experiences A2muscle A2pain A2and A2acidosis A2while A2ascending A2a A2mountain A2during
a A2long, A2steep A2climb. A2Which A2is A2the A2reason A2for A2these A2manifestations?
A2

1. Cellular A2hypoxia
2. Autolysis
3. Heterolysis
4. Cellular A2edema

, A2 15. A 2 Which A2factor A2provides A2DNA A2the A2unique A2molecular A2ability A2to A2replicate?
1. The A2pairing A2of A2nitrogenous A2bases
2. The A2presence A2of A2pyrimidine A2bases
3. The A2presence A2of A2nucleotides
4. The A2nitrogenous A2base A2and A2phosphate A2bond
A2 16. A 2 How A2many A2nitrogenous A2bases A2compose A2a A2single A2codon?
1. A 2 A 2 2
2. A 2 A 2 3
3. A 2 A 2 4
4. A 2 A 2 5

A2 17. A 2 Which A2components A2form A2the A2structure A2of A2DNA?
1. Nucleotides
2. Amino A2acids
3. Fatty A2acids
4. Phosphates
A2 18. A 2 Which A2factor A2is A2essential A2in A2order A2for A2protein A2synthesis A2to A2occur?
1. Free-standing A2ribosomes A2within A2the A2cell
2. Protein A2blueprint A2from A2the A2cell A2of A2the A2DNA
3. Specific A2information A2from A2the A2nucleus A2of A2the A2cell
4. Transfer A2RNA A2to A2move A2the A2protein A2out A2of A2the A2cell
A2 19. A2Tetracycline A2antibiotic A2is A2prescribed A2for A2an A2adult A2client A2with A2chlamydia A2infection.
Which A2is A2the A2mechanism A2of A2action A2of A2the A2drug?
A2

1. It A2prevents A2the A2replication A2of A2bacteria.
2. It A2alters A2the A2configuration A2of A2bacterial A2cytoplasm.
3. It A2interferes A2with A2the A2function A2of A2bacterial A2ribosomes.
4. It A2inhibits A2the A2functions A2of A2bacterial A2mitochondria.
A2 20. A 2 Where A2does A2the A2conversion A2of A2a A2prohormone A2into A2a A2hormone A2take A2place?
1. In A2the A2ribosomes
2. In A2the A2Golgi A2apparatus
3. In A2the A2secretory A2granules
4. In A2the A2endoplasmic A2reticulum
A2 21. A 2 Which A2is A2the A2cell’s A2“master A2mind”?
1. Nucleus
2. Ribosome
3. Golgi A2apparatus
4. Endoplasmic A2reticulum


Multiple A2Response
Identify A2one A2or A2more A2choices A2that A2best A2complete A2the A2statement A2or A2answer A2the A2question.

A2 22. A 2 Which A2statements A2regarding A2the A2microtubules A2are A2true? A2Select A2all A2that A2apply.

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