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Exam 2: NURS 611/ NURS611 (Latest 2024/ 2025 Update) Advanced Pathophysiology Review| Qs & As| 100% Correct| Grade A- Maryville 10,77 €   Añadir al carrito
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Exam 2: NURS 611/ NURS611 (Latest 2024/ 2025 Update) Advanced Pathophysiology Review| Qs & As| 100% Correct| Grade A- Maryville
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  • NURS 611/ NURS611 (NURS611)
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  • Maryville College

Exam 2: NURS 611/ NURS611 (Latest 2024/ 2025 Update) Advanced Pathophysiology Review| Qs & As| 100% Correct| Grade A- Maryville Q: What evidence does the nurse expect to see when a patient experiences trauma to the hypothalamus? Answer: Uneven expression of mood, unstable blood glucose l...

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  • 27 de septiembre de 2024
  • 43
  • 2024/2025
  • Examen
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  • exam 2
  • nurs 611nurs611nurs 611 nurs611 exam
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  • nurs 611nurs611nurs 611 nurs611 exam 2
  • nurs 611nurs611nurs 611 nurs611 exam 3
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ExamI2:INURSI611/INURS611I(LatestI2024
/I2025IUpdate)IAdvancedIPathophysiologyI
Review|IQsI&IAs|I100%ICorrect|IGradeIA-
IMaryville

Q:IWhatIevidenceIdoesItheInurseIexpectItoIseeIwhenIaIpatientIexperiencesItraumaItoItheIhy
pothalamus?


Answer:
UnevenIexpressionIofImood,IunstableIbloodIglucoseIlevels,IandIpoorItempIregulation.ITheIhyp
othalamusIformsItheIbaseIofItheIdiencephalon.IHypothalamicIfunctionIcontrolsIautonomicInerv
ousIsystemIfunction,IregulationIofIbodyItemp,IendocrineIfunctionI(glucoseIlevels),IandIregulati
onIofIemotionalIexpression.




Q:IWhatIisItheIfirstIdefenseIofIourIbodies?

Answer:
SkinIandImucousImembranes




Q:IWhichIactionIisItheIpurposeIofItheIinflammatoryIprocess?

Answer:
ToIpreventIinfectionIofItheIinjuredItissue.IIfItheIepithelialIbarrierIisIdamaged,IthenIaIhighlyIeff
icientIlocalIandIsystemIresponseI(inflammation)IisImobilizedItoIlimitItheIextentIofIdamage,ItoI
protectIagainstIinfection,IandItoIinitiateItheIrepairIofIdamagedItissue.




Q:IWhatIareItheI4IcardinalIsignsIofIinfection?

,Answer:
EdemaI(tumor),IwarmthI(calor),IrednessI(rubor),IandIpainI(dolar).IThereIisIaI5thIsignIknownIa
sIlossIofIfunctionI(functioIlaesa)




Q:IWhichItypeIofIwhiteIbloodIcellIisIfirstItoIarriveIatItheIsiteIofIinfection?

Answer:
Leukocytes




Q:IWhatIcausesItheIedemaIthatIoccursIduringItheIinflammatoryIprocess?

Answer:
IncreasedIcapillaryIpermeability.ITheIincreasedIflowIandIcapillaryIpermeabilityIresultIinIaIleak
ageIofIplasmaIfromItheIvessels,IcausingIswellingIinItheIsurroundingItissueIandIisIsolelyIrespo
nsibleIforIinflammation-inducedIedema.




Q:IWhatIprocessIcausesIheatIandIrednessItoIoccurIduringItheIinflammatoryIprocess?

Answer:
VasodilationIofIbloodIvessels.ITheIincreasedIbloodIflow,IasIaIresultIofIvasodilationIandIincreas
ingIconcentrationIofIredIcellsIatItheIsiteIofIinflammationIcauseIlocallyIincreasedIwarmthIandIr
edness.




Q:ITheIchemotacticIfactorIaffectsItheIinflammatoryIprocessIby?

Answer:
DirectingIleukocytesItoItheIinflamedIarea

,TwoIchemotacticIfactors,IneutrophilIchemotacticIfactorI(NCF)IandIeosinophilIchemotacticIfact
orIofIanaphylaxisI(ECF-
A),IareIreleasedIduringImastIcellIdegranulation.INCFIattractsIneutrophilsI(aItypeIofIleukocytes
),IandIECF-AIattractsIeosinophilsItoItheIsiteIofIinflammation.




Q:IWhatIisIpainImediatedIby?

Answer:
Histamines,Ibradykinins,Ileukotrienes,IandIprostaglandins




Q:ITheIfunctionIofIopsonizationIrelatedItoItheIcomplementIcascadeIisIto:

Answer:
TagItheIpathogenicImicroorganismsIforIdestructionIbyIneutrophilsIandImacrophages.
C3bIadheresItoItheIsurfaceIofIaIpathogenicImicroorganismIandIservesIasIanIefficientIopsonin.I
OpsoninsIareImoleculesIthatItagImicroorganismsIforIdestructionIbyIcellsIofItheIinflammatoryIs
ystem,IprimarilyIneutrophilsIandImacrophages.




Q:IDuringIphagocytosisIwhatIisIoccurringIduringItheIstepIreferredItoIasIopsonization?

Answer:
PhagocytesIrecognizeIandIadhereItoItheIbacteria.IDuringIphagocytosis,IopsonizationIinvolvesIo
nlyItheIrecognitionIandIadherenceIofIphagocytesItoIbacteria.




Q:IWhatIisItheIcorrectIsequenceIofIphagocytosis?

Answer:
RecognitionI(opsonization),Iengulfment,Ifusion,IandIdestruction.

, OnceItheIphagocyticIcellIentersItheIinflammatoryIsite,ItheIonlyIcorrectIsequenceIofIphagocyto
sisIinvolvesIopsonizationIorIrecognitionIofItheItargetIandIadherenceIofItheIphagocyteItoIit,Ieng
ulfment,IorIingestionIorIendocytosis,IandItheIformationIofIphagosome,IfusionIwithIlysosomalI
granulesIwithinItheIphagocyteIandIdestructionIofItheItarget.




Q:IWhichImanifestationsIofIinflammationIisIsystemic?

Answer:
FeverIandIleukocytosis.
TheIonlyIprimaryIsystemicIchangesIassociatedIwithItheIacuteIinflammatoryIresponseIareIfever,
IleukocytosisI(aItransientIincreaseIinIcirculatingIleukocytes),IandIincreasedIlevelsIinIcirculatin
gIplasmaIproteins.




Q:ITheIacuteIinflammatoryIresponseIisIcharacterizedIbyIfeverIthatIisIproducedIbyItheIhypoth
alamusIbeingIaffectedIby?


Answer:
EndogenousIpyrogens
Fever-causingIcytokinesIareIknownIasIendogenousIpyrogens.




Q:IWhenIconsideringIwhiteIbloodIcellIdifferentials,IacuteIinflammatoryIreactionsIareIrelatedI
toIelevationsIofIwhichIleukocyte?


Answer:
NeutrophilsI
OnlyIneutrophilsIareItheIpredominantIphagocytesIinItheIearlyIinflammatoryIsite,IarrivingIwithi
nI6-
12IhoursIafterItheIinitialIinjury,ItheyIingestI(phagocytose)Ibacteria,IdeadIcells,IandIcellularIdeb
risIatItheIinflammatoryIsite.

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