32: Plague, Tularemia, and Brucellosis || with A+ Guaranteed Solutions.
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32: Plague, Tularemia, and Brucellosis || with A+
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32: Plague, Tularemia, And Brucellosis || With A+
human plague: etiology correct answers Yersinia pestis
US zoonoses by prevalence correct answers brucellosis = tularemia (type A - one of the most infectious bacteria) >> plague
Yersinia pestis: microbial characteristics, culture conditions correct answers GNR, facultative anaerobe, BIP...
32: Plague, Tularemia, and Brucellosis || with A+
Guaranteed Solutions.
human plague: etiology correct answers Yersinia pestis
US zoonoses by prevalence correct answers brucellosis = tularemia (type A - one of the most
infectious bacteria) >> plague
Yersinia pestis: microbial characteristics, culture conditions correct answers GNR, facultative
anaerobe, BIPOLAR STAINING (tips darker than the rest); grows on standard culture (in 2
days)
Dx: GNR with dark tips correct answers Yersinia pestis
Y. pestis: diseases caused correct answers bubonic, septicemic, and pneumonic plague
Yersinia species that cause gastroenteritis, and reservoirs correct answers both zoonotic like Y.
pestis: Yersinia enterocolitica and Yersinia pseudotuberculosis (birds especially)
transmission of plague correct answers because zoonosis, man contracts it by exposure to
infected animal - either bitten by infected rodent flea or handling infected animal, OR
bioterrorism
plague: endemic areas correct answers Sub-Saharan Africa, Madagascar; restricted to SW and
California in US (where plague-carrying rats arrived from Asia)
most contagious form of plague correct answers pneumonic plague
most common source of brucellosis in TX correct answers unpasteurized goat milk products
from Mexico
shared features of Yersinia, Francisella, and Brucella spp correct answers all facultatively
anaerobic GNRs that cause zoonoses usually in adults
Y. pestis: pathogenesis for different forms of disease correct answers bacteria grow in high
density in infected flea gut > creates bolus in esophagus that prevents flea from taking bloodmeal
> flea regurgitates bolus into bite site of animal/human > migrate to regional LNs > engulfed by
M0's and proliferate there over next few days while synthesizing more anti-phagocytic Ag ...>
severe painful swelling of LN = BUBONIC plague; lyse LN and disseminate through blood
stream (now resistant to further phagocytosis) causing shock, DIC, and "secondary septicemia"
Y. pestis: host response correct answers bacteria killed by NTs, but they can grow inside M0's
and proliferate > buboe; when they lyse LNs and enter bloodstream, they are resistant to further
phagocytosis due to ++ synthesis of anti-phagocytic Ag while in LN
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