Crna interview - Study guides, Class notes & Summaries
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CRNA Interview Questions and Answers 100% Solved
- Exam (elaborations) • 35 pages • 2024
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CRNA Interview Questions and Answers 
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What are the interventions for cardiogenic shock? ️️-Fluids 
-Vasopressors (Dopamine) 
-Inotropes (Dobutamine, Milrinone for heart failure) 
-IABP, ECMO 
Your ventilator is peak pressure alarming. What could be the problem? ️️-Normal plateau 
pressure - Kink in tubing, patient biting tube, mucous plug, bronchospasm, ETT too small 
-If high plateau pressure - there is an issue with lung compliance ie: developing PNA, pulmonary 
edema, ...
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Comprehensive CRNA Interview Review Questions and Answers 100% Solved
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Comprehensive CRNA Interview Review 
Questions and Answers 100% Solved 
Norepinephrine Mechanism of Action (MOA)️️A1, A2, B1 agonist. 
Primary agent used in distributive shock because it's ability to recruit venous volume and 
augment preload, while increasing arterial tone, and increasing cardiac output. 
Alpha one causing peripheral smooth muscle contraction. (low dose venous, high dose venous 
and arterial). 
Alpha 2 adrenoreceptor agonism actually antagonizes the release of norepine...
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CRNA Interview questions and answers latest update 2024 RATED A+
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CRNA Interview questions and answers latest update 2024 RATED A+
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CRNA Interview Questions and Answers 100% Solved
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CRNA Interview Questions and Answers 
100% Solved 
Norepinephrine Mechanism of Action (MOA) ️️A1, A2, B1 agonist. 
Primary agent used in distributive shock because it's ability to recruit venous volume and 
augment preload, while increasing arterial tone, and increasing cardiac output. 
Alpha one causing peripheral smooth muscle contraction. (low dose venous, high dose venous 
and arterial). 
Alpha 2 adrenoreceptor agonism actually antagonizes the release of norepinephrine in the CNS, ...
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CRNA Interview questions with correct answers
- Exam (elaborations) • 21 pages • 2023
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Norepinephrine Mechanism of Action (MOA) CORRECT ANSWER A1, A2, B1 agonist. 
 
Primary agent used in distributive shock because it's ability to recruit venous volume and augment preload, while increasing arterial tone, and increasing cardiac output. 
 
Alpha one causing peripheral smooth muscle contraction. (low dose venous, high dose venous and arterial). 
 
Alpha 2 adrenoreceptor agonism actually antagonizes the release of norepinephrine in the CNS, but these receptors are less present in p...
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CRNA INTERVIEW (ACTUAL) 2024 QUESTIONS AND ANSWERS
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CRNA INTERVIEW (ACTUAL) 2024 QUESTIONS AND ANSWERS
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CRNA Interview Exam Questions And Correct Answers
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CRNA Interview Exam Questions And Correct Answers...
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CRNA interview Questions and Answers 100% Solved
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CRNA interview Questions and Answers 
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where are alpha 1 receptors located ️️vascular smooth muscle 
where are alpha 2 receptors located ️️presynaptic nerve terminals 
effects of alpha 1 agonism ️️vasoconstriction, increased svr, increased bp 
effects of alpha 2 agonism ️️inhibition of norepinephrine release 
How does dexmedetomidine work? ️️alpha 2 agonism ( blocking release of norepinephrine 
in brain and spinal cord) 
Effects of dexmedetomidine ️️analgesi...
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CRNA Interview Questions and Answers 100% Solved
- Exam (elaborations) • 35 pages • 2024
- Available in package deal
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- $14.99
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CRNA Interview Questions and Answers 
100% Solved 
What are the interventions for cardiogenic shock? ️️-Fluids 
-Vasopressors (Dopamine) 
-Inotropes (Dobutamine, Milrinone for heart failure) 
-IABP, ECMO 
Your ventilator is peak pressure alarming. What could be the problem? ️️-Normal plateau 
pressure - Kink in tubing, patient biting tube, mucous plug, bronchospasm, ETT too small 
-If high plateau pressure - there is an issue with lung compliance ie: developing PNA, pulmonary 
edema, ...
-
CRNA Interview exam 2023/2024 with 100% correct answers
- Exam (elaborations) • 17 pages • 2023
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Norepinephrine Mechanism of Action (MOA) - correct answer A1, A2, B1 agonist. 
 
Primary agent used in distributive shock because it's ability to recruit venous volume and augment preload, while increasing arterial tone, and increasing cardiac output. 
 
Alpha one causing peripheral smooth muscle contraction. (low dose venous, high dose venous and arterial). 
 
Alpha 2 adrenoreceptor agonism actually antagonizes the release of norepinephrine in the CNS, but these receptors are less present ...
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