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NR 324/ NR324 EXAM 1: (NEW 2025/ 2026 UPDATE) ADULT HEALTH I GUIDE | QUESTIONS & ANSWERS| GRADE A| 100% CORRECT (VERIFIED SOLUTIONS) $12.99
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NR 324/ NR324 EXAM 1: (NEW 2025/ 2026 UPDATE) ADULT HEALTH I GUIDE | QUESTIONS & ANSWERS| GRADE A| 100% CORRECT (VERIFIED SOLUTIONS)
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NR 324/ NR324 EXAM 1: (NEW 2025/ 2026 UPDATE) ADULT HEALTH I GUIDE | QUESTIONS & ANSWERS| GRADE A| 100% CORRECT (VERIFIED SOLUTIONS) NR 324/ NR324 EXAM 1: (NEW 2025/ 2026 UPDATE) ADULT HEALTH I GUIDE | QUESTIONS & ANSWERS| GRADE A| 100% CORRECT (VERIFIED SOLUTIONS) NR 324/ NR324 EXAM 1: (NEW 202...

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  • January 16, 2025
  • 40
  • 2024/2025
  • Exam (elaborations)
  • Questions & answers

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NR 324




NR 324/ NR324 EXAM 1: (NEW 2025/ 2026 UPDATE)
ADULT HEALTH I GUIDE | QUESTIONS & ANSWERS| GRADE
A| 100% CORRECT (VERIFIED SOLUTIONS)



1. What occurs during the QRS complex of an ECG?:
Ventricular depolarization
2. What occurs during the T wave of an ECG?: Ventricular
repolarization 3. What happens during atrial
depolarization?: Atria's contract, forces blood across valves
into ventricles.
4. What happens during ventricular depolarization?: The
ventricles contract, pressure increases causing the valves to
shut.
5. What happens during ventricular repolarization?: Blood
pushes into pulmonary trunk and aorta, causing the ventricles
to relax and pressure to drop.
6. What is the term for the time between the end of the QRS
complex and the start of the T wave?: ST segment
(interval)
7. What sound occurs when the semilunar valves (aortic &
pulmonary) close?: S2 "dub"




NR 324

,8. What sound occurs when the atrioventricular valves
(mitral & tricuspid) close?: S1 "lub"
9. Most blood enters the ventricle during?: atrial diastole
10. What causes the murmur sound from the heart?: A
turbulent flow of blood
11. The first waveform, the P wave, begins with the firing of
the?: SA Node
12. How do you calculate cardiac output?: stroke volume x
heart rate
13. Where is the SA node located?: upper wall of the right
atrium
14. What condition occurs when chest pain from ischemia is
prolonged and not immediately reversible?: Acute
coronary syndrome (ACS)
15. Caused by a block in a minor artery or a partial
obstruction in a major artery: NSTEMI
16. Occurs when a ruptured plaque blocks a major artery
completely: STEMI
17. Patients with UA or NSTEMI may have what kind of
changes on an ECG?: ST segment depression
T wave inversion
18. How do you distinguish the difference between UA and
NSTEMI?: Serum cardiac biomarkers




,19. How does a STEMI present on a 12-lead ECG?: ST
elevations
20. How are STEMI treated and what is the first line
treatment?: PCl (first line) Thrombolytic therapy
21. NSTEMI patients are diagnosed by what level? What other
levels are monitored?: Troponin
CK, CK-MB
22. How can chronic stable angina be provoked?: Physical
exertion, stress, or emotional upset
23. When your pattern of angina has been stable for several
months, it may be referred to as?: Chronic stable angina
24 A sudden feeling of chest pain or shortness of breath for
the first time or inconsistently for at least two weeks:
Unstable angina
25. Does chronic angina pain change with positioning and
breathing?: No, it does not change
26. Chronic stable angina is usually resolved by resting,
calming down, or taking what kind of medication?: Sublingual
nitroglycerin
27. What is the term for the rare form of angina that occurs
during rest and often with no increased physical activity?:
Prinzmetal's angina (variant angina) 28. An abrupt stop of






, blood flow through the coronary artery with a thrombus
caused by platelet aggregation: Myocardial infarction
29. What does myocardial infarctions cause?: Irreversible
myocardial cell death (necrosis)
30. This condition is preexisting in occurrences with
myocardial infarctions.-
: Coronary artery disease (CAD)
31. What is the difference between primary and secondary
hypertension?: Primary- no identifiable cause
Secondary- has a cause and can be identified and corrected
32. What type of hypertension is referred to as a 'silent killer'
because of it's asymptomatic symptoms until the severity
increases?: primary hypertension 33. What kind of defects
are caused by primary hypertension? Does it improved
when the HTN is treated?: Defects in glucose, insulin and
lipoprotein metabolism
Does not approve when treated
34. A hypertensive emergency often presents as? This is a
syndrome in which a sudden rise in BP is associated with a
severe headache, nausea, vomiting, seizures, confusion, and
coma.: hypertensive encephalopathy 35. What are some
modifiable risk factors for hypertension?: Diet Exercise
Obesity

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