Patho Quiz 7 Questions with Correct Verified Answers and 2024

Myasthenia gravis is an autoimmune disease in which a. neuronal demyelination disrupts nerve transmission. b. muscles become increasingly bulky but weakened. c. acetylcholine receptors are destroyed or dysfunctional. d. acetylcholine release from motor neurons is disrupted. - c. acetylcholine receptors are destroyed or dysfunctional Myasthenia gravis is an autoimmune disease in which acetylcholine receptors are destroyed or dysfunctional. Multiple sclerosis is an autoimmune disease in which neuronal demyelination disrupts nerve transmission. Muscles become weak, but not bulky, in myasthenia gravis. Acetylcholine receptors are destroyed or dysfunction in myasthenia gravis. The initial treatment of an individual experiencing a seizure is concentrated on a. documenting the seizure pattern. b. maintaining an airway. c. administering anticonvulsant medication. d. obtaining an EEG. - b. maintaining an airway Treatment of an individual experiencing a seizure is concentrated on maintaining an airway and protecting the individual from injury. If the seizures are because of irreversible or unidentifiable factors, anticonvulsant medications specific to the type of seizure are the best management. Recording the course of the seizure episode is useful, but is not the initial focus of care. EEG studies may be useful in determining abnormalities which elicit the pathologic mechanism. Compartment syndrome occurs secondary to a. breakdown of RBCs. b. bone infarction. c. muscle necrosis. d. soft-tissue damage. - d. soft-tissue damage Compartment syndrome occurs because of severe soft-tissue damage. Bone infarction and the breakdown of RBCs do not cause compartment syndrome. Muscle necrosis does not cause compartment syndrome, but can result from it. The gate control theory of pain transmission predicts that activity in touch receptors will a. enhance perception of pain. b. activate opioid receptors in the CNS. c. decrease pain signal transmission in the spinal cord. d. increase secretion of substance P in the spinal cord. - c. decrease pain signal transmission in the spinal cord The gate control theory is used to explain how stimulation of large "touch" neurons could inhibit the transmission of nociceptor impulses. Central to the gate control theory is the capacity for interneurons in the spinal cord to modify the transmission of nociceptor impulses. The gate control theory is not based on a theory that activity in touch receptors will enhance perception of pain. Opioid receptors are thought to be the mediators of presynaptic inhibition. One way to inhibit synaptic transmission is through presynaptic inhibition of substance P release from nociceptor neurons.